Association of DLC1 gene polymorphism with susceptibility to hepatocellular carcinoma in Chinese hepatitis B virus carriers
Tài liệu tham khảo
Chen, 1997, Epidemiological characteristics and risk factors of hepatocellular carcinoma, J Gastroenterol Hepatol, 12, S294, 10.1111/j.1440-1746.1997.tb00513.x
Jou, 2004, Clustering of minimal deleted regions reveals distinct genetic pathways of human hepatocellular carcinoma, Cancer Res, 64, 3030, 10.1158/0008-5472.CAN-03-2320
McGlynn, 2001, International trends and patterns of primary liver cancer, Int J Cancer, 94, 290, 10.1002/ijc.1456
Yuan, 1998, Cloning, characterization, and chromosomal localization of a gene frequently deleted in human liver cancer (DLC1) homologous to rat RhoGAP, Cancer Res, 58, 2196
Chinen, 1996, Isolation of 45 exon-like fragments from 8p22→>p21.3, a region that is commonly deleted in hepatocellular, colorectal, and non-small cell lung carcinomas, Cytogenet Cell Genet, 75, 190, 10.1159/000134480
Wong, 2005, Rho GTPase-activating protein deleted in liver cancer suppresses cell proliferation and invasion in hepatocellular carcinoma, Cancer Res, 65, 8861, 10.1158/0008-5472.CAN-05-1318
Wong, 2003, Genetic and epigenetic alterations of dlc1 gene in hepatocellular carcinoma, Cancer Res, 63, 7646
Ng, 2000, DLC1 is deleted in primary hepatocellular carcinoma and exerts inhibitory effects on the proliferation of hepatoma cell lines with deleted DLC1, Cancer Res, 60, 6581
Kim, 2007, DLC1, a GTPase-activating protein for Rho, is associated with cell proliferation, morphology, and migration in human hepatocellular carcinoma, Biochem Biophys Res Commun, 355, 72, 10.1016/j.bbrc.2007.01.121
Sekimata, 1999, Morphological changes and detachment of adherent cells induced by p122, a GTPase-activating protein for Rho, J Biol Chem, 274, 17757, 10.1074/jbc.274.25.17757
Zhou, 2004, Restoration of DLC1 gene expression induces apoptosis and inhibits both cell growth and tumorigenicity in human hepatocellular carcinoma cells, Oncogene, 23, 1308, 10.1038/sj.onc.1207246
Zhai, 2007, Functional polymorphisms in the promoters of MMP-1, MMP-2, MMP-3, MMP-9, MMP-12 and MMP-13 are not associated with hepatocellular carcinoma risk, Gut, 56, 445, 10.1136/gut.2006.112706
Zhou, 2004, Variants in TNFRSF5 locus and association analysis with Hepatitis B virus (HBV) infection, Hum Mutat, 23, 99, 10.1002/humu.9209
Deng, 2004, Association of estrogen receptor alpha polymorphisms with susceptibility to chronic hepatitis B virus infection, Hepatology, 40, 318, 10.1002/hep.20318
Lake, 2003, Estimation and tests of haplotype–environment interaction when linkage phase is ambiguous, Hum Hered, 55, 56, 10.1159/000071811
Lee, 2008, F-SNP: computationally predicted functional SNPs for disease association studies, Nucleic Acids Res, 36, D820, 10.1093/nar/gkm904
Sung, 2008, Genotype-specific genomic markers associated with primary hepatomas, based on complete genomic sequencing of hepatitis B virus, J Virol, 82, 3604, 10.1128/JVI.01197-07
Lee, 2005, Genetic profiling of human hepatocellular carcinoma, Semin Liver Dis, 25, 125, 10.1055/s-2005-871192
Kim, 2005, Single nucleotide polymorphisms associated with hepatocellular carcinoma in patients with chronic hepatitis B virus infection, Intervirology, 48, 10, 10.1159/000082089