Journal of Cell Communication and Signaling

  1873-961X

  1873-9601

 

Cơ quản chủ quản:  Springer Netherlands , SPRINGER

Lĩnh vực:
Cell BiologyBiochemistryMolecular Biology

Các bài báo tiêu biểu

The role of tenascin-C in tissue injury and tumorigenesis
- 2009
Kim S. Midwood, Gertraud Orend
VEGF-A/VEGFR2 signaling network in endothelial cells relevant to angiogenesis
- 2016
Chandran S. Abhinand, Rajesh Raju, Sasikumar J. Soumya, Prabha S. Arya, P. R. Sudhakaran
Functional role of periostin in development and wound repair: implications for connective tissue disease
- 2008
David A. Hamilton
AbstractIntegrity of the extracellular matrix (ECM) is essential for maintaining the normal structure and function of connective tissues. ECM is secreted locally by cells and organized into a complex meshwork providing physical support to cells, tissues, and organs. Initially thought to act only as a scaffold, the ECM is now known to provide a myriad of signals to cells regulating all aspects of their phenotype from morphology to differentiation. Matricellular proteins are a class of ECM related molecules defined through their ability to modulate cell–matrix interactions. Matricellular proteins are expressed at high levels during development, but typically only appear in postnatal tissue in wound repair or disease, where their levels increase substantially. Members of the CCN family, tenascin‐C, osteopontin, secreted protein acidic rich in cysteine (SPARC), bone sialoprotein, thrombospondins, and galectins have all been classed as matricellular proteins. Periostin, a 90 kDa secreted homophilic cell adhesion protein, was recently added to matricellular class of proteins based on its expression pattern and function during development as well as in wound repair. Periostin is expressed in connective tissues including the periodontal ligament, tendons, skin and bone, and is also prominent in neoplastic tissues, cardiovascular disease, as well as in connective tissue wound repair. This review will focus on the functional role of periostin in tissue physiology. Fundamentally, it appears that periostin influences cell behaviour as well as collagen fibrillogenesis, and therefore exerts control over the structural and functional properties of connective tissues in both health and disease. Periostin is a novel matricellular protein with close homology to Drosophila fasciclin 1. In this review, the functional role of periostin is discussed in the context of connective tissue physiology, in development, disease, and wound repair.
Fibulin-5, an integrin-binding matricellular protein: its function in development and disease
Tập 3 Số 3-4 - Trang 337-347 - 2009
Hiromi Yanagisawa, Marie K. Schluterman, Rolf A. Brekken
Connective tissue growth factor (CCN2, CTGF) and organ fibrosis: lessons from transgenic animals
- 2010
David R. Brigstock
AbstractIn recent months, four different systems have been reported in the literature in which CCN2 transgenes were individually expressed in podocytes, hepatocytes, cardiomyocytes or respiratory epithelial cells to achieve overexpression in, respectively, the kidney, liver, heart, or lung. These transgenic systems have provided valuable information about the contribution of CCN2 to fibrosis in vivo and have begun to reveal the complexities of the underlying mechanisms involved. On the one hand, studies of these animals have revealed that CCN2 overexpression does not necessarily lead directly to fibrotic pathology but may cause severe non‐fibrotic tissue damage due to its other effects on cell function (e.g. heart). On the other hand, overexpression of CCN2 in concert with signaling pathways associated with development (e.g. lung) or fibrosing injuries (e.g. kidney, liver) can lead to the initiation or exacerbation of fibrosis. The significance of these studies is discussed in the context of the requirement for interactions between CCN2 and co‐stimulatory factors in the microenvironment for the manifestation of CCN2‐dependent fibrosis.
A network map of Interleukin-10 signaling pathway
Tập 10 Số 1 - Trang 61-67 - 2016
Renu Verma, Lavanya Balakrishnan, Kusum Sharma, Aafaque Ahmad Khan, Jayshree Advani, Harsha Gowda, Srikanth Tripathy, Mrutyunjay Suar, Akhilesh Pandey, Sheetal Gandotra, T. S. Keshava Prasad, Subramanian Shankar
The role of CCN2 in cartilage and bone development
- 2011
Satoshi Kubota, Masaharu Takigawa