Nature Medicine
1546-170X
1078-8956
Anh Quốc
Cơ quản chủ quản: Nature Publishing Group , NATURE PORTFOLIO
Phân tích ảnh hưởng
Thông tin về tạp chí
Lĩnh vực:
Biochemistry, Genetics and Molecular Biology (miscellaneous)Medicine (miscellaneous)
Nature Medicine publishes research that addresses the needs and goals of contemporary medicine. Original research ranges from new concepts in human biology and disease pathogenesis to robust preclinical bases for new therapeutic modalities and drug development to all phases of clinical work, as well as innovative technologies aimed at improving human health. Current areas of interest also include, but are not limited to: -Gene and cell therapies -Clinical genomics -Regenerative medicine -High-definition medicine -Effects of the environment in human health -Artificial intelligence in health care -Smart wearable devices -Early disease diagnosis -Microbiome -Aging Nature Medicine also publishes Reviews, Perspectives and other content commissioned from leading scientists in their fields to provide expert and contextualized views of the latest research driving the progress of medicine. The News section is editorially independent and provides topical and timely reporting of upcoming trends affecting medicine, researchers and the general audience.
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Interleukin-15 mediates T cell-dependent regulation of tumor necrosis factor-α production in rheumatoid arthritis
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Upregulation of PD-1 expression on HIV-specific CD8+ T cells leads to reversible immune dysfunction
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Classification and diagnostic prediction of cancers using gene expression profiling and artificial neural networks
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Peripheral immunophenotypes in children with multisystem inflammatory syndrome associated with SARS-CoV-2 infection
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In vivo gene therapy of metachromatic leukodystrophy by lentiviral vectors: correction of neuropathology and protection against learning impairments in affected mice
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The morphogen Sonic hedgehog is an indirect angiogenic agent upregulating two families of angiogenic growth factors
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MicroRNA-132–mediated loss of p120RasGAP activates the endothelium to facilitate pathological angiogenesis
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