NFAT-Specific Inhibition by dNP2-VIVIT Ameliorates Autoimmune Encephalomyelitis by Regulation of Th1 and Th17

Hong-Gyun Lee1,2, Li-Kyung Kim1,2, Je-Min Choi1,2,3
1Department of Life Science, College of Natural Sciences, Hanyang University, Seoul, Republic of Korea
2Research Institute for Natural Sciences, Hanyang University, Seoul, Republic of Korea
3Research Institute for Convergence of Basic Sciences, Hanyang University, Seoul, Republic of Korea

Tài liệu tham khảo

Shaw, 1988, Identification of a putative regulator of early T cell activation genes, Science, 241, 202, 10.1126/science.3260404

Chen, 1999, Signal integration by transcription-factor assemblies: interactions of NF-AT1 and AP-1 on the IL-2 promoter, Cold Spring Harb. Symp. Quant. Biol., 64, 527, 10.1101/sqb.1999.64.527

Macian, 2005, NFAT proteins: key regulators of T-cell development and function, Nat. Rev. Immunol., 5, 472, 10.1038/nri1632

Hogan, 2010, Molecular basis of calcium signaling in lymphocytes: STIM and ORAI, Annu. Rev. Immunol., 28, 491, 10.1146/annurev.immunol.021908.132550

Feske, 2007, Calcium signalling in lymphocyte activation and disease, Nat. Rev. Immunol., 7, 690, 10.1038/nri2152

Trama, 2000, The NFAT-related protein NFATL1 (TonEBP/NFAT5) is induced upon T cell activation in a calcineurin-dependent manner, J. Immunol., 165, 4884, 10.4049/jimmunol.165.9.4884

Hogan, 2003, Transcriptional regulation by calcium, calcineurin, and NFAT, Genes Dev., 17, 2205, 10.1101/gad.1102703

Kiani, 1997, Down-regulation of IL-4 gene transcription and control of Th2 cell differentiation by a mechanism involving NFAT1, Immunity, 7, 849, 10.1016/S1074-7613(00)80403-3

Rengarajan, 2002, NFATc2 and NFATc3 regulate T(H)2 differentiation and modulate TCR-responsiveness of naïve T(H)cells, Nat. Immunol., 3, 48, 10.1038/ni744

Lee, 2004, A distal enhancer in the interferon-gamma (IFN-gamma) locus revealed by genome sequence comparison, J. Biol. Chem., 279, 4802, 10.1074/jbc.M307904200

Lee, 2018, Revisiting the Concept of Targeting NFAT to Control T Cell Immunity and Autoimmune Diseases, Front. Immunol., 9, 2747, 10.3389/fimmu.2018.02747

Erb, 2003, Mice deficient in nuclear factor of activated T-cell transcription factor c2 mount increased Th2 responses after infection with Nippostrongylus brasiliensis and decreased Th1 responses after mycobacterial infection, Infect. Immun., 71, 6641, 10.1128/IAI.71.11.6641-6647.2003

Fonseca, 2009, NFAT1 transcription factor regulates pulmonary allergic inflammation and airway responsiveness, Am. J. Respir. Cell Mol. Biol., 40, 66, 10.1165/rcmb.2007-0102OC

Kiani, 2001, Regulation of interferon-gamma gene expression by nuclear factor of activated T cells, Blood, 98, 1480, 10.1182/blood.V98.5.1480

Agarwal, 2000, Cell-type-restricted binding of the transcription factor NFAT to a distal IL-4 enhancer in vivo, Immunity, 12, 643, 10.1016/S1074-7613(00)80215-0

Dietz, 2015, NFAT1 deficit and NFAT2 deficit attenuate EAE via different mechanisms, Eur. J. Immunol., 45, 1377, 10.1002/eji.201444638

Gomez-Rodriguez, 2009, Differential expression of interleukin-17A and -17F is coupled to T cell receptor signaling via inducible T cell kinase, Immunity, 31, 587, 10.1016/j.immuni.2009.07.009

Hermann-Kleiter, 2012, Nuclear orphan receptor NR2F6 directly antagonizes NFAT and RORγt binding to the Il17a promoter, J. Autoimmun., 39, 428, 10.1016/j.jaut.2012.07.007

Weigmann, 2008, The transcription factor NFATc2 controls IL-6-dependent T cell activation in experimental colitis, J. Exp. Med., 205, 2099, 10.1084/jem.20072484

Mok, 2017, Calcineurin inhibitors in systemic lupus erythematosus, Best Pract. Res. Clin. Rheumatol., 31, 429, 10.1016/j.berh.2017.09.010

Azzi, 2013, Calcineurin inhibitors: 40 years later, can’t live without …, J. Immunol., 191, 5785, 10.4049/jimmunol.1390055

Fischer, 1989, Cyclophilin and peptidyl-prolyl cis-trans isomerase are probably identical proteins, Nature, 337, 476, 10.1038/337476a0

Takahashi, 1989, Peptidyl-prolyl cis-trans isomerase is the cyclosporin A-binding protein cyclophilin, Nature, 337, 473, 10.1038/337473a0

Harding, 1989, A receptor for the immunosuppressant FK506 is a cis-trans peptidyl-prolyl isomerase, Nature, 341, 758, 10.1038/341758a0

Bram, 1993, Identification of the immunophilins capable of mediating inhibition of signal transduction by cyclosporin A and FK506: roles of calcineurin binding and cellular location, Mol. Cell. Biol., 13, 4760, 10.1128/MCB.13.8.4760

Bechstein, 2000, Neurotoxicity of calcineurin inhibitors: impact and clinical management, Transpl. Int., 13, 313, 10.1111/j.1432-2277.2000.tb01004.x

Abramowicz, 2000, Nephrotoxicity of calcineurin inhibitors: new therapeutic approaches, Transplant. Proc., 32, 3S, 10.1016/S0041-1345(00)00807-1

Miroux, 2012, In vitro effects of cyclosporine A and tacrolimus on regulatory T-cell proliferation and function, Transplantation, 94, 123, 10.1097/TP.0b013e3182590d8f

Miroux, 2009, Inhibitory effects of cyclosporine on human regulatory T cells in vitro, Transplant. Proc., 41, 3371, 10.1016/j.transproceed.2009.08.043

Aramburu, 1999, Affinity-driven peptide selection of an NFAT inhibitor more selective than cyclosporin A, Science, 285, 2129, 10.1126/science.285.5436.2129

Aramburu, 1998, Selective inhibition of NFAT activation by a peptide spanning the calcineurin targeting site of NFAT, Mol. Cell, 1, 627, 10.1016/S1097-2765(00)80063-5

Lim, 2016, Cell Type Preference of a Novel Human Derived Cell-Permeable Peptide dNP2 and TAT in Murine Splenic Immune Cells, PLoS ONE, 11, e0155689, 10.1371/journal.pone.0155689

Lim, 2015, dNP2 is a blood-brain barrier-permeable peptide enabling ctCTLA-4 protein delivery to ameliorate experimental autoimmune encephalomyelitis, Nat. Commun., 6, 8244, 10.1038/ncomms9244

Noguchi, 2004, A new cell-permeable peptide allows successful allogeneic islet transplantation in mice, Nat. Med., 10, 305, 10.1038/nm994

Yamaguchi, 2014, Patient age and the prognosis of idiopathic membranous nephropathy, PLoS ONE, 9, e110376, 10.1371/journal.pone.0110376

Alfaadhel, 2015, Management of Membranous Nephropathy in Western Countries, Kidney Dis. (Basel), 1, 126, 10.1159/000437287

Zhang, 2008, Cyclosporin A inhibits the production of IL-17 by memory Th17 cells from healthy individuals and patients with rheumatoid arthritis, Cytokine, 42, 345, 10.1016/j.cyto.2008.03.006

Wang, 2015, Cyclosporine A Suppresses the Activation of the Th17 Cells in Patients with Primary Sjögren’s Syndrome, Iran. J. Allergy Asthma Immunol., 14, 198

Naniwa, 2017, Mod. Rheumatol., 27, 29, 10.3109/14397595.2016.1174394

Snyder, 1998, Immunophilins in the nervous system, Neuron, 21, 283, 10.1016/S0896-6273(00)80538-3

Chen, 2014, Calcineurin inhibitor induces pain hypersensitivity by potentiating pre- and postsynaptic NMDA receptor activity in spinal cords, J. Physiol., 592, 215, 10.1113/jphysiol.2013.263814

Khanna, 1999, Transforming growth factor (TGF)-beta mimics and anti-TGF-beta antibody abrogates the in vivo effects of cyclosporine: demonstration of a direct role of TGF-beta in immunosuppression and nephrotoxicity of cyclosporine, Transplantation, 67, 882, 10.1097/00007890-199903270-00016

Khanna, 1999, Tacrolimus induces increased expression of transforming growth factor-beta1 in mammalian lymphoid as well as nonlymphoid cells, Transplantation, 67, 614, 10.1097/00007890-199902270-00021

Kakita, 2001, Calcineurin pathway is required for endothelin-1-mediated protection against oxidant stress-induced apoptosis in cardiac myocytes, Circ. Res., 88, 1239, 10.1161/hh1201.091794

Slowinski, 2002, Interaction of the endothelin system and calcineurin inhibitors after kidney transplantation, Clin. Sci. (Lond.), 103, 396S, 10.1042/CS103S396S

Choi, 2012, Cell permeable NFAT inhibitory peptide Sim-2-VIVIT inhibits T-cell activation and alleviates allergic airway inflammation and hyper-responsiveness, Immunol. Lett., 143, 170, 10.1016/j.imlet.2012.01.016

Trapp, 1999, Axonal pathology in multiple sclerosis: relationship to neurologic disability, Curr. Opin. Neurol., 12, 295, 10.1097/00019052-199906000-00008

Goverman, 2009, Autoimmune T cell responses in the central nervous system, Nat. Rev. Immunol., 9, 393, 10.1038/nri2550

Lee, 2019, Pathogenic function of bystander-activated memory-like CD4+ T cells in autoimmune encephalomyelitis, Nat. Commun., 10, 709, 10.1038/s41467-019-08482-w