The synaptic dysfunction in myasthenia gravis (MG) is caused by a deficiency of
the nicotonic postsynaptic acetylcholine receptor (AChR) protein. The AChR
deficiency in acquired MG is caused by an autoimmune attack on the postsynaptic
membrane. Immunoglobulin G (IgG) can be localized at the MG end-plate with
peroxidase-labelled staphylococcal protein A, and the third component of
complement (C3) c... hiện toàn bộ