Resident CD141 (BDCA3)+ dendritic cells in human skin produce IL-10 and induce regulatory T cells that suppress skin inflammation

Journal of Experimental Medicine - Tập 209 Số 5 - Trang 935-945 - 2012
Chung-Ching Chu1,2,3,4, Niwa Ali1,2,4,5, Panagiotis Karagiannis1,2,4, Paola Di Meglio1,2,4, Ania Skowera1,2,4, L Napolitano1,2,4, G. Barinaga1,2,4, Katarzyna Grys1,2,4, Ehsan Sharif‐Paghaleh1,2,4, Sophia N. Karagiannis1,2,4, Mark Peakman1,2,4, Giovanna Lombardi1,2,4, Frank O. Nestlé1,2,4
1Department of Immunology, King’s College London and National Institutes for Health Research Biomedical Research Centre, SE1 9RT London, UK 1 , 2 , and 3
2MRC Centre for Transplantation 1 , 2 , and 3
3Shanghai Institute of Immunology, Institute of Medical Sciences, Shanghai Jiao Tong University School of Medicine, 200025 Shanghai, China 4
4St. John's Institute of Dermatology, MRC Centre for Transplantation, and Department of Immunology, King's College London and National Institutes for Health Research Biomedical Research Centre, SE1 9RT London, UK
5St. John’s Institute of Dermatology 1 , 2 , and 3

Tóm tắt

Human skin immune homeostasis, and its regulation by specialized subsets of tissue-residing immune sentinels, is poorly understood. In this study, we identify an immunoregulatory tissue-resident dendritic cell (DC) in the dermis of human skin that is characterized by surface expression of CD141, CD14, and constitutive IL-10 secretion (CD141+ DDCs). CD141+ DDCs possess lymph node migratory capacity, induce T cell hyporesponsiveness, cross-present self-antigens to autoreactive T cells, and induce potent regulatory T cells that inhibit skin inflammation. Vitamin D3 (VitD3) promotes certain phenotypic and functional properties of tissue-resident CD141+ DDCs from human blood DCs. These CD141+ DDC-like cells can be generated in vitro and, once transferred in vivo, have the capacity to inhibit xeno-graft versus host disease and tumor alloimmunity. These findings suggest that CD141+ DDCs play an essential role in the maintenance of skin homeostasis and in the regulation of both systemic and tumor alloimmunity. Finally, VitD3-induced CD141+ DDC-like cells have potential clinical use for their capacity to induce immune tolerance.

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