Viscoelastic measurements of platelet function, not fibrinogen function, predicts sensitivity to tissue‐type plasminogen activator in trauma patients
Tài liệu tham khảo
Sauaia, 1995, Epidemiology of trauma deaths: a reassessment, J Trauma, 38, 185, 10.1097/00005373-199502000-00006
Tisherman, 2015, Detailed description of all deaths in both the shock and traumatic brain injury hypertonic saline trials of the Resuscitation Outcomes Consortium, Ann Surg, 261, 586, 10.1097/SLA.0000000000000837
Brohi, 2003, Acute traumatic coagulopathy, J Trauma, 54, 1127, 10.1097/01.TA.0000069184.82147.06
Brohi, 2008, Acute coagulopathy of trauma: hypoperfusion induces systemic anticoagulation and hyperfibrinolysis, J Trauma, 64, 1211
Ostrowski, 2012, Endothelial glycocalyx degradation induces endogenous heparinization in patients with severe injury and early traumatic coagulopathy, J Trauma Acute Care Surg, 73, 60, 10.1097/TA.0b013e31825b5c10
Schlimp, 2013, The role of fibrinogen in trauma‐induced coagulopathy, Hamostaseologie, 34, 29, 10.5482/HAMO-13-07-0038
Wohlauer, 2012, Early platelet dysfunction: an unrecognized role in the acute coagulopathy of trauma, J Am Coll Surg, 214, 739, 10.1016/j.jamcollsurg.2012.01.050
Kashuk, 2010, Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma, Ann Surg, 252, 434, 10.1097/SLA.0b013e3181f09191
Cotton, 2012, Hyperfibrinolysis at admission is an uncommon but highly lethal event associated with shock and prehospital fluid administration, J Trauma Acute Care Surg, 73, 365, 10.1097/TA.0b013e31825c1234
Moore, 2014, Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapy, J Trauma Acute Care Surg, 77, 811, 10.1097/TA.0000000000000341
Ives, 2012, Hyperfibrinolysis elicited via thromboelastography predicts mortality in trauma, J Am Coll Surg, 215, 496, 10.1016/j.jamcollsurg.2012.06.005
Cardenas, 2014, Elevated tissue plasminogen activator and reduced plasminogen activator inhibitor promote hyperfibrinolysis in trauma patients, Shock, 41, 514, 10.1097/SHK.0000000000000161
Booth, 1999, Fibrinolysis and thrombosis, Bailliere's Best Pract Res Clin Haematol, 12, 423, 10.1053/beha.1999.0034
Moore, 2015, Plasma is the physiologic buffer of tissue plasminogen activator‐mediated fibrinolysis: rationale for plasma‐first resuscitation after life‐threatening hemorrhage, J Am Coll Surg, 220, 872, 10.1016/j.jamcollsurg.2015.01.026
Bennett, 1990, Complexing of tissue plasminogen activator with PAI‐1, alpha 2‐macroglobulin, and C1‐inhibitor: studies in patients with defibrination and a fibrinolytic state after electroshock or complicated labor, Blood, 75, 671, 10.1182/blood.V75.3.671.671
Mutch, 2010, Model thrombi formed under flow reveal the role of factor XIII‐mediated cross‐linking in resistance to fibrinolysis, J Thromb Haemost, 8, 2017, 10.1111/j.1538-7836.2010.03963.x
Bouma, 2006, Thrombin activatable fibrinolysis inhibitor (TAFI) – how does thrombin regulate fibrinolysis?, Ann Med, 38, 378, 10.1080/07853890600852898
Moore, 2015, Hemolysis exacerbates hyperfibrinolysis, whereas platelolysis shuts down fibrinolysis: evolving concepts of the spectrum of fibrinolysis in response to severe injury, Shock, 43, 39, 10.1097/SHK.0000000000000245
Dirkmann, 2012, Factor XIII and tranexamic acid but not recombinant factor VIIa attenuate tissue plasminogen activator‐induced hyperfibrinolysis in human whole blood, Anest Analg, 114, 1182, 10.1213/ANE.0b013e31823b6683
Harr, 2013, Functional fibrinogen assay indicates that fibrinogen is critical in correcting abnormal clot strength following trauma, Shock, 39, 45, 10.1097/SHK.0b013e3182787122
Castellino, 2014, Traumatic brain injury causes platelet adenosine diphosphate and arachidonic acid receptor inhibition independent of hemorrhagic shock in humans and rats, J Trauma Acute Care Surg, 76, 1169, 10.1097/TA.0000000000000216
D'Alessandro, 2015, Early hemorrhage triggers metabolic responses that build up during prolonged shock, Am J Physiol Regul, 308, 1034
D'Alessandro, 2015, Routine storage of red blood cell (RBC) units in additive solution‐3: a comprehensive investigation of the RBC metabolome, Transfusion, 55, 1034, 10.1111/trf.12975
Wiener, 2015, Shock releases bile acid inducing platelet inhibition and fibrinolysis, J Surg Res, 195, 390, 10.1016/j.jss.2015.01.046
Salooja, 2001, Thrombelastography, Blood Coagul Fibrinolysis, 12, 327, 10.1097/00001721-200107000-00001
Chapman, 2013, Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy, J Trauma Acute Care Surg, 75, 961, 10.1097/TA.0b013e3182aa9c9f
Moore, 2015, Fibrinolysis shutdown phenotype masks changes in rodent coagulation in tissue injury versus hemorrhagic shock, Surgery, 158, 386, 10.1016/j.surg.2015.04.008
Chin, 2015, Exploring ethical conflicts in emergency trauma research: the COMBAT (Control of Major Bleeding after Trauma) study experience, Surgery, 157, 10, 10.1016/j.surg.2014.05.021
Kutcher, 2013, A principal component analysis of coagulation after trauma, J Trauma Acute Care Surg, 74, 1223, 10.1097/01586154-201305000-00006
Duan, 2014, A time course study of acute traumatic coagulopathy prior to resuscitation: from hypercoagulation to hypocoagulation caused by hypoperfusion?, Transfus Apher Sci, 50, 399, 10.1016/j.transci.2014.03.008
Prat, 2015, Comprehensive evaluation of coagulation in swine subjected to isolated primary blast injury, Shock, 43, 598, 10.1097/SHK.0000000000000346
Schreiber, 2005, Hypercoagulability is most prevalent early after injury and in female patients, J Trauma, 58, 475, 10.1097/01.TA.0000153938.77777.26
Muller, 2014, Thromboelastometry and organ failure in trauma patients: a prospective cohort study, Crit Care, 18, 687, 10.1186/s13054-014-0687-6
Deras, 2014, Early coagulopathy at hospital admission predicts initial or delayed fibrinogen deficit in severe trauma patients, J Trauma Acute Care Surg, 77, 433, 10.1097/TA.0000000000000314
Schlimp, 2014, The effectiveness of different functional fibrinogen polymerization assays in eliminating platelet contribution to clot strength in thromboelastometry, Anesth Analg, 118, 269, 10.1213/ANE.0000000000000058
Brohi, 2007, Acute traumatic coagulopathy: initiated by hypoperfusion: modulated through the protein C pathway?, Ann Surg, 245, 812, 10.1097/01.sla.0000256862.79374.31
Schochl, 2013, Hyperfibrinolysis is common in out‐of‐hospital cardiac arrest: results from a prospective observational thromboelastometry study, Resuscitation, 84, 454, 10.1016/j.resuscitation.2012.08.318
Hoffman, 2001, A cell‐based model of hemostasis, Thromb Haemost, 85, 958, 10.1055/s-0037-1615947
Stalker, 2013, Hierarchical organization in the hemostatic response and its relationship to the platelet‐signaling network, Blood, 121, 1875, 10.1182/blood-2012-09-457739
Brass, 2005, Minding the gaps to promote thrombus growth and stability, J Clin Invest, 115, 3385, 10.1172/JCI26869
Brass, 2012, Minding the gaps – and the junctions, too, Circulation, 125, 2414, 10.1161/CIRCULATIONAHA.112.106377
Colace, 2014, Microfluidic assay of hemophilic blood clotting: distinct deficits in platelet and fibrin deposition at low factor levels, J Thromb Haemost, 12, 147, 10.1111/jth.12457
Muthard, 2012, Blood clots are rapidly assembled hemodynamic sensors: flow arrest triggers intraluminal thrombus contraction, Arterioscler Thromb Vasc Biol, 32, 2938, 10.1161/ATVBAHA.112.300312
Li, 2010, Signaling during platelet adhesion and activation, Arterioscler Thromb Vasc Biol, 30, 2341, 10.1161/ATVBAHA.110.207522
Mazepa, 2013, Superactivated platelets: thrombus regulators, thrombin generators, and potential clinical targets, Arterioscler Thromb Vasc Biol, 33, 1747, 10.1161/ATVBAHA.113.301790
Peltz, 2015, Pathologic metabolism: an exploratory study of the plasma metabolome of critical injury, J Trauma Acute Care Surg, 78, 742, 10.1097/TA.0000000000000589
Pereira, 1995, In vivo effect of bile salts on platelet aggregation in rats, Thromb Res, 80, 357, 10.1016/0049-3848(95)00187-V
Shiao, 1993, The mode of action of primary bile salts on human platelets, Biochim Biophys Acta, 1146, 282, 10.1016/0005-2736(93)90367-9
de la Pena, 2000, Inhibition of platelet aggregation by putrescine, spermidine, and spermine in hypercholesterolemic rabbits, Arch Med Res, 31, 546, 10.1016/S0188-4409(00)00238-1
Mendez, 1997, Inhibition of platelet aggregation by L‐arginine and polyamines in alloxan treated rats, Biochem Mol Biol Int, 43, 311
Martini, 2007, Evaluation of tris‐hydroxymethylaminomethane on reversing coagulation abnormalities caused by acidosis in pigs, Crit Care Med, 35, 1568, 10.1097/01.CCM.0000266682.74602.6A
Darlington, 2011, Coagulation changes to systemic acidosis and bicarbonate correction in swine, J Trauma, 71, 1271
Cohen, 2009, Early release of high mobility group box nuclear protein 1 after severe trauma in humans: role of injury severity and tissue hypoperfusion, Crit Care, 13, R174, 10.1186/cc8152
Fink, 2007, Bench‐to‐bedside review: high‐mobility group box 1 and critical illness, Crit Care, 11, 229, 10.1186/cc6088
Ding, 2014, Toll‐like receptor 4 regulates platelet function and contributes to coagulation abnormality and organ injury in hemorrhagic shock and resuscitation, Circ Cardiovasc Genet, 7, 615, 10.1161/CIRCGENETICS.113.000398
Solomon, 2011, Platelet function following trauma. A multiple electrode aggregometry study, Thromb Haemost, 106, 322, 10.1160/TH11-03-0175
Windelov, 2014, Platelet aggregation following trauma: a prospective study, Blood Coagul Fibrinolysis, 25, 67, 10.1097/MBC.0b013e328364c2da
Ellis, 2007, Thrombelastographic measures of clot propagation: a comparison of alpha with the maximum rate of thrombus generation, Blood Coagul Fibrinolysis, 18, 45, 10.1097/MBC.0b013e3280111a8e
Darlington, 2011, Effect of hemodilution on coagulation and recombinant factor VIIa efficacy in human blood in vitro, J Trauma, 71, 1152
Ostrowski, 2015, Association between biomarkers of endothelial injury and hypocoagulability in patients with severe sepsis: a prospective study, Crit Care, 19, 191, 10.1186/s13054-015-0918-5