Không có mối tương quan giữa con đường p38 MAPK và suy giảm chức năng co bóp trong các tế bào cơ tim tiểu đường

Adeghate E (2004) Molecular and cellular basis of the aetiology and management of diabetic cardiomyopathy: a short review. Mol Cell Biochem 261:187–191 Afzal M, Matsugo S, Sasai M, Xu B, Aoyama K, Takeuchi T (2003) Method to overcome photoreaction, a serious drawback to the use of dichlorofluorescin in evaluation of reactive oxygen species. Biochem Biophys Res Commun 304:619–624 Babick AP, Cantor EJ, Babick JT, Takeda N, Dhalla NS, Netticadan T (2004) Cardiac contractile dysfunction in J2N-k cardiomyopathic hamsters is associated with impaired SR function and regulation. Am J Physiol Cell Physiol 287:1202–1208 Dyntar D, Eppenberger-Eberhardt M, Maedler K, Pruschy M, Eppenberger HM, Spinas GA, Donath MY (2001) Glucose and palmitic acid induce degeneration of myofibrils and modulate apoptosis in rat adult cardiomyocytes. Diabetes 50:2105–2113 Factor SM, Minase T, Sonnenblick EH (1980) Clinical and morphological features of human hypertensive-diabetic cardiomyopathy. Am Heart J 99:446–458 Fang ZY, Prins JB, Marwick TH (2004) Diabetic cardiomyopathy: evidence, mechanisms, and therapeutic implications. Endocr Rev 25:543–567 Fatkin D, Graham RM (2002) Molecular mechanisms of inherited cardiomyopathies. Physiol Rev 82:945–980 Guo M, Wu MH, Korompai F, Yuan SY (2003) Upregulation of PKC genes and isozymes in cardiovascular tissues during early stages of experimental diabetes. Physiol Genomics 12:139–146 Howarth FC, Qureshi MA, White E (2002) Effects of hyperosmotic shrinking on ventricular myocyte shortening and intracellular Ca(2+) in streptozotocin-induced diabetic rats. Pflugers Arch 444:446–451 Huang XP, Du JF (2004) Troponin I, cardiac diastolic dysfunction and restrictive cardiomyopathy. Acta Pharmacol Sin 25:1569–1575 Igarashi M, Wakasaki H, Takahara N, Ishii H, Jiang ZY, Yamauchi T, Kuboki K, Meier M, Rhodes CJ, King GL (1999) Glucose or diabetes activates p38 mitogen-activated protein kinase via different pathways. J Clin Invest 103:185–195 Kannel WB, Belanger AJ (1991) Epidemiology of heart failure. Am Heart J 121:951–957 Kent RL, Mann DL, Urabe Y, Hisano R, Hewett KW, Loughnane M, Cooper G IV (1989) Contractile function of isolated feline cardiocytes in response to viscous loading. Am J Physiol 257:H1717–H1727 Norby FL, Aberle NS II, Kajstura J, Anversa P, Ren J (2004) Transgenic overexpression of insulin-like growth factor I prevents streptozotocin-induced cardiac contractile dysfunction and beta-adrenergic response in ventricular myocytes. J Endocrinol 180:175–182 Privratsky JR, Wold LE, Sowers JR, Quinn MT, Ren J (2003) AT1 blockade prevents glucose-induced cardiac dysfunction in ventricular myocytes: role of the AT1 receptor and NADPH oxidase. Hypertension 42:206–212 Raimondi L, De Paoli P, Mannucci E, Lonardo G, Sartiani L, Banchelli G, Pirisino R, Mugelli A, Cerbai E (2004) Restoration of cardiomyocyte functional properties by angiotensin II receptor blockade in diabetic rats. Diabetes 53:1927–1933 Ren J, Davidoff AJ (1997) Diabetes rapidly induces contractile dysfunctions in isolated ventricular myocytes. Am J Physiol 272:148–158 Ren J, Gintant GA, Miller RE, Davidoff AJ (1997) High extracellular glucose impairs cardiac E-C coupling in a glycosylation-dependent manner. Am J Physiol 273:2876–2883 Severson DL (2004) Diabetic cardiomyopathy: recent evidence from mouse models of type 1 and type 2 diabetes. Can J Physiol Pharmacol 82:813–823 Schluter K-D, Simm A, Schafer M, Taimor G and Piper HM (1999) Early response kinase and PI3-kinase activation in adult cardiomyocytes and their role in hypertrophy. Am J Physiol 276:1655–1663 Schulze PC, Yoshioka J, Takahashi T, He Z, King GL, Lee RT (2004) Hyperglycemia promotes oxidative stress through inhibition of thioredoxin function by thioredoxin-interacting protein. J Biol Chem 279:30369–30374 Smoak IW (2004) Hyperglycemia-induced TGFbeta and fibronectin expression in embryonic mouse heart. Dev Dyn 231:179–189 Stmiskova M, Barancik M, Neckar J, Ravingerova T (2003) Mitogen-activated protein kinases in the acute diabetic myocardium. Mol Cell Biochem 249:59–65 Swift LM, Sarvazyan N (2000) Localization of dichlorofluorescin in cardiac myocytes: implications for assessment of oxidative stress. Am J Physiol Heart Circ Physiol 278:982–990 Tarantini L, Di Lenarda A, Velussi M, Faggiano P, Comaschi M, Faglia E, Maggioni AP (2004) Diabetes mellitus, left ventricular dysfunction and congestive heart failure. Ital Heart J Suppl 5:605–615 Wenzel S, Taimor G, Piper HM, Schluter KD (2001) Redox-sensitive intermediates mediate angiotensin II-induced p38 MAP kinase activation, AP-1 binding activity, and TGF-beta expression in adult ventricular cardiomyocytes. FASEB J 15:2291–2293 Wenzel S, Müller C, Piper HM, Schlüter KD (2005) p38 MAPK in cultured adult rat ventricular cardiomyocytes: expression and involvement in hypertrophic signalling. Euro J Heart Fail 7:453−460 Wilmer WA, Dixon CL, Hebert C (2001) Chronic exposure of human mesangial cells to high glucose environments activates the p38 MAPK pathway. Kidney Int 60:858–871 Wold LE, Ren J (2004) Streptozotocin directly impairs cardiac contractile function in isolated ventricular myocytes via a p38 map kinase-dependent oxidative stress mechanism. Biochem Biophys Res Commun 318:1066–1071 Ye G, Metreveli NS, Donthi RV, Xia S, Xu M, Carlson EC, Epstein PN (2004) Catalase protects cardiomyocyte function in models of type 1 and type 2 diabetes. Diabetes 53:1336–1343