Transgenic mice expressing mutant A53T human alpha-synuclein show neuronal dysfunction in the absence of aggregate formation

Molecular and Cellular Neurosciences - Tập 24 - Trang 419-429 - 2003
Suzana Gispert1,2, Domenico Del Turco3, Lisa Garrett2, Amy Chen2, David J Bernard2, John Hamm-Clement3, Horst-Werner Korf1, Thomas Deller3, Heiko Braak3, Georg Auburger2,4, Robert L Nussbaum2
1Department of Anatomy II (Experimental Neurobiology), University Frankfurt/M, Frankfurt, Germany
2National Human Genome Research Institute, NIH, Bethesda, MD, USA
3Department of Anatomy I (Clinical Neuroanatomy), University Frankfurt/M, Frankfurt, Germany
4Section Molecular Neurogenetics, Bldg 26, University Hospital, Frankfurt/M, Frankfurt, Germany

Thông tin
Thông tin xuất bản

Molecular and Cellular Neurosciences

Tập 24

419-429

Thông tin tác giả