Estrogen Receptor (ER)-β Reduces ERα-Regulated Gene Transcription, Supporting a “Ying Yang” Relationship between ERα and ERβ in Mice

The Endocrine Society - Tập 17 Số 2 - Trang 203-208 - 2003
Marie K. Lagerquist1, Sofia Movérare1, Stanko Skrtic1, Hui Gao2, Karin Dahlman‐Wright2, Jan-Ακε Gustafsson2, Claes Ohlsson1
1Division of Endocrinology (M.K.L., S.M., S.S., C.O.), Department of Internal Medicine, Sahlgrenska University Hospital, S-41345 Gothenburg, Sweden
2Department of Medical Nutrition and Department of BioSciences (J.-Å.G., H.G., K.D.-W.), Karolinska Institute, Novum, S-14157 Huddinge, Sweden

Tóm tắt

Abstract

Estrogen is of importance for the regulation of adult bone metabolism. The aim of the present study was to determine the role of estrogen receptor-β (ERβ) in vivo on global estrogen-regulated transcriptional activity in bone. The effect of estrogen in bone of ovariectomized mice was determined using microarray analysis including 9400 genes. Most of the genes (95% = 240 genes) that were increased by estrogen in wild-type (WT) mice were also increased by estrogen in ERβ-inactivated mice. Interestingly, the average stimulatory effect of estrogen on the mRNA levels of these genes was 85% higher in ERβ-inactivated than in WT mice, demonstrating that ERβ reduces estrogen receptor-α (ERα)-regulated gene transcription in bone. The average stimulatory effect of estrogen on estrogen-regulated bone genes in ERα-inactivated mice was intermediate between that seen in WT and ERαβ double-inactivated mice. Thus, ERβ inhibits ERα-mediated gene transcription in the presence of ERα, whereas, in the absence of ERα, it can partially replace ERα. In conclusion, our in vivo data indicate that an important physiological role of ERβ is to modulate ERα-mediated gene transcription supporting a “Ying Yang” relationship between ERα and ERβ in mice.

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