CRISPR transcript processing: a mechanism for generating a large number of small interfering RNAs

Springer Science and Business Media LLC - Tập 7 - Trang 1-11 - 2012
Marko Djordjevic1, Magdalena Djordjevic2, Konstantin Severinov3,4,5
1Institute of Physiology and Biochemistry, Faculty of Biology, University of Belgrade, Belgrade, Serbia
2Institute of Physics Belgrade, University of Belgrade, Belgrade, Serbia
3Institutes of Molecular Genetics and Gene Biology, Russian Academy of Sciences, Moscow, Russia
4Department of Molecular Biology and Biochemistry, Rutgers, State University of New Jersey, Piscataway, USA
5Waksman Institute, Rutgers, State University of New Jersey, Piscataway, USA

Tóm tắt

CRISPR/Cas (Clustered Regularly Interspaced Short Palindromic Repeats/CRISPR associated sequences) is a recently discovered prokaryotic defense system against foreign DNA, including viruses and plasmids. CRISPR cassette is transcribed as a continuous transcript (pre-crRNA), which is processed by Cas proteins into small RNA molecules (crRNAs) that are responsible for defense against invading viruses. Experiments in E. coli report that overexpression of cas genes generates a large number of crRNAs, from only few pre-crRNAs. We here develop a minimal model of CRISPR processing, which we parameterize based on available experimental data. From the model, we show that the system can generate a large amount of crRNAs, based on only a small decrease in the amount of pre-crRNAs. The relationship between the decrease of pre-crRNAs and the increase of crRNAs corresponds to strong linear amplification. Interestingly, this strong amplification crucially depends on fast non-specific degradation of pre-crRNA by an unidentified nuclease. We show that overexpression of cas genes above a certain level does not result in further increase of crRNA, but that this saturation can be relieved if the rate of CRISPR transcription is increased. We furthermore show that a small increase of CRISPR transcription rate can substantially decrease the extent of cas gene activation necessary to achieve a desired amount of crRNA. The simple mathematical model developed here is able to explain existing experimental observations on CRISPR transcript processing in Escherichia coli. The model shows that a competition between specific pre-crRNA processing and non-specific degradation determines the steady-state levels of crRNA and is responsible for strong linear amplification of crRNAs when cas genes are overexpressed. The model further shows how disappearance of only a few pre-crRNA molecules normally present in the cell can lead to a large (two orders of magnitude) increase of crRNAs upon cas overexpression. A crucial ingredient of this large increase is fast non-specific degradation by an unspecified nuclease, which suggests that a yet unidentified nuclease(s) is a major control element of CRISPR response. Transcriptional regulation may be another important control mechanism, as it can either increase the amount of generated pre-crRNA, or alter the level of cas gene activity. This article was reviewed by Mikhail Gelfand, Eugene Koonin and L Aravind.

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