One-year follow-up of patients of the ongoing Dutch Q fever outbreak: clinical, serological and echocardiographic findings

Springer Science and Business Media LLC - Tập 38 - Trang 471-477 - 2010
G. J. M. Limonard1, M. H. Nabuurs-Franssen2,3, G. Weers-Pothoff4, C. Wijkmans5, R. Besselink6, A. M. Horrevorts2, P. M. Schneeberger4, C. A. R. Groot7
1Department of Pulmonary Diseases, Canisius-Wilhelmina Hospital, Nijmegen, The Netherlands
2Department of Medical Microbiology, Canisius-Wilhelmina Hospital, Nijmegen, The Netherlands
3Department of Medical Microbiology, Radboud University Nijmegen Medical Centre, Nijmegen, The Netherlands
4Department of Medical Microbiology, Jeroen Bosch Hospital, ‘s-Hertogenbosch, The Netherlands
5Gemeentelijke Gezondheidsdienst “Hart voor Brabant” (Municipal Health Service “Hart voor Brabant”), ‘s-Hertogenbosch, The Netherlands
6Huisartsenpraktijk Herpen (GP Practice Herpen), Herpen, The Netherlands
7Bernhoven Hospital, Oss, The Netherlands

Tóm tắt

In 2007, a large goat-farming-associated Q fever outbreak occurred in the Netherlands. Data on the clinical outcome of Dutch Q fever patients are lacking. The current advocated follow-up strategy includes serological follow-up to detect evolution to chronic disease and cardiac screening at baseline to identify and prophylactically treat Q fever patients in case of valvulopathy. However, serological follow-up using commercially available tests is complicated by the lack of validated cut-off values. Furthermore, cardiac screening in the setting of a large outbreak has not been implemented previously. Therefore, we report here the clinical outcome, serological follow-up and cardiac screening data of the Q fever patients of the current ongoing outbreak. The implementation of a protocol including clinical and serological follow-up at baseline and 3, 6 and 12 months after acute Q fever and screening echocardiography at baseline. Eighty-five patients with acute Q fever were identified (male 62%, female 38%). An aspecific, flu-like illness was the most common clinical presentation. Persistent symptoms after acute Q fever were reported by 59% of patients at 6 months and 30% at 12 months follow-up. We observed a typical serological response to Coxiella burnetii infection in both anti-phase I and anti-phase II IgG antibodies, with an increase in antibody titres up to 3 months and a subsequent decrease in the following 9 months. Screening echocardiography was available for 66 (78%) out of 85 Q fever patients. Cardiac valvulopathy was present in 39 (59%) patients. None of the 85 patients developed chronic Q fever. Clinical, serological and echocardiographic data of the current ongoing Dutch Q fever outbreak cohort are presented. Screening echocardiography is no longer part of the standard work-up of Q fever patients in the Netherlands.

Tài liệu tham khảo

Karagiannis I, Morroy G, Rietveld A, et al. Q fever outbreak in the Netherlands: a preliminary report. Euro Surveill. 2007;12:E070809.2. Van Steenbergen JE, Morroy G, Groot CA, et al. An outbreak of Q fever in The Netherlands—possible link to goats. Ned Tijdschr Geneeskd. 2007;151:1998–2003. Karagiannis I, Schimmer B, Van Lier A, et al. Investigation of a Q fever outbreak in a rural area of The Netherlands. Epidemiol Infect. 2009;137:1283–94. Maurin M, Raoult D. Q fever. Clin Microbiol Rev. 1999;12:518–53. Parker NR, Barralet JH, Bell AM. Q fever. Lancet. 2006;367:679–88. Raoult D, Marrie T, Mege J. Natural history and pathophysiology of Q fever. Lancet Infect Dis. 2005;5:219–26. Fenollar F, Thuny F, Xeridat B, et al. Endocarditis after acute Q fever in patients with previously undiagnosed valvulopathies. Clin Infect Dis. 2006;42:818–21. Landais C, Fenollar F, Thuny F, et al. From acute Q fever to endocarditis: serological follow-up strategy. Clin Infect Dis. 2007;44:1337–40. Fine MJ, Auble TE, Yealy DM, et al. A prediction rule to identify low-risk patients with community-acquired pneumonia. N Engl J Med. 1997;336:243–50. Fournier PE, Casalta JP, Habib G, et al. Modification of the diagnostic criteria proposed by the Duke Endocarditis Service to permit improved diagnosis of Q fever endocarditis. Am J Med. 1996;100:629–33. Fournier PE, Raoult D. Comparison of PCR and serology assays for early diagnosis of acute Q fever. J Clin Microbiol. 2003;41:5094–8. Zoghbi WA, Enriquez-Sarano M, Foster E, et al. Recommendations for evaluation of the severity of native valvular regurgitation with two-dimensional and Doppler echocardiography. J Am Soc Echocardiogr. 2003;16:777–802. Quinones MA, Otto CM, Stoddard M, et al. Recommendations for quantification of Doppler echocardiography: a report from the Doppler Quantification Task Force of the Nomenclature and Standards Committee of the American Society of Echocardiography. J Am Soc Echocardiogr. 2002;15:167–84. Lang RM, Bierig M, Devereux RB, et al. Recommendations for chamber quantification: a report from the American Society of Echocardiography’s Guidelines and Standards Committee and the Chamber Quantification Writing Group, developed in conjunction with the European Association of Echocardiography, a branch of the European Society of Cardiology. J Am Soc Echocardiogr. 2005;18:1440–63. Lang RM, Bierig M, Devereux RB, et al. Recommendations for chamber quantification. Eur J Echocardiogr. 2006;7:79–108. Leone M, Honstettre A, Lepidi H, et al. Effect of sex on Coxiella burnetii infection: protective role of 17beta-estradiol. J Infect Dis. 2004;189:339–45. Marmion BP, Shannon M, Maddocks I, et al. Protracted debility and fatigue after acute Q fever. Lancet. 1996;347:977–8. Ayres JG, Smith EG, Flint N. Protracted fatigue and debility after acute Q fever. Lancet. 1996;347:978–9. Wildman MJ, Smith EG, Groves J, et al. Chronic fatigue following infection by Coxiella burnetii (Q fever): ten-year follow-up of the 1989 UK outbreak cohort. QJM. 2002;95:527–38. Hatchette TF, Hayes M, Merry H, et al. The effect of C. burnetii infection on the quality of life of patients following an outbreak of Q fever. Epidemiol Infect. 2003;130:491–5. Hickie I, Davenport T, Wakefield D, et al. Post-infective and chronic fatigue syndromes precipitated by viral and non-viral pathogens: prospective cohort study. BMJ. 2006;333:575. Dupuis G, Peter O, Peacock M, et al. Immunoglobulin responses in acute Q fever. J Clin Microbiol. 1985;22:484–7. Dupont HT, Thirion X, Raoult D. Q fever serology: cutoff determination for microimmunofluorescence. Clin Diagn Lab Immunol. 1994;1:189–96. Fenollar F, Fournier PE, Carrieri MP, et al. Risks factors and prevention of Q fever endocarditis. Clin Infect Dis. 2001;33:312–6. Million M, Lepidi H, Raoult D. Q fever: current diagnosis and treatment options. Med Mal Infect. 2009;39:82–94. Singh JP, Evans JC, Levy D, et al. Prevalence and clinical determinants of mitral, tricuspid, and aortic regurgitation (the Framingham Heart Study). Am J Cardiol. 1999;83:897–902. Jones EC, Devereux RB, Roman MJ, et al. Prevalence and correlates of mitral regurgitation in a population-based sample (the Strong Heart Study). Am J Cardiol. 2001;87:298–304. Lewin MB, Otto CM. The bicuspid aortic valve: adverse outcomes from infancy to old age. Circulation. 2005;111(7):832–4. Hayek E, Gring CN, Griffin BP. Mitral valve prolapse. Lancet. 2005;365:507–18. Schimmer B, Dijkstra F, Vellema P, et al. Sustained intensive transmission of Q fever in the south of the Netherlands, 2009. Euro Surveill. 2009;14:19210. Balakrishnan N, Menon T, Fournier PE, et al. Bartonella quintana and Coxiella burnetii as causes of endocarditis, India. Emerg Infect Dis. 2008;14:1168–9.