Sympathomimetische Effekte auch bei niedriger Dosierung von Esketamin

Adams HA (1997) Endokrine Reaktionen nach S(+)-Ketamin. Anaesthesist 46 [Suppl 1]: 30–37

Adams HA, Bauer E, Gebhardt B et al. (1994) TIVA mit S-(+)-Ketamin in der orthopädischen Alterschirurgie. Anaesthesist 43: 92–100

Adams HA, Thiel A, Jung A et al. (1992) Untersuchungen mit S-(+)-Ketamin an Probanden. Endokrine- und Kreislaufreaktionen, Aufwachverhalten und Traumerlebnisse. Anaesthesist 41: 588–596

Böttiger BW, Martin E (2000) Prävention perioperativer Myokardischämien – ein Update. Anaesthesist 49: 174–186

Burton AW, Lee DH, Saab C, Chung JM (1999) Preemptive intrathecal ketamine injection produces a long-lasting decrease in neuropathic pain behaviors in a rat model. Reg Anesth Pain Med 24: 208–213

Craven R (2007) Ketamine. Anaesthesia 62: 48–53

Charlson ME, MacKenzie CR, Gold JP et al. (1989) The preoperative and intraoperative hemodynamic predictors of postoperative myocardial infarction or ischemia in patients undergoing noncardiac surgery. Ann Surg 210: 637–648

Charlson ME, MacKenzie CR, Gold JP et al. (1990) Intraoperative blood pressure. What patterns identify patients at risk for postoperative complications? Ann Surg 212: 567–580

Crozier TA, Sumpf E (1996) Der Einfluss einer totalen intravenösen Anaesthesie mit S-(+)-Ketamin/Propofol auf hämodynamische, endokrine und metabolische Streßreaktionen im Vergleich zu Alfentanil/Propofol bei Laparotomien. Anaesthesist 45: 1015–1023

Davies SN, Lodge D (1987) Evidence for involvement of N-methyl-aspartate receptors in“wind up” of class 2 neurones in the dorsal horn of the rat. Brain Res 424: 402–406

Deedwania PC, Nelson JR (1990) Pathophysiology of silent myocardial ischemia during daily life. Hemodynamic evaluation by simultaneous electrocardiographic and blood pressure monitoring. Circulation 82: 1296–1304

Dertwinkel R, Heinrichs C, Senne I et al. (2002) Prevention of severe phantom limb pain by perioperative administration of ketamine – an observational study. Acute Pain 4: 9–13

Doenicke A, Angster R, Mayer M et al. (1992) Die Wirkung von S-(+)-Ketamin auf Katecholamine und Cortisol im Serum, Vergleich zu Ketamin-Razemat. Anaesthesist 41: 597–603

Hoeft A, Sonntag H, Stephan H, Kettler D (1991) Validation of myocardial oxygen demand indices in patients awake and during anesthesia. Anesthesiology 75: 49–56

Ivankovich AD, Miletich DJ, Reimann C et al. (1974) Cardiovascular effects of centrally administered ketamine in goats. Anesth Analg 53: 924–933

Kal JE, Wezel HB van, Vergroesen I (1999) A critical appraisal of the rate pressure product as index of myocardial oxygen consumption for the study of metabolic coronary flow regulation. Int J Cardiol 71: 141–148

Kettler D (1973) Sauerstoffbedarf und Sauerstoffversorgung des Herzens in Narkose. Springer, Berlin Heidelberg New York Tokio

Lundy PM, Lockwood PA, Thompson G, Frew R (1986) Differential effects of ketamine isomers on neuronal and extraneuronal catecholamine uptake mechanisms. Anesthesiology 64: 359–363

Miletich DJ, Ivankovic AD, Albrecht RF et al. (1973) The effect of ketamine on catecholamine metabolism in the isolated perfused rat heart. Anesthesiology 39: 271–277

Nikolajsen L, Hansen CL, Nielsen J et al. (1996) The effect of ketamine on phantom pain: a central neuropathic disorder maintained by peripheral input. Pain 67: 69–77

Nikolajsen L, Hansen PO, Jensen TS (1997) Oral ketamine therapy in the treatment of postamputation stump pain. Acta Anaesthesiol Scand 41: 427–429

Patschke D (1976) Koronardurchblutung und myokardialer Sauerstoffverbrauch während Narkoseeinleitung. Anaesthesiologie und Wiederbelebung, Bd 96. Springer, Berlin Heidelberg New York Tokio

Pfenninger EG, Durieux ME, Himmelseher S (2002) Cognitive impairment after small-dose ketamine isomers in comparison to equianalgetic racemic ketamine in human volunteers. Anesthesiology 96: 357–366

Robinson BF (1976) Relation of heart rate and systolic blood pressure to the onset of pain in angina pectoris. Circulation 35: 1073–1083

Stubhaug A, Breivik H, Eide PK et al. (1997) Mapping of punctuate hyperalgesia around a surgical incision demonstrates that ketamine is a powerful suppressor of centralsensitization to pain following surgery. Acta Anaesthesiol Scand 41: 1124–1132

Takki S, Nikki P, Jaattela A, Tammisto T (1972) Ketamine and plasma catecholamines. Br J Anaesth 44: 1318–1322

Tarnow J, Heß W, Schmidt D, Eberlein HJ (1979) Narkoseeinleitung bei Patienten mit koronarer Herzkrankheit: Flunitrazepam, Diazepam, Ketamin, Fentanyl. Anaesthesist 28: 9–19

Thiemicke J, Doenicke A, Mückter H et al. (2002) Der Einfluss von S(+)-Ketamin auf die endokrine Reaktion und das Kreislaufverhalten während totaler intravenöser Anästhesie mit Etomidat und Propofol. Anaesthesiol Intensivmed 43: 584–592

Treede RD, Meyer RA, Raja SN, Campbell JN (1992) Peripheral and central mechanisms of cutaneous hyperalgesia. Prog Neurobiol 38: 397–421

Weinbroum AA (2003) A single small dose of postoperative ketamine provides rapid and sustained improvement in morphine analgesia in the presence of morphine-resistant pain. Anesth Analg 96: 789–795

White WB (1999) Heart rate and the rate-pressure product as determinants of cardiovascular risk in patients with hypertension. Am J Hypertens 12: 50–55

Woolf CJ, Thompson SWN (1991) The induction and maintenance of central sensitization is dependent on N-methyl-D-aspartic acid receptor activation; implications for the treatment of post-injury pain hypersensitivity states. Pain 44: 293–299

Zielmann S, Kazmaier S, Schnüll S, Weyland A (1997) S-(+)-Ketamin und Kreislauf. Anaesthesist 46: 43–46