Questionable association between a monoamine oxidase A promoter polymorphism and sudden infant death syndrome

Neurogenetics - Tập 11 - Trang 367-368 - 2010
Michael Klintschar1, Christian Heimbold2
1Institute of Legal Medicine, Hannover Medical School, Hannover, Germany
2Institute of Legal Medicine, University Göttingen, Göttingen, Germany

Tài liệu tham khảo

Filonzi L, Magnani C, Lavezzi AM, Rindi G, Parmigiani S, Bevilacqua G, Matturri L, Marzano FN (2009) Association of dopamine transporter and monoamine oxidase molecular polymorphisms with sudden infant death syndrome and stillbirth: new insights into the serotonin hypothesis. Neurogenetics 10(1):65–72 Nonnis Marzano F, Maldini M, Filonzi L, Lavezzi AM, Parmigiani S, Magnani C, Bevilacqua G, Matturri L (2008) Genes regulating the serotonin metabolic pathway in the brain stem and their role in the etiopathogenesis of the sudden infant death syndrome. Genomics 91(6):485–491 Poetsch M, Nottebaum BJ, Wingenfeld L, Frede S, Vennemann M, Bajanowski T (2010) Impact of sodium/proton exchanger 3 gene variants on sudden infant death syndrome. J Pediatr 156(1):44–48 Klintschar M, Reichenpfader B (2008) Saternus KS (2008) A functional polymorphism in the tyrosine hydroxylase gene indicates a role of noradrenalinergic signaling in sudden infant death syndrome. J Pediatr 153(2):190–193 Mage DT, Donner M (2004) The X-linkage hypotheses for SIDS and the male excess in infant mortality. Med Hypotheses 62(4):564–567