LncRNA MIAT impairs cardiac contractile function by acting on mitochondrial translocator protein TSPO in a mouse model of myocardial infarction

Signal Transduction and Targeted Therapy - Tập 6 Số 1
Xue Bai1, Chao Yang1, Lei Jiao1, Hongtao Diao1, Ziyu Meng1, Lei Wang1, Hao Cui1, Lihua Sun1, Yong Zhang2, Baofeng Yang1
1Department of Pharmacology (State-Province Key Laboratories of Biomedicine-Pharmaceutics of China and Key Laboratory of Cardiovascular Medicine Research, Ministry of Education), College of Pharmacy, Harbin Medical University, Harbin, Heilongjiang, P. R. China
2Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Harbin, China

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Tài liệu tham khảo

Qu, X. et al. MIAT is a pro-fibrotic long non-coding RNA governing cardiac fibrosis in post-infarct myocardium. Sci. Rep. 7, 42657 (2017).

Kroemer, G., Galluzzi, L. & Brenner, C. Mitochondrial membrane permeabilization in cell death. Physiol. Rev. 87, 99–163 (2007).

Gatliff, J. & Campanella, M. The 18 kda translocator protein (tspo): a new perspective in mitochondrial biology. Curr. Mol. Med. 12, 356–368 (2012).

Caballero, B., Veenman, L. & Gavish, M. Role of mitochondrial translocator protein (18 kda) on mitochondrial related cell death processes. Recent Pat. Endocr. Metab. Immune Drug. Disco. 7, 86–101 (2013).

Dorstyn, L., Akey, C. W. & Kumar, S. New insights into apoptosome structure and function. Cell. Death. Differ. 25, 1194–1208 (2018).

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Signal Transduction and Targeted Therapy

Tập 6 Số 1

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