NMDA redox site modulates long-term potentiation of NMDA but not of AMPA receptors

European Journal of Pharmacology - Tập 262 - Trang R3-R4 - 1994
Henri Gozlan1, Patrick Chinestra1, Diabé Diabira1, Yezekiel Ben-Ari1
1INSERM U-29, Hôspital de Port-Royal, 123, Boulevard de Port-Royal, 75014 Paris, France

Tài liệu tham khảo

Asztely, 1992, The relative contribution of NMDA receptor channels in the expression of long-term potentiation in the hippocampal CA1 region, Eur. J. Neurosci., 4, 681, 10.1111/j.1460-9568.1992.tb00177.x Ben-Ari, 1992, Protein kinase C modulation of NMDA currents: an important link for LTP induction, Trends Neurosci., 15, 333, 10.1016/0166-2236(92)90049-E Burns, 1991, Selective reduction of disulfides by tris(2-carboxyethyl)phosphine, J. Org. Chem., 56, 2648, 10.1021/jo00008a014 Crépel, 1993, A selective LTP of NMDA receptor-mediated currents induced by anoxia in CA1 hippocampal neurons, J. Neurophysiol., 70, 2045, 10.1152/jn.1993.70.5.2045 McNaughton, 1993, The mechanism of expression of long-term enhancement of hippocampal synapses: current issues and theoretical implications, Annu. Rev. Physiol., 55, 375, 10.1146/annurev.ph.55.030193.002111 Tang, 1993, The modulation of N-methyl-d-aspartate receptors by redox and alkylating reagents in rat cortical neurones in vitro, J. Physiol. (London), 465, 303, 10.1113/jphysiol.1993.sp019678