Arteriosclerosis, Thrombosis, and Vascular Biology
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Sắp xếp:
HDL Phospholipid Content and Composition as a Major Factor Determining Cholesterol Efflux Capacity From Fu5AH Cells to Human Serum
Abstract
The relationships of cell cholesterol efflux to HDL phospholipid (PL) content and composition in human serum were analyzed in two groups of subjects selected on the basis of their HDL cholesterol (HDL-C) levels: a norm-HDL group (1.10 mmol/L<HDL-C<1.50 mmol/L) and a high-HDL group (HDL-C>1.75 mmol/L). In the high-HDL gro... ... hiện toàn bộ
Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 17 Số 11 - Trang 2685-2691 - 1997
Vascular Adhesion Molecules in AtherosclerosisNumerous reports document the role of vascular adhesion molecules in the development and progression of atherosclerosis. Recent novel findings in the field of adhesion molecules require an updated summary of current research. In this review, we highlight the role of vascular adhesion molecules including selectins, vascular cell adhesion molecule (VCAM)-1, intercellular adhesion molecule1 (... ... hiện toàn bộ
Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 27 Số 11 - Trang 2292-2301 - 2007
Natural Killer Cells and CD4 + T-Cells Modulate Collateral Artery Development
Objective—
The immune system is thought to play a crucial role in regulating collateral circulation (arteriogenesis), a vital compensatory mechanism in patients with arterial obstructive disease. Here, we studied the role of lymphocytes in a murine model of hindlimb ischemia.
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Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 27 Số 11 - Trang 2310-2318 - 2007
Aging Causes Collateral Rarefaction and Increased Severity of Ischemic Injury in Multiple Tissues
Objective—
Aging is a major risk factor for increased ischemic tissue injury. Whether collateral rarefaction and impaired remodeling contribute to this is unknown. We quantified the number and diameter of native collaterals and their remodeling in 3-, 16-, 24-, and 31-month-old mice.
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Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 31 Số 8 - Trang 1748-1756 - 2011
Key Role of the NO-Pathway and Matrix Metalloprotease-9 in High Blood Flow-Induced Remodeling of Rat Resistance Arteries
Objective—
Blood flow is altered in metabolic and ischemic diseases with dramatic consequences. Resistance arteries structure and function remodel in response to chronic blood flow changes through a mechanism remaining mainly unknown. We hypothesized that the NO pathway and matrix metallo... ... hiện toàn bộ
Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 27 Số 2 - Trang 317-324 - 2007
Vascular Development
The vascular system forms as a branching network of endothelial cells that acquire identity as arterial, venous, hemogenic, or lymphatic. Endothelial specification depends on gene targets transcribed by Ets domain–containing factors, including Ets variant gene 2 (Etv2), together with the activity of chromatin-remodeling complexes containing Brahma-related gene-1 (Brg1). Once s... ... hiện toàn bộ
Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 38 Số 3 - 2018
Determinants of Flow-Mediated Outward Remodeling in Female Rodents
Objective—
Flow (shear stress)-mediated outward remodeling (FMR) of resistance arteries is a key adaptive process allowing collateral growth after arterial occlusion but declining with age. 17-β-estradiol (E2) has a key role in this process through activation of estrogen receptor α (ERα). Thus, we investigated the impact of age an... ... hiện toàn bộ
Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 34 Số 6 - Trang 1281-1289 - 2014
Key Role of Estrogens and Endothelial Estrogen Receptor α in Blood Flow–Mediated Remodeling of Resistance Arteries
Objective—
Flow- (shear stress–)mediated outward remodeling of resistance arteries is involved in collateral growth during postischemic revascularization. As this remodeling is especially important during pregnancy, we hypothesized that estrogens may be involved. A surgical model eliciting a local increase in blood flow in 1 mesen... ... hiện toàn bộ
Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 33 Số 3 - Trang 605-611 - 2013
Role of NO in Flow-Induced Remodeling of the Rabbit Common Carotid Artery
Flow-induced changes in vessel caliber tend to restore baseline wall shear stress (WSS) and have been reported to be endothelium-dependent. To investigate the role of endothelium-derived nitric oxide (NO) in the adaptive increase in artery diameter in response to a chronic increase in blood flow, an arteriovenous fistula was constructed between the left common carotid artery (... ... hiện toàn bộ
Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 16 Số 10 - Trang 1256-1262 - 1996
Mechanisms of Amplified Arteriogenesis in Collateral Artery Segments Exposed to Reversed Flow Direction
Objective—
Collateral arteriogenesis, the growth of existing arterial vessels to a larger diameter, is a fundamental adaptive response that is often critical for the perfusion and survival of tissues downstream of chronic arterial occlusion(s). Shear stress regulates arteriogenesis; however, the arteriogenic significance of revers... ... hiện toàn bộ
Arteriosclerosis, Thrombosis, and Vascular Biology - Tập 35 Số 11 - Trang 2354-2365 - 2015
Tổng số: 450
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