Wild-Type Transthyretin Cardiac Amyloidosis Is Associated with Increased Antecedent Physical Activity

Journal of Cardiovascular Translational Research - Tập 15 - Trang 689-691 - 2022
Yi Zhen Joan Lee1,2, Johana Fajardo1,3, Emily Brown1, Christopher R. D’Adamo4, Daniel P. Judge1,3
1Johns Hopkins University, Baltimore, USA
2Sinai Hospital, Baltimore, USA
3Medical University of South Carolina, Charleston, USA
4University of Maryland, Baltimore, USA

Tài liệu tham khảo

Mohammed, S. F., Mirzoyev, S. A., Edwards, W. D., et al. (2014). Left ventricular amyloid deposition in patients with heart failure and preserved ejection fraction. JACC Heart Failure, 2, 113–122. Mangione, P. P., Verona, G., Corazza, A., et al. (2018). Plasminogen activation triggers transthyretin amyloidogenesis in vitro. Journal of Biological Chemistry, 293, 14192–14199. Ton, V. K., Bhonsale, A., Gilotra, N. A., et al. (2017). Baseline characteristics predict the presence of amyloid on endomyocardial biopsy. Journal of Cardiac Failure, 23, 340–344. Bull, F. C., Maslin, T. S., & Armstrong, T. (2009). Global physical activity questionnaire (GPAQ): Nine country reliability and validity study. Journal of Physical Activity & Health, 6, 790–804. Carlson, S. A., Fulton, J. E., Schoenborn, C. A., & Loustalot, F. (2010). Trend and prevalence estimates based on the 2008 physical activity guidelines for Americans. American Journal of Preventive Medicine, 39, 305–313.