Origin of ventricular reflexes caused by coronary arteriography.

Heart - Tập 39 Số 9 - Trang 967-973 - 1977
Francisco Pérez-Gómez1, A García-Aguado1
1From the Departamento Cardiopulmonar, Hospital Clinico de la Facultad de Medicina, Madrid, Spain

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Tài liệu tham khảo

Adgey, A. A. J., Geddes, J. S., Mulholland, H. C., Keegan, D. A. J., and Pantridge, J. F. (1968). Incidence, significance, and management of early bradyarrhythmia complicating acute myocardial infarction. Lancet, 2, 1097-1101.

Agress, C. M., Glassner, H. F., Binder, M. J., and Fields, J. (1957). Hemodynamic measurements in experimental coronary shock. Journal of Applied Physiology, 10, 469-475.

Carson, R. P., and Lazzara, R. (1970). Hemodynamic responses initiated by coronary stretch receptors with special reference to coronary arteriography. American Jrournal of Cardiology, 25, 571-578.

Costantin, L. (1963). Extracardial factors contributing to hypotension during coronary occlusion. American Journal of Cardiology, 11, 205-217.

Fluck, D. C., Oslen, E., Pentcost, B. L., Thomas, M., Fillmore, S. J., Shillingford, J. P., and Mounsey, J. P. D. (1967). Natural history and clinical significance of arrhythmias after acute cardiac infarction. British Heart journal, 29, 170-189.

George, M., and Greenwood, T. (1967). Relation between bradycardia and the site of the myocardial infarction. Lancet, 2, 739-740.

Hanley, H. G., Costin, J. C., and Skinner, N. S. (1971). Differential reflex adjustments in cutaneous and muscle vascular beds during experimental coronary artery occlusion. American J7ournal of Cardiology, 27, 513-521.

Hanley, H. G., Raizner, A. E., Inglesby, T. V., and Skinner, N. S. (1972). Response of the renal vascular bed to acute experimental coronary arterial occlusion. American J'ournal of Cardiology, 29, 803-808.

Judkins, M. P. (1968). Percutaneous transfemoral selective coronary arteriography. Radiologic Clinics of North America, 6, 467-492.

Julian, D. G., Valentine, P. A., and Miller, G. C. (1964). Disturbances of rate, rhythm and conduction in acute myocardial infarction. American Journal of Medicine, 37, 915-927.

Kezdi, P., Kordenat, R. K., and Misra, S. N. (1974). Reflex inhibitory effects of vagal afferents in experimental myocardial infarction. American Journal of Cardiology, 33, 853-860.

Kuhn, L. A. (1967). Treatment of cardiogenic shock. American Heart Journal, 74, 578-581.

Linden, R. J. (1975). Reflexes from the heart. Progress in Cardiovascular Diseases, 18, 201-221.

Oberg, B., and Thoren, P. (1972). Studies on left ventricular receptors, signalling non-medullated vagal afferents. Acta Physiologica Scandinavica, 85, 145-163.

Sleight, P., and Widdicombe, J. G. (1965). Action potentials in fibres from receptors in the epicardium and myocardium of the dog's left ventricle. Journal of Physiology, 181, 235-258.

Sones, F. M., and Shirey, E. K. (1962). Cine coronary arteriography. Modern Concepts of Cardiovascular Disease, 31, 735-738.

Thomas, M., and Woodgate, D. (1966). Effect of atropine on bradycardia and hypotension in acute myocardial infarction. British Heart Journal, 28, 409-413.

Thoren, P. (1972). Left ventricular receptors activated by severe asphyxia and by coronary artery occlusion. Acta coronary artery in the cat. Acta Physiologica Scandinavica, Physiologica Scandinavica, 85, 455-463.

Thor6n, P. (1973a). Reflex bradycardia elicited from left ventricular receptors during acute severe hypoxia in cats. Requests for reprints to Dr. F. Perez-Gomez, Acta Physiologica Thoren, P. (1973b).