Gut

Công bố khoa học tiêu biểu

* Dữ liệu chỉ mang tính chất tham khảo

Sắp xếp:  
Inheritance of type 2 Crigler-Najjar hyperbilirubinaemia
Gut - Tập 14 Số 1 - Trang 46-49 - 1973
John O. Hunter, R. P. H. Thompson, P M Dunn, Rebecca Williams
Taste receptors of the gut: emerging roles in health and disease
Gut - Tập 63 Số 1 - Trang 179-190 - 2014
Inge Depoortere
Switchable CAR-T cells mediate remission in metastatic pancreatic ductal adenocarcinoma
Gut - Tập 68 Số 6 - Trang 1052-1064 - 2019
Deepak B. Thimiri Govinda Raj, Ming-Hsin Yang, David T. Rodgers, Eric Hampton, Julfa Begum, A. F. Mustafa, Daniela Lorizio, Irene Garces, David Propper, James G. Kench, Hemant M. Kocher, Travis S. Young, Alexandra Aicher, Christopher Heeschen
ObjectivePancreatic ductal adenocarcinoma (PDAC) is a disease of unmet medical need. While immunotherapy with chimeric antigen receptor T (CAR-T) cells has shown much promise in haematological malignancies, their efficacy for solid tumours is challenged by the lack of tumour-specific antigens required to avoid on-target, off-tumour effects. Switchable CAR-T cells whereby activity of the CAR-T cell is controlled by dosage of a tumour antigen-specific recombinant Fab-based ‘switch’ to afford a fully tunable response may overcome this translational barrier.DesignIn this present study, we have used conventional and switchable CAR-T cells to target the antigen HER2, which is upregulated on tumour cells, but also present at low levels on normal human tissue. We used patient-derived xenograft models derived from patients with stage IV PDAC that mimic the most aggressive features of PDAC, including severe liver and lung metastases.ResultsSwitchable CAR-T cells followed by administration of the switch directed against human epidermal growth factor receptor 2 (HER2)-induced complete remission in difficult-to-treat, patient-derived advanced pancreatic tumour models. Switchable HER2 CAR-T cells were as effective as conventional HER2 CAR-T cells in vivo testing a range of different CAR-T cell doses.ConclusionThese results suggest that a switchable CAR-T system is efficacious against aggressive and disseminated tumours derived from patients with advanced PDAC while affording the potential safety of a control switch.
PROSTAGLANDINS AND CANCER
Gut - Tập 55 Số 1 - Trang 115-122 - 2006
Diandian Wang
Generation of gaseous sulfur-containing compounds in tumour tissue and suppression of gas diffusion as an antitumour treatment
Gut - Tập 61 Số 4 - Trang 554-561 - 2012
Kazue Yamagishi, Kazuo Onuma, Yota Chiba, Shinya Yagi, Shigenobu Aoki, Tomoyuki Sato, Yasushi Sugawara, Noriyasu Hosoya, Yasutake Saeki, Minoru Takahashi, Masayoshi Fuji, Takeo Ohsaka, Takeyoshi Okajima, Kenji Akita, Takashi Suzuki, Pisol Senawongse, Akio Urushiyama, Kiyohiko Kawai, Hirofumi Shoun, Yoshimasa Ishii, Hiroya Ishikawa, Shigeru Sugiyama, Madoka Nakajima, Masaru Tsuboi, Tateo Yamanaka
Functional polymorphisms in the promoters of MMP-1, MMP-2, MMP-3, MMP-9, MMP-12 and MMP-13 are not associated with hepatocellular carcinoma risk
Gut - Tập 56 Số 3 - Trang 445-447 - 2007
Yao Zhai, Wei Qiu, Xiaokang Dong, X.-M. Zhang, Weimin Xie, H.-X. Zhang, Xiangliang Yuan, Gangqiao Zhou, Fuchu He
Gut microbiota-derived metabolites as central regulators in metabolic disorders
Gut - Tập 70 Số 6 - Trang 1174-1182 - 2021
Allison Agus, Karine Clément, Harry Sokol
Metabolic disorders represent a growing worldwide health challenge due to their dramatically increasing prevalence. The gut microbiota is a crucial actor that can interact with the host by the production of a diverse reservoir of metabolites, from exogenous dietary substrates or endogenous host compounds. Metabolic disorders are associated with alterations in the composition and function of the gut microbiota. Specific classes of microbiota-derived metabolites, notably bile acids, short-chain fatty acids, branched-chain amino acids, trimethylamine N-oxide, tryptophan and indole derivatives, have been implicated in the pathogenesis of metabolic disorders. This review aims to define the key classes of microbiota-derived metabolites that are altered in metabolic diseases and their role in pathogenesis. They represent potential biomarkers for early diagnosis and prognosis as well as promising targets for the development of novel therapeutic tools for metabolic disorders.
Choice of management strategy for colorectal cancer based on a diagnostic immunohistochemical test for defective mismatch repair
Gut - Tập 45 Số 3 - Trang 409-415 - 1999
Lynn Cawkwell, Sally Gray, Harry Murgatroyd, Francis Sutherland, Louise Haine, M Longfellow, Samantha M. O’Loughlin, D Cross, O Kronbörg, C. Fenger, N Mapstone, M F Dixon, Philip Quirke
BACKGROUNDDespite intensive research into the molecular abnormalities associated with colorectal cancer (CRC), no diagnostic tests have emerged which usefully complement standard histopathological assessments.AIMSTo assess the feasibility of using immunohistochemistry to detect replication error (RER) positive CRCs and determine the incidence of RER positivity within distinct patient subgroups.METHODS502 CRCs were analysed for RER positivity (at least two markers affected) and/or expression of hMSH2 and hMLH1.RESULTSThere were 15/30 (50%) patients with metachronous CRCs, 16/51 (31%) with synchronous CRCs, 14/45 (31%) with a proximal colon carcinoma, and 4/23 (17%) who developed a CRC under the age of 50 showed RER positivity. However, 0/54 patients who developed a solitary carcinoma of the rectum/left colon over the age of 50 showed RER positivity. Immunohistochemical analysis revealed that 66/66 (100%) RER positive carcinomas were associated with complete lack of expression of either hMSH2 or hMLH1. This correlation was confirmed using a further 101 proximal colon carcinomas. Patients with a mismatch repair defective carcinoma showed improved survival but a 5.54 times relative risk of developing a metachronous CRC. A prospective immunohistochemical study revealed 13/117 (11%) patients had a mismatch repair defective carcinoma. A fivefold excess of hMLH1 defective cases was noted.CONCLUSIONSAll RER positive carcinomas were identified by the immunohistochemical test. This is the first simple laboratory test which can be performed routinely on all CRCs. It will provide a method for selecting patients who should be investigated for HNPCC, offered long term follow up, and who may not respond to standard chemotherapy regimens.
Is gastric cancer becoming a rare disease? A global assessment of predicted incidence trends to 2035
Gut - Tập 69 Số 5 - Trang 823-829 - 2020
Melina Arnold, Jin Young Park, M. Constanza Camargo, Nuno Lunet, David Forman, Isabelle Soerjomataram
ObjectivesThe incidence of gastric cancer continues to decrease globally, approaching levels that in some populations could define it as a rare disease. To explore this on a wider scale, we predict its future burden in 34 countries with long-standing population-based data.MethodsData on gastric cancer incidence by year of diagnosis, sex and age were extracted for 92 cancer registries in 34 countries included inCancer Incidence in Five ContinentsPlus. Numbers of new cases and age-standardised incidence rates (ASR per 100 000) were predicted up to 2035 by fitting and extrapolating age–period–cohort models.ResultsOverall gastric cancer incidence rates are predicted to continue falling in the future in the majority of countries, including high-incidence countries such as Japan (ASR 36 in 2010 vs ASR 30 in 2035) but also low-incidence countries such as Australia (ASR 5.1 in 2010 vs ASR 4.6 in 2035). A total of 16 countries are predicted to fall below the rare disease threshold (defined as 6 per 100 000 person-years) by 2035, while the number of newly diagnosed cases remains high and is predicted to continue growing. In contrast, incidence increases were seen in younger age groups (below age 50 years) in both low-incidence and high-incidence populations.ConclusionsWhile gastric cancer is predicted to become a rare disease in a growing number of countries, incidence levels remain high in some regions, and increasing risks have been observed in younger generations. The predicted growing number of new cases highlights that gastric cancer remains a major challenge to public health on a global scale.
Inhibition of RAF1 kinase activity restores apicobasal polarity and impairs tumour growth in human colorectal cancer
Gut - Tập 66 Số 6 - Trang 1106-1115 - 2017
Tijana Borovski, Thomas T. Vellinga, Jamila Laoukili, Evan E. Santo, Szabolcs Fátrai, Susanne van Schelven, André Verheem, Dieuwke L. Marvin, Inge Ubink, Inne H.M. Borel Rinkes, Onno Kranenburg
Tổng số: 359   
  • 1
  • 2
  • 3
  • 4
  • 5
  • 6
  • 10