Alteration of the micro RNA network during the progression of Alzheimer's disease

EMBO Molecular Medicine - Tập 5 Số 10 - Trang 1613-1634 - 2013
Pierre Lau1,2, Koen Bossers3, Rekin’s Janky4, Evgenia Salta1,2, Carlo Sala Frigerio1,2, Shahar Barbash5, Roy Rothman5, Annerieke Sierksma1,2, Amantha Thathiah1,2, David Greenberg5, Aikaterini S. Papadopoulou1,2, Tilmann Achsel1,2, Torik Ayoubi1,6,2, Hermona Soreq5, Joost Verhaagen3, Dick F. Swaab7, Stein Aerts4, Bart De Strooper1,2
1Center for Human Genetics Leuven Institute for Neurodegenerative Disorders (LIND) University Hospitals Leuven, and University of Leuven O&N4 Herestraat Leuven Belgium
2VIB Center for the Biology of Disease, Leuven, Belgium
3Neurogeneration Group Netherlands Institute for Neuroscience an Institute of the Royal Netherlands Academy of Arts and Sciences Amsterdam The Netherlands
4Laboratory of Computational Biology Center for Human Genetics and University of Leuven O&N4 Herestraat Leuven Belgium
5Department of Biological Chemistry the Silberman Institute of Life Sciences and the Edmond and Lily Safra Center of Brain Science Interdisciplinary Center for Neural Computation Jerusalem Israel
6Saint James School of Medicine, Plaza Juliana, Kralendijk, Bonaire, Dutch Caribbean, The Netherlands
7Neuropsychiatric Disorders Group, Netherlands Institute for Neuroscience, An Institute of the Royal Netherlands Academy of Arts and Sciences, Amsterdam, The Netherlands

Tóm tắt

Từ khóa


Tài liệu tham khảo

Alkalay A, 2013, Plasma acetylcholinesterase activity correlates with intracerebral beta‐amyloid load, Curr Alzheimer Res, 10, 48

10.1101/gr.131573.111

10.1186/gb-2010-11-10-r106

10.1002/emmm.201100995

10.1093/brain/awm276

10.1073/pnas.0308512100

10.1093/brain/awq258

10.1007/BF00308809

10.1371/journal.pone.0006826

10.1073/pnas.1218887109

10.1523/JNEUROSCI.0521-12.2012

10.1101/gad.1884710

10.3233/JAD-2008-14103

10.1016/j.neuron.2010.01.005

10.1371/journal.pone.0010153

10.1073/pnas.0710263105

Hebert SS, 2013, A study of small RNAs from cerebral neocortex of pathology‐verified Alzheimer's disease, dementia with lewy bodies, hippocampal sclerosis, frontotemporal lobar dementia, and non‐demented human controls, J Alzheimers Dis, 35, 335, 10.3233/JAD-122350

10.1016/j.molcel.2010.05.004

10.1093/nar/gks543

10.1016/j.modgep.2005.07.003

10.1002/cne.903010105

10.1016/j.nbd.2007.11.001

10.1523/JNEUROSCI.0600-12.2013

10.1093/brain/awp300

10.1016/j.semcdb.2010.01.009

10.1523/JNEUROSCI.1932-08.2008

10.1371/journal.pone.0040498

10.1371/journal.pone.0019077

10.1073/pnas.1015691107

10.1038/nn.2909

10.1073/pnas.1113793109

10.1016/j.neuron.2010.08.044

10.1371/journal.pone.0008898

10.1007/s00335-001-2044-8

10.1038/nrg3198

10.1042/BJ20100024

10.1016/S1474-4422(11)70286-1

10.1371/journal.pone.0011070

10.1016/j.immuni.2009.09.019

10.1007/s00429-011-0376-z

10.1016/j.neurobiolaging.2011.12.026

10.1093/hmg/ddr330

10.1101/cshperspect.a006296

10.1038/nn.2920

10.1073/pnas.0508448102

10.1093/bioinformatics/bts188

10.1093/nar/gks151

10.1007/s00401-010-0756-0

10.1523/JNEUROSCI.5065-07.2008

10.1016/j.brainresbull.2009.08.006

10.1093/hmg/ddt164

10.1101/gr.118059.110