Biological and cultural sources of familial resemblance in plasma lipids: A comparison between North America and Israel—the lipid research clinics program

Genetic Epidemiology - Tập 5 Số 1 - Trang 17-33 - 1988
Kathleen D. Bucher1, Yechiel Friedlander2,3, Elizabeth Kaplan1, K. K. Namboodiri4, Jeremy D. Kark2, Shlomo Eisenberg3, Y. Stein3, B. M. Rifkind5, D. C. Rao
1Department of Biostatistics, University of North Carolina, Chapel Hill
2Department of Social Medicine, School of Public Health and Community Medicine, Hebrew University-Hadassah Faculty of Medicine, Jerusalem, Israel
3Lipid Research Clinic, Department of Medicine "B," Hadassah University Hospital, Jerusalem, Israel
4Cancer Control Consortium, Ohio State University, Columbus
5Lipid Metabolism–Atherogenesis Branch, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Maryland

Tóm tắt

Abstract

Heterogeneity in determinants of familial resemblance of lipid and lipoprotein levels between populations in North America and Israel was investigated using path analysis. A common protocol, identical measurement techniques, and the same statistical procedures were used in the two samples. Both genetic (h2) and cultural (c2) determinants of inheritance were significant for all lipid variables in the two studies. Genetic and cultural heritability of total cholesterol (h2 = 0.61, c2 = 0.02), low‐density lipoprotein cholesterol (h2 = 0.59, c2 = 0.02), and high‐density lipoprotein cholesterol (h2 = 0.55, c2 = 0.06) did not differ significantly between North America and Israel, while there was a significant difference for triglyceride (h2 = 0.41, c2 = 0.07 in North America; h2 = 0.61, c2 = 0.05 in Israel). Secondary parameters of the path model describing intrafamilial environmental relationships differed between the two countries. In particular, there was a higher correlation between marital environments in Israel for all traits except triglyceride, and a larger effect of father's environment on offspring's environment in Israel for all traits. Within both populations, variation of plasma lipids and lipoproteins was mostly explained by genetic factors and random unmeasured environmental factors. The contribution of common family environment was found to be small, though statistically significant. This is probably due to homogeneity of the distribution of familian environmental determinants within both countries.

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