Modeling the cellular basis of altered excitation–contraction coupling in heart failure
Tài liệu tham khảo
Arai, 1993, Alterations in sarcoplasmic reticulum gene expression in human heart failure: a possible mechanism for alterations in systolic and diastolic properties of the failing myocardium, Circ. Res., 72, 463, 10.1161/01.RES.72.2.463
Bers, D.M., 1991. Excitation–Contraction Coupling and Cardiac Contractile Force. Kluwer, Boston
Beuckelmann, 1992, Intracellular calcium handling in isolated ventricular myocytes from patients with terminal heart failure, Circulation, 85, 1046, 10.1161/01.CIR.85.3.1046
Beuckelmann, 1993, Alterations of K+ currents in isolated human ventricular myocytes from patients with terminal heart failure, Circ. Res., 73, 379, 10.1161/01.RES.73.2.379
Cannell, 1997, Numerical analysis of ryanodine receptor activation by L-type channel activity in the cardiac muscle diad, Biophys. J., 73, 112, 10.1016/S0006-3495(97)78052-4
Davies, 1995, Reduced contraction and altered frequency response of isolated ventricular myocytes from patients with heart failure, Circulation, 92, 2540, 10.1161/01.CIR.92.9.2540
Dec, 1994, Idiopathic dilated cardiomyopathy, N. Engl. J. Med., 331, 1564, 10.1056/NEJM199412083312307
Ghali, 1990, Trends in hospitalization rates for heart failure in the United States 1973–1986. Evidence for increasing prevalence, Arch. Int. Med., 150, 769, 10.1001/archinte.1990.00390160043010
Gomez, 1997, Defective excitation–contraction coupling in experimental heart cardiac hypertrophy and heart failure, Science, 276, 800, 10.1126/science.276.5313.800
Group, 1987, Effects of enalapril on mortality in severe congestive heart failure: results of the cooperative North Scandinavian enalapril survival study (Consensus), N. Engl. J. Med., 316, 1429, 10.1056/NEJM198706043162301
Gwathmey, 1987, Abnormal intracellular calcium handling in myocardium from patients with end-stage heart failure, Circ. Res., 61, 70, 10.1161/01.RES.61.1.70
Hasenfuss, 1994, Relation between myocardial function and expression of sarcoplasmic reticulum Ca–ATPase in failing and nonfailing human myocardium, Circ. Res., 75, 434, 10.1161/01.RES.75.3.434
Hasenfuss, 1996, Calcium cycling proteins and force–frequency relationships in heart failure, Basic Res. Cardiol., 91, 17, 10.1007/BF00795357
Imredy, 1994, Mechanism of Ca2+-sensitive inactivation of L-type Ca2+ channels, Neuron, 12, 1301, 10.1016/0896-6273(94)90446-4
Investigators, 1991, Effect of enalapril on survival in patients with reduced left ventricular ejection fractions and congestive heart failure, N. Engl. J. Med., 325, 293, 10.1056/NEJM199108013250501
Jafri, S., Rice, J.J., Winslow, R.L., 1998. Cardiac Ca2+ dynamics: the roles of ryanodine receptor adaptation and sarcoplasmic reticulum load. Biophys. J. 74, 1149–1168
Janssen, P.M.L., Hunter, W.C., 1995. Force, not sarcomere length, correlates with prolongation of isometric contraction. Am. J. Physiol. H676–H685
Kaab, S., Nuss, H.B., Chiamvimonvat, N., O'Rourke, B., Pak, P.H., Kass, D.A., Marban, E., Tomaselli, G.F., 1996. Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacing-induced heart failure. Circ. Res. 78(2), 262–273
Keizer, 1996, Ryanodine receptor adaptation and Ca2+-induced Ca2+ release-dependent Ca2+ oscillations, Biophys. J., 71, 3477, 10.1016/S0006-3495(96)79543-7
Koumi, 1995, Characterization of inwardly rectifying K+ channel in human cardiac myocytes: alterations in channel behavior in myocytes isolated from patients with idiopathic dilated cardiomyopathy, Circulation, 92, 164, 10.1161/01.CIR.92.2.164
Kubicek, M., Marek, M., 1983. Computational Methods in Bifurcation Theory and Dissipative Structures. Springer-Verlag, New York
Langer, 1996, Calcium concentration and movement in the diadic cleft space of the cardiac ventricular cell, Biophys. J., 70, 1169, 10.1016/S0006-3495(96)79677-7
Li, 1992, Electrophysiologic substrate associated with pacing-induced hert failure in dogs: potential value of programmed stimulation in predicting sudden death, J. Am. Coll. Cardiol., 19, 444, 10.1016/0735-1097(92)90503-F
Limas, 1987, Calcium uptake by cardiac sarcoplasmic reticulum in human dilated cardiomyopathy, Cardiovasc. Res., 21, 601, 10.1093/cvr/21.8.601
Luo, 1994, A dynamic model of the cardiac ventricular action potential: I. Simulations of ionic currents and concentration changes, Circ. Res., 74, 1071, 10.1161/01.RES.74.6.1071
Lytton, 1992, Functional comparisons between isoforms of the sarcoplasmic and endoplasmic reticulum family of calcium pump, J. Biol. Chem., 267, 14483, 10.1016/S0021-9258(19)49738-X
Mercadier, 1990, Altered sarcoplasmic reticulum Ca2+–ATPase gene expression in the human ventricle during end-stage heart failure, J. Clin. Invest., 85, 305, 10.1172/JCI114429
Meyer, 1995, Alterations of sarcoplasmic reticulum proteins in failing human dilated cardiomyopathy, Circulation, 92, 778, 10.1161/01.CIR.92.4.778
Monte, 1995, Cell geometry and contractile abnormalities of myocytes from failing human left ventricle, Cardiovasc. Res., 30, 281, 10.1016/S0008-6363(95)00040-2
Movsesian, 1989, Ca uptake by cardiac sarcoplasmic reticulum from patients with idiopathic dilated cardiomyopathy, Circ. Res., 65, 1141, 10.1161/01.RES.65.4.1141
Nabauer, 1996, Regional differences in current density and rate-dependent properties of the transient outward current in subepicardial and subendocardial myocytes of human left ventricle, Circulation, 93, 168, 10.1161/01.CIR.93.1.168
Ouadid, 1995, Calcium currents in diseased human cardiac cells, J. Cardiovasc. Pharmacol., 25, 282, 10.1097/00005344-199502000-00014
Packer, 1996, The effect of carvedilol on morbidity and mortality in patients with chronic heart failure, N. Engl. J. Med., 334, 1349, 10.1056/NEJM199605233342101
Parmley, 1991, Congestive heart failure: new frontiers, West. J. Med., 154, 427
Philips, 1990, Post-extrasystolic potentiation and the force–frequency relationships: differential augmentation of myocardial contractility in working myocardium from patients with end-stage heart failure, J. Mol. Cell. Cardiol., 22, 99, 10.1016/0022-2828(90)90975-8
Pieske, 1992, Alterations of the force–frequency relationship in the failing human heart depend on the underlying cardiac disease, Basic Res. Cardiol., 87, 213
Rasmussen, 1990, Calcium antagonist binding sites in failing and nonfailing human ventricular myocardium, Biochem. Pharmacol., 39, 691, 10.1016/0006-2952(90)90147-D
Reinecke, 1996, Cardiac Na/Ca exchange activity in patients with end-stage heart failure, Cardiovasc. Res., 31, 48, 10.1016/S0008-6363(95)00176-X
Rice, J.J., Winslow, R.L., Hunter, W.C., 1998. Comparison of putative cooperative mechanisms in cardiac muscle: length-dependence and dynamic responses. Am. J. Physiol. (in review)
Shannon, 1997, SR Ca uptake rate in permeabilized ventricular myocytes is limited by reverse rate of the SR Ca pump, Biophys. J., 72, A167
Soeller, 1997, Numerical simulation of local calcium movements during L-type calcium channel gating in the cardiac diad, Biophys. J., 73, 97, 10.1016/S0006-3495(97)78051-2
Studer, 1994, Gene expression of the cardiac Na–Ca exchanger in end-stage human heart failure, Circ. Res., 75, 443, 10.1161/01.RES.75.3.443
Takahashi, 1992, Expression of dihydropyridine receptor (Ca2+ channel) and calsequestrin genes in the myocardium of patients with end-stage heart failure, J. Clin. Invest., 90, 927, 10.1172/JCI115969
Vermeulen, 1994, Triggered activity and automaticity in ventricular trabeculae of failing human and rabbit hearts, Cardiovasc. Res., 28, 1547, 10.1093/cvr/28.10.1547
Yasui, 1994, Negative control mechanisms with features of adaptation controls Ca2+ release in cardiac myocytes, Biophys. J., 67, 457, 10.1016/S0006-3495(94)80501-6