Ung thư miệng ở người sử dụng Khat: Tìm kiếm bằng chứng từ phân tích DNA các đột biến của chín gen liên quan đến ung thư

Yip TT, Ngan RK, Fong AH, et al. Application of circulating plasma/serum EBV DNA in the clinical management of nasopharyngeal carcinoma. Oral Oncol. 2014;50(6):527–38. Alqahtani WS, Almufareh NA, Al-Johani HA, et al. Oral and oropharyngeal cancers and possible risk factors across Gulf Cooperation Council countries: a systematic review. World J Oncol. 2020;11(4):173. Al-Jaber A, Al-Nasser L, El-Metwally A. Epidemiology of oral cancer in Arab countries. Saudi Med J. 2016;37(3):249. Alqahtani WS, Almufareh NA, Domiaty DM, et al. Epidemiology of cancer in Saudi Arabia thru 2010–2019: a systematic review with constrained meta-analysis. AIMS Public Health. 2020;7(3):679. Capote-Moreno A, Brabyn P, Muñoz-Guerra MF, et al. Oral squamous cell carcinoma: epidemiological study and risk factor assessment based on a 39-year series. Int J Oral Maxillofac Surg. 2020;49(12):1525–34. Kujan O, Farah CS, Johnson NW. Oral and oropharyngeal cancer in the Middle East and North Africa: Incidence, mortality, trends, and gaps in public databases as presented to the Global Oral Cancer Forum. Transl Res Oral Oncol. 2017;2:2057178X17698480. South AP, den Breems NY, Richa T, et al. Mutation signature analysis identifies increased mutation caused by tobacco smoke associated DNA adducts in larynx squamous cell carcinoma compared with oral cavity and oropharynx. Sci Rep. 2019;9(1):1–9. Campbell JD, Yau C, Bowlby R, et al. Genomic, pathway network, and immunologic features distinguishing squamous carcinomas. Cell Rep. 2018;23(1):194–212. Al-Mugahed L. Khat chewing in Yemen: turning over a new leaf: Khat chewing is on the rise in Yemen, raising concerns about the health and social consequences. Bull World Health Org. 2008;86(10):741–3. Al-Hebshi N, Skaug N. Khat (Catha edulis)—an updated review. Add Biol. 2005;10(4):299–307. Al-Maweri SA, Warnakulasuriya S, Samran A. Khat (Catha edulis) and its oral health effects: an updated review. J Invest Clin Dent. 2018;9(1):e12288. Alshahrani SA, Almufareh NA, Almarshady B, et al. Effects of Consuming Catha Edulis Forsk (khat) on the Gene Manifestation of CHRM1 and CHRM3 in relation to Salivary Glands, Saliva Flow Rates, pH and Dental Caries in Yemeni Consumers. Open Dent J. 2020;14(1). Faulkner S, Jobling P, March B, et al. Tumor neurobiology and the war of nerves in cancer. Cancer Discov. 2019;9(6):702–10. Dasgupta A. Abuse of magic mushroom, peyote cactus, LSD, khat, and volatiles. In Critical issues in alcohol and drugs of abuse testing. Academic Press. 2019: 477–494. Zahalka AH, Frenette PS. Nerves in cancer. Nat Rev Cancer. 2020;23:1–5. Horvathova L, Mravec B. Effect of the autonomic nervous system on cancer progression depends on the type of tumor: solid are more affected then ascitic tumors. Endocrine Regul. 2016;50(4):215–24. Kuol N, Stojanovska L, Apostolopoulos V, et al. Crosstalk between cancer and the neuro-immune system. J Neuroimmunol. 2018;315:15–23. Suliman RS, Algebaly AS, Alqahtani WS. Role of human PTEN and TP53 sequence mutations in the Etiology of Breast Cancer in Saudi Patients. Pak J Biol Sci. 2020;23(3):321–30. French SW, Lee J, Zhong J, et al. Alcoholic liver disease-Hepatocellular carcinoma transformation. J Gastrointest Oncol. 2012;3(3):174. Yang J, Trépo E, Nahon P, et al. A 17-beta‐hydroxysteroid dehydrogenase 13 variant protects from hepatocellular carcinoma development in alcoholic liver disease. Hepatology. 2019;70(1):231–40. Bosetti C, Carioli G, Santucci C, et al. Global trends in oral and pharyngeal cancer incidence and mortality. Int J Cancer. 2020;15(4):1040–9. Conforti F, Pala L, Giaccone G, De Pas T. Thymic epithelial tumors: from biology to treatment. Cancer Treat Rev. 2020;86:102014. Warnakulasuriya S, Greenspan JS. Epidemiology of oral and oropharyngeal cancers. WarnakulasuriyaGreenspaneditors Textbook of oral cancer. Berlin: Springer; 2020. p. 5–22. Jiang X, Wu J, Wang J, et al. Tobacco and oral squamous cell carcinoma: a review of carcinogenic pathways. Tobacco induced diseases. 2019;17. Al-Motarreb A, Al-Habori M, Broadley KJ. Khat chewing, cardiovascular diseases and other internal medical problems: the current situation and directions for future research. J Ethnopharmacol. 2010;132(3):540–8. Balint EE, Falkay G, Balint GA. Khat–a controversial plant. Wiener Klinische Wochenschrift. 2009;121(19):604–14. Pennings EJ, Opperhuizen A, Van Amsterdam JG. Risk assessment of khat use in the Netherlands: a review based on adverse health effects, prevalence, criminal involvement and public order. Regul Toxicol Pharmacol. 2008;52(3):199–207. Al-Habori M. The potential adverse effects of habitual use of Catha edulis (khat). Expert Opin Drug Saf. 2005;4(6):1145–54. Wiergowski M, Aszyk J, Kaliszan M, et al. Identification of novel psychoactive substances 25B-NBOMe and 4-CMC in biological material using HPLC-Q-TOF-MS and their quantification in blood using UPLC–MS/MS in case of severe intoxications. J Chromatogr B. 2017;1041:1–0. Wabe NT. Chemistry, pharmacology, and toxicology of khat (Catha edulis forsk): a review. Addict Health. 2011;3(3–4):137. Wabe NT, Mohammed MA. What science says about khat (Catha edulis Forsk)? Overview of chemistry, toxicology and pharmacology. J Exp Integrat Med. 2012;2(1):29–37. Ademe BW, Brimer L, Dalsgaard A, et al. Chemical and microbiological hazards of Khat (Catha edulis) from field to chewing in Ethiopia. GSC Biol Pharm Sci. 2020;11(1):024–35. Brenneisen R, Fisch HU, Koelbing U, et al. Amphetamine-like effects in humans of the khat alkaloid cathinone. Br J Clin Pharmacol. 1990;30(6):825–8. Hassan NA, Gunaid AA, Abdo-Rabbo AA, et al. The effect of Qat chewing on blood pressure and heart rate in healthy volunteers. Trop Doctor. 2000;30(2):107–8. Al-Motarreb A, Briancon S, Al‐Jaber N, et al. Khat chewing is a risk factor for acute myocardial infarction: a case‐control study. Br J Clin Pharmacol. 2005;59(5):574–81. Al-Sharafi BA, Gunaid AA. Effect of habitual khat chewing on glycemic control, body mass index, and age at diagnosis of diabetes in patients with type 2 diabetes mellitus in Yemen. Clin Med Insights: Endocrinol Diabetes. 2015;8:CMED-S26045. Hassan NA, Gunaid AA, Murray Lyon IM. Khat [Catha edulis]: health aspects of khat chewing. EMHJ-Eastern Mediterranean Health J. 2007;13(3):706–18. Lui VW, Hedberg ML, Li H, Vangara BS, et al. Frequent mutation of the PI3K pathway in head and neck cancer defines predictive biomarkers. Cancer Disc. 2013;3(7):761–9. Galot R, van Marcke C, Helaers R, et al. Liquid biopsy for mutational profiling of locoregional recurrent and/or metastatic head and neck squamous cell carcinoma. Oral Oncol. 2020;104:104631. Husain N, Neyaz A. Molecular Diagnostics in Head and Neck Squamous Cell Carcinoma. In Molecular Diagnostics in Cancer Patients 2019 (pp. 165–185). Springer, Singapore. Su SC, Lin CW, Liu YF, et al. Exome sequencing of oral squamous cell carcinoma reveals molecular subgroups and novel therapeutic opportunities. Theranostics. 2017;7(5):1088. Ma X, Sheng S, Wu J, et al. LncRNAs as an intermediate in HPV16 promoting myeloid-derived suppressor cell recruitment of head and neck squamous cell carcinoma. Oncotarget. 2017;8(26):42061. Er TK, Wang YY, Chen CC, et al. Molecular characterization of oral squamous cell carcinoma using targeted next-generation sequencing. Oral Diseases. 2015;21(7):872–8. Liu H, Zhao H. Prognosis related miRNAs, DNA methylation, and epigenetic interactions in lung adenocarcinoma. Neoplasma. 2019;66(3):487–93. Abrahao AC, Bonelli BV, Nunes FD, et al. Immunohistochemical expression of p53, p16 and hTERT in oral squamous cell carcinoma and potentially malignant disorders. Braz Oral Res. 2011;25:34–41. Renzi A, De Bonis P, Morandi L, et al. Prevalence of p53 dysregulations in feline oral squamous cell carcinoma and non-neoplastic oral mucosa. Plos one. 2019;14(4):e0215621. Su W, Sun S, Wang F, et al. Circular RNA hsa_circ_0055538 regulates the malignant biological behavior of oral squamous cell carcinoma through the p53/Bcl-2/caspase signaling pathway. J Transl Med. 2019;17(1):1–2. Yao Y, Zhou WY, He RX. Down-regulation of JMJD5 suppresses metastasis and induces apoptosis in oral squamous cell carcinoma by regulating p53/NF-κB pathway. Biomed Pharmacother. 2019;109:1994–2004.