Polymorphisms in the NQO1, GSTT and GSTM genes are associated with coronary heart disease and biomarkers of oxidative stress
Tài liệu tham khảo
Fried, 2003
N.C.f.H. Statistics National Health Interview Survey 2000, Centers for Disease Control and Prevention, Hyattsville, 2000, p. 92.
Lusis, 2000, Atherosclerosis, Nature, 407, 233, 10.1038/35025203
Braunwald, 1997
Eagles, 1998, Non-pharmacological modification of cardiac risk factors: Part 3. Smoking cessation and alcohol consumption, J. Clin. Pharmacol. Ther., 23, 1, 10.1046/j.1365-2710.1998.00137.x
Khot, 2003, Prevalence of conventional risk factors in patients with coronary heart disease, J. Am. Med. Assoc., 290, 898, 10.1001/jama.290.7.898
Steinberg, 1989, Beyond cholesterol. Modifications of low-density lipoprotein that increase its atherogenicity, N. Engl. J. Med., 320, 915
Pasricha, 2002, When young hearts are broken: profiles of premature myocardial infarction, Am. Heart J., 143, 4, 10.1067/mhj.2002.120301
Ross, 1999, Atherosclerosis—an inflammatory disease, N. Engl. J. Med., 340, 115, 10.1056/NEJM199901143400207
Waart, 2001, Effect of glutathione S-transferase M1 genotype on progression of atherosclerosis in lifelong male smokers, Atherosclerosis, 158, 227, 10.1016/S0021-9150(01)00420-8
Li, 2000, Glutathione S-transferase genotype as a susceptibility factor in smoking-related coronary heart disease, Atherosclerosis, 149, 451, 10.1016/S0021-9150(99)00483-9
Miller, 2003, Glutathione-S-transferase genotypes, smoking, and their association with markers of inflammation, hemostasis, and endothelial function: the atherosclerosis risk in communities (ARIC) study, Atherosclerosis, 171, 265, 10.1016/j.atherosclerosis.2003.07.007
Clairmont, 1999, Association of NAD(P)H:quinone oxidoreductase (NQO1) null with numbers of basal cell carcinomas: use of a multivariate model to rank the relative importance of this polymorphism and those at other relevant loci, Carcinogenesis, 20, 1235, 10.1093/carcin/20.7.1235
Park, 2003, An association between NQO1 genetic polymorphism and risk of bladder cancer, Mut. Res., 536, 131, 10.1016/S1383-5718(03)00041-X
Sachse, 2002, A pharmacogenetic study to investigate the role of dietary carcinogens in the etiology of colorectal cancer, Carcinogenesis, 23, 1839, 10.1093/carcin/23.11.1839
Siegelmann-Danieli, 2002, Significance of genetic variation at the glutathione S-transferase M1 and NAD(P)H:quinone oxidoreductase 1 detoxification genes in breast cancer development, Oncology, 62, 39, 10.1159/000048245
Ross, 2000, NAD(P)H: quinone oxidoreductase 1 (NQO1): chemoprotection, bioactivation, gene regulation and genetic polymorphisms, Chemico-Biol. Interact., 129, 77, 10.1016/S0009-2797(00)00199-X
Gilfix, 1997, Novel reductant for determination of total plasma homocysteine, Clin. Chem., 43, 687, 10.1093/clinchem/43.4.687
Kim, 2000, Polymorphisms of N-acetyltransferase 2, glutathione S-transferase mu and theta genes as risk factors of bladder cancer in relation to asthma and tuberculosis, J. Urol., 164, 209, 10.1016/S0022-5347(05)67496-4
Chen, 1999, Association of the NAD(P)H:quinone oxidoreductase 609C→T polymorphism with a decreased lung cancer risk, Cancer Res., 59, 3045
Pepys, 2003, C-reactive protein: a critical update, J. Clin. Invest., 111, 1805, 10.1172/JCI200318921
Braunwald, 2008, Biomarkers in heart failure, N. Engl. J. Med., 358, 2148, 10.1056/NEJMra0800239
Rasouli, 2005, Plasma homocysteine predicts progression of atherosclerosis, Atherosclerosis, 181, 159, 10.1016/j.atherosclerosis.2005.01.001
Surekha, 2007, Oxidative stress and total antioxidant status in myocardial infarction, Singapore Med. J., 48, 137
Li, 2000, Glutathione S-transferase genotype as a susceptibility factor in smoking-related coronary heart disease, Atherosclerosis, 149, 451, 10.1016/S0021-9150(99)00483-9
Parsricha, 2002, When young hearts are broken: profiles of premature myocardial infarction, Am. Heart J., 143, 4, 10.1067/mhj.2002.120301
Masetti, 2003, Interactive effect of the glutathione S-transferase genes and cigarette smoking on occurrence and severity of coronary artery risk, J. Mol. Med., 81, 488, 10.1007/s00109-003-0448-5
Hong, 2007, Genetic variability in iron-related oxidative stress pathways (Nrf2, NQ01, NOS3, and HO-1), iron intake, and risk of postmenopausal breast cancer, Cancer Epidemiol. Biomarkers Prev., 16, 1784, 10.1158/1055-9965.EPI-07-0247
Tijhuis, 2008, NQO1 and NFE2L2 polymorphisms, fruit and vegetable intake and smoking and the risk of colorectal adenomas in an endoscopy-based population, Int. J. Cancer, 122, 1842, 10.1002/ijc.23246
Steiner, 1998, 606C→T polymorphism in NAD(P)H:quinone oxidoreductase gene in patients with prostatic adenocarcinoma or benign prostatic hyperplasia, Cancer Lett., 135, 67, 10.1016/S0304-3835(98)00269-9
Ross, 2004, Quinone reductases multitasking in the metabolic world, Drug Metab. Rev., 36, 639, 10.1081/DMR-200033465
William, 2005, Carcinogenic polycyclic aromatic hydrocarbon-DNA adducts and mechanism of action, Environ. Mol. Mutagen., 45, 106, 10.1002/em.20095
Beall, 1996, Role of NAD(P)H:quinone oxidoreductase (DT-diaphorase) in cytotoxicity and induction of DNA damage by streptonigrin, Biochem. Pharmacol., 51, 645, 10.1016/S0006-2952(95)00223-5
Gibson, 1992, Relationship between DT-diaphorase-mediated metabolism of a series of aziridinylbenzoquinones and DNA damage and cytotoxicity, Mol. Pharmacol., 42, 531
Nygard, 1997, Plasma homocysteine levels and mortality in patients with coronary artery disease, N. Engl. J. Med., 337, 230, 10.1056/NEJM199707243370403
Ridker, 2001, Measurement of C-reactive protein for the targeting of statin therapy in the primary prevention of acute coronary events, N. Engl. J. Med., 344, 1959, 10.1056/NEJM200106283442601
Danesh, 2004, C-reactive protein and other circulating markers of inflammation in the prediction of coronary heart disease, N. Engl. J. Med., 350, 1387, 10.1056/NEJMoa032804