Neuroendocrinology
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* Dữ liệu chỉ mang tính chất tham khảo
Sắp xếp:
Facial Receptive Fields of Trigeminal Neurons: Increased Size Following Estrogen Treatment in Female Rats
Neuroendocrinology - Tập 18 Số 1 - Trang 115-124 - 1975
Glucocorticoid Feedback Regulation of Adrenocortical Responses to Neural Stimuli: Role of CRF-41 and Corticosteroid Type I and Type II Receptors
Neuroendocrinology - Tập 58 Số 1 - Trang 49-56 - 1993
Progesterone Promotes Rapid Desensitization of α<sub>1</sub>-Adrenergic Receptor Augmentation of cAMP Formation in Rat Hypothalamic Slices
Neuroendocrinology - Tập 55 Số 1 - Trang 1-8 - 1992
Regional Specificity of Gamma-Aminobutyric Acid Receptor Regulation by Estradiol
Neuroendocrinology - Tập 47 Số 6 - Trang 473-481 - 1988
Estrogen-Dependent and Estrogen-Independent Effects of Progesterone on the Electrophysiological Excitability of Dorsal Midbrain Neurons in Golden Hamsters
Neuroendocrinology - Tập 48 Số 2 - Trang 120-129 - 1988
Quantitative Assessment of Early and Discontinuous Estradiol-Induced Effects on Ventromedial Hypothalamic and Preoptic Area Proteins in Female Rat Brain
Neuroendocrinology - Tập 48 Số 5 - Trang 561-568 - 1988
Effect of Experimental Alterations in Serum Levels of 5α-Androstane-3β,17β-Diol on the Timing of Puberty in the Female Rat
Neuroendocrinology - Tập 39 Số 1 - Trang 19-24 - 1984
Membrane Mechanism Mediates Progesterone Stimulatory Effect on LHRH Release from Superfused Rat Hypothalami in vitro
Neuroendocrinology - Tập 45 Số 6 - Trang 514-517 - 1987
Regulation of High-Affinity GABAa Receptors in Specific Brain Regions by Ovarian Hormones
Neuroendocrinology - Tập 50 Số 3 - Trang 315-320 - 1989
Forced Swimming Differentially Affects Male and Female Brain Corticosteroid Receptors Corticosteroid receptors are key mediators of the neuroendocrine response to stress. Previously, we have determined the effects of restraint stress on the regulation of corticosteroid receptor genes in the brain and pituitary of male and female rats. Significant gender- and regional-specific regulation of receptor mRNAs was observed. To further investigate the stressor specificity in the same context, we have determined glucocorticoid receptor (GR) and mineralocorticoid receptor (MR) mRNAs following exposure to swimming stress paradigms applied alone, or in combination with restraint stress. Our data revealed stressor-specific alterations in GR or MR mRNA levels, which were more pronounced in males, the gender most affected by swimming stress. No alterations in GR or MR mRNA levels were detected in the female hippocampus and hypothalamus upon exposure to swimming paradigms, while in males the same stressors down-regulated GR mRNA in the hippocampus (chronic exposure) and up-regulated both genes in the hypothalamus (acute exposure). In the frontal cortex, acute swimming stress caused a reciprocal change in GR mRNA levels in the two sexes. The above difference is not due to circulating ovarian steroids, since ovariectomy did not change the female pattern of GR gene expression following acute stress. Our results further showed a hypothalamic-pituitary-adrenal axis facilitation to a novel superimposed stressor expressed at the level of limbic corticosteroid receptors: When chronically restrained rats of both sexes were exposed to acute swimming stress, a reduced GR/MR mRNA ratio, implying reduced feedback axis sensitivity, was detected in both the hippocampus and the hypothalamus. In conclusion, our work provides additional evidence on stressor, gender and region specificity in the regulation of brain corticosteroid receptors.
Neuroendocrinology - Tập 75 Số 4 - Trang 217-226 - 2002
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