Carcinosarcoma of the breast: two case reports and review of the literature

Kristi M Esses1, Ramona M Hagmaier2, Susan A Blanchard3, John Lazarchick4, Adam I. Riker5
1Department of Pathology, University of South Alabama Medical Center, Mobile, USA
2Mitchell Cancer Institute, University of South Alabama, Mobile, USA
3Diagnostic and Medical Center, Alabama, USA
4Gulf Regional Pathologists, Mobile Infirmary Medical Center, Mobile, USA
5Mitchell Cancer Institute-University of South Alabama, Mobile, USA

Tóm tắt

Abstract

Carcinosarcoma of the breast, often referred to as metaplastic carcinoma of the breast, is a rare malignancy with two distinct cell lines described as a breast carcinoma of ductal type with a sarcoma-like component. Clinically, carcinosarcoma of the breast is an aggressive breast cancer. The prognosis for carcinosarcoma of the breast is less favorable compared to more common types of breast cancer such as infiltrating ductal or lobular carcinoma. Currently, the evaluation of breast carcinoma includes hormone receptor analysis of the tumor tissue, with those positive for estrogen or progesterone responding better to both hormonal and chemotherapy.

Trastuzumab (Herceptin®) is available as an adjunct treatment for tumors which over-express the HER2/neu gene. Typically, metaplastic carcinomas of the breast do not express the estrogen or progesterone receptors and do not over-express the HER2/neu oncogene. As a result of this "triple negative" phenotype, such tumors tend to be more aggressive and are unlikely to respond to targeted therapy with Herceptin. The epidermal growth factor receptor HER-1/EGFR protein is expressed in the majority of metaplastic carcinomas and thus may serve as a potential therapeutic target for EGFR inhibitors such as gefitinib and cetuximab. The two cases we describe exemplify the aggressive nature of carcinosarcoma of the breast and support the findings that this tumor type does not express the common receptors found in other breast carcinomas. These case reports also emphasize the need for investigating the role for blockade of the HER-1/EGFR receptor with targeted therapies when found to be over-expressed in the primary tumor.

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Tài liệu tham khảo

Gutman H, Pollock RE, Janjan NA, Johnson DA: Biologic distinctions and therapeutic implications of sarcomatoid metaplasia of epithelial carcinoma of the breast. J Am Coll Surg. 1995, 180 (2): 193-199.

Foschini MP, Dina RE, Eusebi V: Sarcomatoid neoplasms of the breast: Proposed definitions for biphasic and monophasic sarcomatoid mammary carcinomas. Semin Diagn Pathol. 1993, 10 (2): 128-136.

Rosai J: Special techniques in surgical pathology. Rosai and Ackerman's Surgical Pathology, Mosby. 2004, 1810-1812. Ninth

Wargotz ES, Norris HJ: Metaplastic carcinomas of the breast. II. Spindle Cell Carcinoma. Human Pathology. 1989, 20: 732-740. 10.1016/0046-8177(89)90065-8.

Wargotz ES, Norris HJ: Metaplastic carcinomas of the breast. III. Carcinosarcoma. Cancer. 1989, 67: 1490-1499. 10.1002/1097-0142(19891001)64:7<1490::AID-CNCR2820640722>3.0.CO;2-L.

Rosen , Peter Paul: Carcinoma with metaplasia. Rosen's Breast Pathology. 2009, Philadelphia: Lippincott Williams & Wilkins, 470-474. Third

Beatty JD, Atwood M, Tickman R, Reiner M: Metaplastic breast cancer: clinical significance. The American Journal of Surgery. 2006, 191: 657-664. 10.1016/j.amjsurg.2006.01.038.

Tse GM, Tan PH, Putti TC, Lui PCW, Chaiwun B, Law BKB: Metaplastic carcinoma of the breast: a clinicopathologic review. J Clin Pathol. 2006, 59: 1079-1083. 10.1136/jcp.2005.030536.

Hennessy BT, Giordano S, Broglio K, Duan Z, Trent J, Buchholz , Babiera G, Hortobagyi GN, Valero V: Biphasic metaplastic sarcomatoid carcinoma of the breast. Annals of Oncology. 2006, 17: 605-613. 10.1093/annonc/mdl006.

Tokudome N, Sakamoto G, Sakai T, Sarumaru S, Okuyama N, Hori F, Horii R, Akiyama F, Tanabe M, Saito K, Takahashi K, Kasumi F: A case of carcinosarcoma of the breast. Breast Cancer. 2005, 12 (2): 149-153. 10.2325/jbcs.12.149.

Leibl S, Moinfar F: Metaplastic breast carcinomas are negative for Her-2 but frequently express EGFR (Her-1): potential relevance to adjuvant treatment with EGFR tyrosine kinase inhibitors?. J Clin Pathol. 2005, 58: 700-704. 10.1136/jcp.2004.025163.

Okumo S, Kurebayashi J, Otsuki T, Yamamoto Y, Tanaka K, Sonoo H: Additive antitumor effect of the epidermal growth factor receptor tyrosine kinase inhibitor gefitinib (Iressa, ZD1839) and the antioestrogen fulvestrant (Faslodex, ICI 182,780) in breast cancer cells. Br J Cancer. 2004, 12;90 (1): 236-244.