Molecular Carcinogenesis

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Colorectal carcinoma development in inducible nitric oxide synthase‐deficient mice with dextran sulfate sodium‐induced ulcerative colitis
Molecular Carcinogenesis - Tập 46 Số 5 - Trang 341-353 - 2007
Darren N. Seril, Jie Liao, Guang‐Yu Yang
AbstractThe overproduction of reactive oxygen and nitrogen species (RONS) may play an important role in ulcerative colitis (UC)‐associated carcinogenesis. In order to study the role of nitric oxide (NO) in UC‐associated colorectal carcinogenesis, the development of colorectal carcinoma was studied using the DSS‐induced and iron‐enhanced model of chronic UC in induc...... hiện toàn bộ
Peroxisome proliferator–activated receptor α in the human breast cancer cell lines MCF‐7 and MDA‐MB‐231
Molecular Carcinogenesis - Tập 34 Số 4 - Trang 165-171 - 2002
K. M. Suchanek, Fiona J. May, J. A. Robinson, Won‐Jae Lee, N. A. Holman, Gregory R. Monteith, Sarah J. Roberts‐Thomson
AbstractPeroxisome proliferator–activated receptor (PPAR) α is a ligand‐activated transcription factor that has been linked with rodent hepatocarcinogenesis. It has been suggested that PPARα mRNA expression levels are an important determinant of rodent hepatic tumorigenicity. Previous work in rat mammary gland epithelial cells showed sign...... hiện toàn bộ
Diallyl trisulfide‐induced apoptosis in human cancer cells is linked to checkpoint kinase 1‐mediated mitotic arrest
Molecular Carcinogenesis - Tập 48 Số 11 - Trang 1018-1029 - 2009
Dong Xiao, Yan Zeng, Shivendra V. Singh
AbstractGrowth suppressive effect of diallyl trisulfide (DATS), a promising cancer chemopreventive constituent of garlic, against cultured human cancer cells correlates with checkpoint kinase 1 (Chk1)‐mediated mitotic arrest, but the fate of the cells arrested in mitosis remains elusive. Using LNCaP and HCT‐116 human cancer cells as a model, we now demonstrate that...... hiện toàn bộ
Ki‐ras oncogene mutations in tumors and DNA adducts formed by benz[j]aceanthrylene and benzo[a]pyrene in the lungs of strain A/J mice
Molecular Carcinogenesis - Tập 8 Số 3 - Trang 186-192 - 1993
Marc J. Mass, Jeffrey A. Ross, Stephen Nesnow, Anita J. Jeffers, Garret B. Nelson, Anthony J. Galati, Gary D. Stoner
AbstractStrain A/J mice received intraperitoneal injections of benz[j]aceanthrylene (B[j]A) or benzo[a]pyrene (B[a]P). At 24, 48, and 72 h, lung tissues were removed for analysis of B[a]P‐ or B[j]A‐derived...... hiện toàn bộ
Suppression of radiation‐induced neoplastic transformation by overexpression of mitochondrial superoxide dismutase
Molecular Carcinogenesis - Tập 6 Số 4 - Trang 238-242 - 1992
Daret K. St. Clair, X. Steven Wan, Terry D. Oberley, Kenneth E. Muse, William H. St. Clair
AbstractManganese superoxide dismutase (MnSOD) scavenges toxic superoxide radicals produced in the mitochondria. Transfection of the human MnSOD gene into mouse C3H 10T1/2 cells resulted in production of active MnSOD, which was properly transported into mitochondria. Overexpression of MnSOD protected cells from radiation‐, but not chemically‐induced neoplastic tran...... hiện toàn bộ
BRCA1 and acetyl‐CoA carboxylase: The metabolic syndrome of breast cancer
Molecular Carcinogenesis - Tập 47 Số 2 - Trang 157-163 - 2008
Joan Brunet, Alejandro Vázquez‐Martín, Rámón Colomer, Begoña Graña-Suárez, Begoña Martı́n-Castillo, Javier A. Menéndez
AbstractBreast cancer‐associated mutations affecting the highly‐conserved C‐terminal BRCT domains of the tumor suppressor gene breast cancer susceptibility gene 1 (BRCA1) fully disrupt the ability of BRCA1 to interact with acetyl co...... hiện toàn bộ
Pharmacological inhibition of fatty acid synthase (FAS): A novel therapeutic approach for breast cancer chemoprevention through its ability to suppress Her‐2/neu (erbB‐2) oncogene‐induced malignant transformation
Molecular Carcinogenesis - Tập 41 Số 3 - Trang 164-178 - 2004
Javier A. Menéndez, Inderjit Mehmi, Vishal Verma, Poh K. Teng, Ruth Lupu
AbstractWe designed our experiments to evaluate whether fatty acid synthase (FAS), a lipogenic enzyme linked to tumor virulence in population studies of human cancer, is necessary for the malignant transformation induced by Her‐2/neu (erbB‐2) oncogene, which is overexpressed not only in invasive breast cancer but also in premalignant atypical duct proliferations an...... hiện toàn bộ
KAI1, A prostate metastasis suppressor: Prediction of solvated structure and interactions with binding partners; integrins, cadherins, and cell‐surface receptor proteins†
Molecular Carcinogenesis - Tập 32 Số 3 - Trang 139-153 - 2001
Rachelle J. Bienstock, J. Carl Barrett
AbstractThe solution structure of the transmembrane‐4 superfamily protein KAI1, a recently identified prostate cancer metastasis suppressor gene that encodes a 267–amino acid protein, was modeled. The structure of this four‐helical transmembrane protein was developed by defining and modeling sections individually. A complete three‐dimensional structure for the solv...... hiện toàn bộ
Lysosomal destabilization and cathepsin B contributes for cytochrome c release and caspase activation in embelin‐induced apoptosis
Molecular Carcinogenesis - Tập 49 Số 4 - Trang 324-336 - 2010
Beena Joy, Rajeeve Sivadasan, Abraham T. Emilia, Mohan John, Praveen K. Sobhan, Mahendra Seervi, Santhoshkumar T.R.
AbstractXIAP is an important antiapoptotic protein capable of conferring resistance to cancer cells. Embelin, the small molecular inhibitor of XIAP, possesses wide spectrum of biological activities with strong inhibition of nuclear factor kappa B and downstream antiapoptotic genes. However, the mechanism of its cell death induction is not known. Our studies using c...... hiện toàn bộ
Allelic loss at the tuberous sclerosis 2 locus in spontaneous tumors in the Eker rat
Molecular Carcinogenesis - Tập 14 Số 1 - Trang 28-36 - 1995
Raymond S. Yeung, Guang‐Hui Xiao, Jeffrey I. Everitt, Fang Jin, Cheryl Lyn Walker
AbstractSomatic events leading to the inactivation of tumor suppressor genes often involve chromosomal alterations that can be detected as loss of heterozygosity(a). In the Eker rat, spontaneous tumors of the kidney, uterus, and spleen develop as a result of a germline mutation of the tuberous sclerosis 2(Tsc2) gene. We examined the patte...... hiện toàn bộ
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