Du, 2016, Follicular fluid and urinary concentrations of phthalate metabolites among infertile women and associations with in vitro fertilization parameters, Reprod Toxicol, 61, 142, 10.1016/j.reprotox.2016.04.005
Czubacka, 2021, The overview of current evidence on the reproductive toxicity of dibutyl phthalate, Int J Occup Med Environ Health, 34, 15, 10.13075/ijomeh.1896.01658
Blount, 2000, Levels of seven urinary phthalate metabolites in a human reference population, Environ Health Perspect, 108, 979, 10.1289/ehp.00108979
Guo, 2011, Phthalate metabolites in urine from China, and implications for human exposures, Environ Int, 37, 893, 10.1016/j.envint.2011.03.005
Hines, 2011, Estimated daily intake of phthalates in occupationally exposed groups, J Expo Sci Environ Epidemiol, 21, 133, 10.1038/jes.2009.62
Manikkam, 2013, Plastics derived endocrine disruptors (BPA, DEHP and DBP) induce epigenetic transgenerational inheritance of obesity, reproductive disease and sperm epimutations, PLoS One, 8, 10.1371/journal.pone.0055387
Barazzoni, 2018, Insulin resistance in obesity: an overview of fundamental alterations, Eat Weight Disord, 23, 149, 10.1007/s40519-018-0481-6
Radke, 2019, Phthalate exposure and metabolic effects: a systematic review of the human epidemiological evidence, Environ Int, 132, 10.1016/j.envint.2019.04.040
Li, 2017, PTEN, insulin resistance and cancer, Curr Pharmaceut Des, 23, 3667, 10.2174/1381612823666170704124611
Hu, 2018, Bisphenol A initiates excessive premature activation of primordial follicles in mouse ovaries via the PTEN signaling pathway, Reprod Sci, 25, 609, 10.1177/1933719117734700
Deng, 2018, Dibutyl phthalate exposure aggravates type 2 diabetes by disrupting the insulin-mediated PI3K/AKT signaling pathway, Toxicol Lett, 290, 1, 10.1016/j.toxlet.2018.03.004
So, 2020, Relation between HOMA-IR and insulin sensitivity index determined by hyperinsulinemic-euglycemic clamp analysis during treatment with a sodium-glucose cotransporter 2 inhibitor, Endocr J, 67, 501, 10.1507/endocrj.EJ19-0445
Li, 2020, Intrauterine exposure to low-dose DBP in the mice induces obesity in offspring via suppression of UCP1 mediated ER stress, Sci Rep, 10, 16360, 10.1038/s41598-020-73477-3
Xiong, 2020, Exposure to dibutyl phthalate impairs lipid metabolism and causes inflammation via disturbing microbiota-related gut-liver axis, Acta Biochim Biophys Sin, 52, 1382, 10.1093/abbs/gmaa128
Majeed, 2017, Sub-chronic exposure to low concentration of dibutyl phthalate affects anthropometric parameters and markers of obesity in rats, Environ Sci Pollut Res Int, 24, 25462, 10.1007/s11356-017-9952-y
Chen, 2020, Dibutyl phthalate contamination accelerates the uptake and metabolism of sugars by microbes in black soil, Environ Pollut, 262, 10.1016/j.envpol.2020.114332
Williams, 2016, Dibutyl phthalate exposure disrupts evolutionarily conserved insulin and glucagon-like signaling in drosophila males, Endocrinology, 157, 2309, 10.1210/en.2015-2006
Li, 2021, Typical phthalic acid esters induce apoptosis by regulating the PI3K/Akt/Bcl-2 signaling pathway in rat insulinoma cells, Ecotoxicol Environ Saf, 208, 10.1016/j.ecoenv.2020.111461
Wang, 2017, Role of PI3K/AKT/mTOR signaling pathway in DBP-induced apoptosis of testicular sertoli cells in vitro, Environ Toxicol Pharm, 53, 145, 10.1016/j.etap.2017.05.013
Euling, 2013, Use of genomic data in risk assessment case study: II. Evaluation of the dibutyl phthalate toxicogenomic data set, Toxicol Appl Pharm, 271, 349, 10.1016/j.taap.2011.06.014
Neier, 2020, Short- and long-term effects of perinatal phthalate exposures on metabolic pathways in the mouse liver, Environ Epigenet, 6