Investigation into cardiac sympathetic innervation during the commencement of haemodialysis in patients with chronic kidney disease

Springer Science and Business Media LLC - Tập 1 - Trang 1-7 - 2017
Walter Noordzij1, Akin Özyilmaz2,3, Andor W. J. M. Glaudemans1, René A. Tio4, Esther R. Goet2,3, Casper F. M. Franssen2, Riemer H. J. A. Slart1
1Department of Nuclear Medicine and Molecular Imaging, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands
2Division of Nephrology, Department of Internal Medicine, University of Groningen – University Medical Center Groningen, Groningen, the Netherlands
3Dialysis Center Groningen, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands
4Department of Cardiology, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands

Tóm tắt

Patients with chronic kidney disease (CKD) who undergo chronic haemodialysis (HD) show altered sympathetic tone, which is related to a higher cardiovascular mortality. The purpose of this study was to investigate the effect of transition from pre-HD to HD on cardiac sympathetic innervation. Eighteen patients aged 58 ± 18 years (mean ± standard deviation [SD]), 13 males and five females, with stage 5 CKD and nine healthy control subjects aged 52 ± 17 (mean ± SD), three males and six females, were included in this prospective study between May 2010 and December 2013. All patients underwent 123I-labelled meta-iodobenzylguanidine (123I-MIBG) scintigraphy for cardiac sympathetic innervation and electrocardiographically gated adenosine stress and rest 99mTc-labelled tetrofosmin single-photon emission computed tomography for myocardial perfusion imaging prior to (pre-HD) and 6 months after the start of HD. Results of 123I-MIBG scans in patients were compared to controls. Impaired cardiac sympathetic innervation was defined as late heart-to-mediastinum ratio (HMR) < 2.0. Mean late HMR was lower in patients during HD (2.3) than in controls (2.9) (p = 0.035); however, in patients it did not differ between pre-HD and after the start of HD. During HD, two patients showed new sympathetic innervation abnormalities, and in three patients innervation abnormalities seemed to coincide with myocardial perfusion abnormalities. CKD patients show cardiac sympathetic innervation abnormalities, which do not seem to progress during the maintenance HD. The relationship between sympathetic innervation abnormalities and myocardial perfusion abnormalities in HD patients needs further exploration.

Tài liệu tham khảo

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