Twenty‐four hour variation of transferrin saturation in treated and untreated haemochromatosis homozygotes

Journal of Internal Medicine - Tập 226 Số 5 - Trang 373-379 - 1989
Corwin Q. Edwards1, L M Griffen2, Jerry Kaplan3, J P Kushner2
1Department of Internal Medicine, University of Utah College of Medicine, Salt Lake City.
2Department of Internal Medicine, Salt Lake City, UT 841 32, USA
3Department of Pathology, University of Utah College of Medicine, Salt Lake City, UT 841 32, USA

Tóm tắt

Abstract Edwards CQ, Griffen LM, Kaplan J, Kushner JP. (Departments of Internal Medicine and Pathology, University of Utah College of Medicine, and LDS Hospital, Salt Lake City, UT 84132, USA). Twenty‐four hour variation of transferrin saturation in treated and untreated haemochromatosis homozygotes.

We studied 43 haemochromatosis homozygotes and seven normal individuals to test the hypothesis that there is an almost continuous relative deficiency of apotransferrin in treated and untreated homozygotes. Transferrin saturation was measured at 2‐h intervals for 24 h while all subjects ate their usual diet. Subjects with haemochromatosis were separated into four groups based on sex and whether or not they had been iron‐depleted. Nineteen treated and 11 untreated male homozygotes had mean transferrin saturation values of 70 and 81%, respectively. Five treated and eight untreated female homozygotes had mean transferrin saturation values of 71 and 69%. Normal subjects had mean transferrin saturation values of 29% (3 males) and 21% (4 females). These data demonstrate continuously high transferrin saturation values, greater than 69% in most treated and untreated male and female homozygotes, resulting in hepatic iron accumulation of non‐transferrin‐bound iron from the portal circulation.

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