Reduced Mycobacterium tuberculosis association with monocytes from diabetes patients that have poor glucose control

World Health Organization, 2011

International Diabetes Federation, 2009

Restrepo, 2011, Cross-sectional assessment reveals high diabetes prevalence among newly-diagnosed tuberculosis patients, Bull World Health Organ, 89, 352, 10.2471/BLT.10.085738

Jeon, 2008, Diabetes mellitus increases the risk of active tuberculosis: a systematic review of 13 observational studies, PLoS Med, 5, 1091

2005, American Thoracic Society, Infectious Diseases Society of America, Centers for Disease Control and Prevention. Controlling tuberculosis in the United States, Am J Respir Crit Care Med, 172, 1169, 10.1164/rccm.2508001

Stalenhoef, 2008, The role of interferon-gamma in the increased tuberculosis risk in type 2 diabetes mellitus, Eur J Clin Microbiol Infect Dis, 27, 97, 10.1007/s10096-007-0395-0

Gonzalez-Curiel, 2011, Differential expression of antimicrobial peptides in active and latent tuberculosis and its relationship with diabetes mellitus, Hum Immunol, 72, 656, 10.1016/j.humimm.2011.03.027

Martens, 2007, Tuberculosis susceptibility of diabetic mice, Am J Respir Cell Mol Biol, 37, 518, 10.1165/rcmb.2006-0478OC

Vallerskog, 2010, Diabetic mice display a delayed adaptive immune response to Mycobacterium tuberculosis, J Immunol, 184, 6275, 10.4049/jimmunol.1000304

Komura, 2010, CD14+ monocytes are vulnerable and functionally impaired under endoplasmic reticulum stress in patients with type 2 diabetes, Diabetes, 59, 634, 10.2337/db09-0659

Ding, 2007, Activation of RAGE induces elevated O2-generation by mononuclear phagocytes in diabetes, J Leukoc Biol, 81, 520, 10.1189/jlb.0406262

Tan, 2012, Glutathione deficiency in type 2 diabetes impairs cytokine responses and control of intracellular bacteria, J Clin Invest, 122, 2289, 10.1172/JCI57817

Glass, 1987, Impairment of monocyte “lectin-like” receptor activity in type 1 (insulin-dependent) diabetic patients, Diabetologia, 30, 228, 10.1007/BF00270420

Hill, 1983, Defective monocyte chemotactic responses in diabetes mellitus, J Clin Immunol, 3, 70, 10.1007/BF00919141

Katz, 1983, Phagocytotic activity of monocytes from diabetic patients, Diabetes Care, 6, 479, 10.2337/diacare.6.5.479

Chang, 1995, Decreased cell-mediated immunity in patients with non-insulin-dependent diabetes mellitus, Diabetes Res Clin Pract, 28, 137, 10.1016/0168-8227(95)00168-8

Mathews, 2012, Impaired function of antibodies to pneumococcal surface protein A and not to capsular polysaccharide in Mexican American adults with type 2 diabetes mellitus, Clin Vaccine Immunol, 19, 1360, 10.1128/CVI.00268-12

Noritake, 1992, Intracellular hydrogen peroxide production by peripheral phagocytes from diabetic patients. Dissociation between polymorphonuclear leucocytes and monocytes, Clin Exp Immunol, 88, 269, 10.1111/j.1365-2249.1992.tb03072.x

Schlesinger, 1996, Entry of Mycobacterium tuberculosis into mononuclear phagocytes, Curr Top Microbiol Immunol, 215, 71

Schlesinger, 1998, Mycobacterium tuberculosis and the complement system, Trends Microbiol, 6, 47, 10.1016/S0966-842X(97)01203-1

Schlesinger, 1990, Phagocytosis of Mycobacterium tuberculosis is mediated by human monocyte complement receptors and complement component C3, J Immunol, 144, 2771, 10.4049/jimmunol.144.7.2771

Bardana, 1973, Universal occurrence of antibodies to tubercle bacilli in sera from non-tuberculous and tuberculous individuals, Clin Exp Immunol, 13, 65

Geisler, 1982, Monocyte functions in diabetes mellitus, Acta Pathol Microbiol Immunol Scand C, 90, 33

American Diabetes Association, 2010, Standards of medical care in diabetes – 2010, Diabetes Care, 33, S11, 10.2337/dc10-S011

Schlesinger, 1993, Macrophage phagocytosis of virulent but not attenuated strains of Mycobacterium tuberculosis is mediated by mannose receptors in addition to complement receptors, J Immunol, 150, 2920, 10.4049/jimmunol.150.7.2920

Zhang, 1998, Growth of virulent and avirulent Mycobacterium tuberculosis strains in human macrophages, Infect Immun, 66, 794, 10.1128/IAI.66.2.794-799.1998

Ferguson, 2004, Complement protein C3 binding to Mycobacterium tuberculosis is initiated by the classical pathway in human bronchoalveolar lavage fluid, Infect Immun, 72, 2564, 10.1128/IAI.72.5.2564-2573.2004

Ernst, 1998, Macrophage receptors for Mycobacterium tuberculosis, Infect Immun, 66, 1277, 10.1128/IAI.66.4.1277-1281.1998

Marth, 1997, Regulation of interleukin-12 by complement receptor 3 signaling, J Exp Med, 185, 1987, 10.1084/jem.185.11.1987

Sutterwala, 1997, Selective suppression of interleukin-12 induction after macrophage receptor ligation, J Exp Med, 185, 1977, 10.1084/jem.185.11.1977

Muller-Eberhard, 1988, Molecular organization and function of the complement system, Annu Rev Biochem, 57, 321, 10.1146/annurev.bi.57.070188.001541

Goldstein, 2004, Tests of glycemia in diabetes, Diabetes Care, 27, S91

Arai, 2010, Metformin, an antidiabetic agent, suppresses the production of tumor necrosis factor and tissue factor by inhibiting early growth response factor-1 expression in human monocytes in vitro, J Pharmacol Exp Ther, 334, 206, 10.1124/jpet.109.164970

Labuzek, 2010, Metformin increases phagocytosis and acidifies lysosomal/endosomal compartments in AMPK-dependent manner in rat primary microglia, Naunyn Schmiedebergs Arch Pharmacol, 381, 171, 10.1007/s00210-009-0477-x

Pearce, 2009, Enhancing CD8 T-cell memory by modulating fatty acid metabolism, Nature, 460, 103, 10.1038/nature08097

Bagdade, 1972, Reversible abnormalities in phagocytic function in poorly controlled diabetic patients, Am J Med Sci, 263, 451, 10.1097/00000441-197206000-00005

Rayfield, 1982, Infection and diabetes: the case for glucose control, Am J Med, 72, 439, 10.1016/0002-9343(82)90511-3

Davidson, 1984, Defective phagocytosis in insulin controlled diabetics: evidence for a reaction between glucose and opsonising proteins, J Clin Pathol, 37, 783, 10.1136/jcp.37.7.783

Hair, 2012, Hyperglycemic conditions inhibit C3-mediated immunologic control of Staphylococcus aureus, J Transl Med, 10, 35, 10.1186/1479-5876-10-35