Interferon-γ: an overview of signals, mechanisms and functions

Journal of Leukocyte Biology - Tập 75 Số 2 - Trang 163-189 - 2004
Kate Schroder1,2, Paul J. Hertzog1,3, Timothy Ravasi1,2, David Hume1,2
1CRC for Chronic Inflammatory Diseases , Parkville, Victoria , Australia
2Institute for Molecular Bioscience, University of Queensland, St Lucia, Brisbane, Australia
3Molecular Genetics Group, Institute of Reproduction and Development, Monash Medical Centre, Monash University , Clayton, Victoria , Australia

Tóm tắt

Abstract

Interferon-γ (IFN-γ) coordinates a diverse array of cellular programs through transcriptional regulation of immunologically relevant genes. This article reviews the current understanding of IFN-γ ligand, receptor, ignal transduction, and cellular effects with a focus on macrophage responses and to a lesser extent, responses from other cell types that influence macrophage function during infection. The current model for IFN-γ signal transduction is discussed, as well as signal regulation and factors conferring signal specificity. Cellular effects of IFN-γ are described, including up-regulation of pathogen recognition, antigen processing and presentation, the antiviral state, inhibition of cellular proliferation and effects on apoptosis, activation of microbicidal effector functions, immunomodulation, and leukocyte trafficking. In addition, integration of signaling and response with other cytokines and pathogen-associated molecular patterns, such as tumor necrosis factor-α, interleukin-4, type I IFNs, and lipopolysaccharide are discussed.

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