Regulation of neuronal cdc20 (p55cdc) expression by the plasticity‐related transcription factor zif268

Synapse - Tập 61 Số 6 - Trang 463-468 - 2007
Ann-Marie Conway1, Allan B. James2, Jie Zang2, Brian Morris3
1Division of Neuroscience and Biomedical Systems, Institute of Biomedical and Life Sciences, University of Glasgow, Glasgow, Scotland, UK
2Division of Neuroscience and Biomedical Systems, Institute of Biomedical and Life Sciences, University of Glasgow, Glasgow, Scotland, United Kingdom.
3Division of Neuroscience and Biomedical Systems, Institute of Biomedical and Life Sciences, West Medical Building, University of Glasgow, Glasgow G12 8QQ, Scotland, UK

Tóm tắt

Abstract

Most forms of neuronal plasticity are associated with induction of the transcription factor zif268 (egr1). Down‐regulation of cdc20 (p55cdc)—a regulatory protein for the anaphase‐promoting complex, which controls access of specific substrates to the proteasome—was observed after transfection of a neuronal cell line with zif268. Treatment of cultured hippocampal neurones with NMDA, which elevates endogenous zif268 levels, also decreased cdc20 levels. Conversely, the levels of cdc20 were found to be increased in the cerebral cortex of mice with targeted deletion of the zif268 gene, when compared with wild‐type controls. Our findings indicate that expression of the cdc20 gene is down‐regulated by zif268 in neuronal cells, and provide new evidence that altered expression of proteasome‐regulatory genes following zif268 induction may be a key component of long‐lasting CNS plasticity. Synapse 61:463‐468, 2007. © 2007 Wiley‐Liss, Inc.

Từ khóa


Tài liệu tham khảo

10.1124/mol.61.2.379

10.1016/S0197-0186(96)00136-2

10.1002/hipo.10100

10.1016/S0896-6273(03)00674-3

10.1016/j.febslet.2004.07.055

10.1016/S0306-4522(98)00556-9

10.1016/S0021-9258(19)85286-9

10.1038/340474a0

10.1016/j.yexcr.2004.06.022

10.1016/S0169-328X(96)00272-0

10.1093/nar/gkg192

Dinkel A, 1997, Transcription factor Egr‐1 activity down‐regulates Fas and CD23 expression in B cells, J Immunol, 159, 2678, 10.4049/jimmunol.159.6.2678

10.1038/ncb984

10.1002/hipo.450040505

10.1074/jbc.M101354200

10.1073/pnas.96.20.11317

10.1006/bbrc.1999.0182

10.1074/jbc.M403190200

10.1078/0171-9335-00126

10.1016/0304-3940(90)90158-6

10.1016/j.cellsig.2004.04.001

10.1111/j.1471-4159.2005.03400.x

10.1523/JNEUROSCI.4199-05.2006

10.1038/85138

10.1016/j.cub.2004.11.010

10.1093/emboj/18.20.5528

10.1126/science.1093712

Levkovitz Y, 2002, A dominant negative Egr inhibitor blocks nerve growth factor‐induced neurite outgrowth by suppressing c‐Jun activation: Role of an Egr/c‐Jun complex, J Neurosci, 22, 3845, 10.1523/JNEUROSCI.22-10-03845.2002

10.1615/CritRevOncog.v7.i1-2.70

10.1073/pnas.89.15.6818

10.1073/pnas.0306069101

10.1006/nlme.1998.3848

Morris BJ, 1995, Stimulation of immediate‐early gene expression in striatal neurones by nitric oxide, J Biol Chem, 270, 24740, 10.1016/S0021-9258(18)87042-9

10.1093/acprof:oso/9780198509981.003.0014

10.1111/j.1460-9568.1996.tb00733.x

10.1101/gad.918201

10.1016/S0014-5793(99)01420-9

10.1083/jcb.200203035

10.1016/S0169-328X(96)00148-9

10.1074/jbc.275.22.16879

10.1038/nn1531

10.1074/jbc.274.5.3199

10.1210/mend.12.1.0049

10.1083/jcb.200104130

10.1073/pnas.091062498

10.1016/j.cell.2004.11.028

10.1016/j.cardiores.2004.09.026

10.1093/emboj/cdg071

10.1083/jcb.149.7.1325

10.1074/jbc.M505019200

10.1016/0896-6273(90)90118-Y

10.1073/pnas.88.12.5106

10.1016/S1534-5807(04)00419-8

10.1186/gb-2003-4-4-r28

10.1152/ajprenal.00426.2002

10.1073/pnas.082092399