Melatonin as an antioxidant: under promises but over delivers

Journal of Pineal Research - Tập 61 Số 3 - Trang 253-278 - 2016
Rüssel J. Reiter1, Juan C. Mayo1, Dun‐Xian Tan1, Rosa M. Sáinz1, Moisés Alejandro Alatorre-Jiménez1, Liuqun Qin1
1Department of Cellular and Structural Biology, University of Texas Health Science Center, San Antonio, TX USA

Tóm tắt

Abstract

Melatonin is uncommonly effective in reducing oxidative stress under a remarkably large number of circumstances. It achieves this action via a variety of means: direct detoxification of reactive oxygen and reactive nitrogen species and indirectly by stimulating antioxidant enzymes while suppressing the activity of pro‐oxidant enzymes. In addition to these well‐described actions, melatonin also reportedly chelates transition metals, which are involved in the Fenton/Haber–Weiss reactions; in doing so, melatonin reduces the formation of the devastatingly toxic hydroxyl radical resulting in the reduction of oxidative stress. Melatonin's ubiquitous but unequal intracellular distribution, including its high concentrations in mitochondria, likely aid in its capacity to resist oxidative stress and cellular apoptosis. There is credible evidence to suggest that melatonin should be classified as a mitochondria‐targeted antioxidant. Melatonin's capacity to prevent oxidative damage and the associated physiological debilitation is well documented in numerous experimental ischemia/reperfusion (hypoxia/reoxygenation) studies especially in the brain (stroke) and in the heart (heart attack). Melatonin, via its antiradical mechanisms, also reduces the toxicity of noxious prescription drugs and of methamphetamine, a drug of abuse. Experimental findings also indicate that melatonin renders treatment‐resistant cancers sensitive to various therapeutic agents and may be useful, due to its multiple antioxidant actions, in especially delaying and perhaps treating a variety of age‐related diseases and dehumanizing conditions. Melatonin has been effectively used to combat oxidative stress, inflammation and cellular apoptosis and to restore tissue function in a number of human trials; its efficacy supports its more extensive use in a wider variety of human studies. The uncommonly high‐safety profile of melatonin also bolsters this conclusion. It is the current feeling of the authors that, in view of the widely diverse beneficial functions that have been reported for melatonin, these may be merely epiphenomena of the more fundamental, yet‐to‐be identified basic action(s) of this ancient molecule.

Từ khóa


Tài liệu tham khảo

10.1152/physiol.00011.2014

Fao C, 1912, Hypertrophie des testicules et de la crete après l'extirpation de la glande pineale chez le cuq, Arch Ital Biol, 57, 233

Izawa Y, 1926, The effect of pinealectomy at 20 days of age on the growth of the reproductive system of male and female albino rat, Trans Soc Pathol Jap, 16, 72

10.1210/endo-54-1-114

10.1021/ja01543a060

10.1021/ja01531a060

10.1210/endo-75-2-238

10.1126/science.148.3677.1609

10.1038/207658a0

10.1210/endo-92-2-423

10.1159/000122222

10.1095/biolreprod23.5.1069

10.1098/rspb.2015.1745

10.1016/0024-3205(74)90180-5

10.1210/endo-113-1-293

Reiter RJ, 1974, Circannual reproductive rhythms in mammals related to photoperiod and pineal function: a review, Chronobiologia, 1, 365

10.1002/jmor.1050980306

10.1007/BF00408855

10.1126/science.142.3595.1071

10.3181/00379727-115-29014

10.1016/0024-3205(65)90202-X

10.1210/endo-76-4-798

10.1126/science.143.3612.1328

10.1038/2011134a0

10.1126/science.169.3950.1093

10.1111/j.1600-079X.2005.00223.x

10.1111/jpi.12336

10.1111/jpi.12274

10.1210/endo-79-6-1168

10.1007/BF00338980

Wurtman RJ, 2013, The pineal gland, Sci Am, 1965, 50

10.1210/endo-92-5-1560

Benson B, 1972, Presence of a non‐melatonin pineal antigonadotropin, Acta Endocrinol, 69, 257

10.1016/0003-2697(73)90220-0

10.1007/BF02324326

Pevet P, 1977, Are the pineal active compounds of mammals proteinaceous in nature? An ultrastructural contribution, Acta Med Pol, 18, 351

10.1007/s10517-006-0365-z

10.1007/s10517-012-1873-7

Quay WB, 1965, Indole derivatives of pineal and related neural and retinal tissues, Pharmacol Rev, 17, 321

10.1210/endo-89-1-301

10.1083/jcb.6.1.133

10.1177/0748730416642657

10.1007/s00429-014-0719-7

10.1016/j.neurol.2014.05.008

10.1111/jpi.12189

10.1017/S0952523814000352

10.1007/0-306-46814-X_3

10.1007/BF01946561

10.3748/wjg.v17.i34.3888

10.1007/s00018-014-1579-2

10.1016/0024-3205(83)90192-3

10.1007/BF00691508

10.1111/joa.12040

10.1111/j.1600-079X.1989.tb00403.x

Dauchy RT, 2013, Effects of spectral transmittance through standard laboratory cages on circadian metabolism and physiology in nude rats, J Am Assoc Lab Anim Sci, 52, 146

10.1111/jpi.12221

10.1038/415493a

10.1002/cne.20970

10.1385/ENDO:27:2:179

10.1111/jpi.12239

10.1530/ERC-15-0030

10.1146/annurev-pharmtox-010814-124742

10.1111/jne.12134

10.1016/S0024-3205(99)00519-6

10.1016/S0047-6374(99)00058-5

Manchester LC, 1995, Melatonin immunoreactivity in the photosynthetic prokaryote Rhodospirillum rubrum: implications for an ancient antioxidant defense system, Cell Mol Biol Res, 41, 391

10.1111/j.1600-079X.1994.tb00106.x

10.1159/000109557

10.1034/j.1600-079X.2003.00040.x

10.1111/j.1600-079X.1995.tb00136.x

Hattori A, 1995, Identification of melatonin in plants and its effects on plasma melatonin levels and binding to melatonin receptors in vertebrates, Biochem Mol Biol Int, 35, 627

10.1111/jpi.12253

Hardeland R, 1996, Chronobiology of indoleamines in the dinoflagellate Gonyaulax polyedra: metabolism and effects related to circadian rhythmicity and photoperiodism, Braz J Med Biol Res, 29, 119

10.1096/fj.06-7745com

10.1111/jpi.12115

10.1093/jxb/erq378

10.3390/molecules20047396

10.3390/molecules201018886

Tan DX, 1993, Melatonin: a potent, endogenous hydroxyl radical scavenger, Endocr J, 1, 57

10.1006/bbrc.1998.9826

10.1016/0891-5849(96)00046-9

10.1111/j.1600-079X.1995.tb00170.x

Pierrefiche G, 1993, Antioxidant activity of melatonin in mice, Res Commun Chem Pathol Pharmacol, 80, 211

10.1111/j.1600-079X.1994.tb00119.x

10.1111/j.1600-079X.1995.tb00164.x

10.1016/0024-3205(94)00417-Q

10.1016/0891-5849(95)00101-3

10.1016/0165-7992(95)90065-9

Vijayalaxmi, 1995, Marked reduction of radiation‐induced micronuclei in human blood lymphocytes pre‐treated with melatonin, Radiat Res, 18, 104

10.1016/S0891-5849(96)00614-4

10.1111/j.1600-079X.1998.tb00387.x

10.1016/S0891-5849(98)00226-3

10.1034/j.1600-079X.2001.280201.x

10.1039/c0cp02801k

10.1016/j.ab.2011.09.008

10.1111/jpi.12128

10.1006/frne.1995.1014

10.1111/j.1600-079X.2005.00311.x

10.1016/j.tem.2007.10.007

10.1111/jpi.12202

10.1111/jpi.12254

10.1111/jpi.12247

10.1111/jpi.12090

10.1111/jpi.12162

10.1111/jpi.12267

10.1016/0197-0186(94)00154-M

10.1111/j.1600-079X.1995.tb00178.x

10.1159/000109459

10.1016/S0197-0186(97)00043-0

10.1046/j.1600-079X.2003.00092.x

10.1111/jpi.12018

10.1016/S0891-5849(99)00131-8

10.1023/B:MCBI.0000028742.83086.43

10.1111/j.1600-079X.2005.00295.x

Poeggeler B, 1995, Melatonin, a mediator of electron transfer and repair reactions, acts synergistically with the chain breaking antioxidant ascorbate, trolox, and glutathione, Neuroendocrinol Lett, 17, 87

10.1211/0022357011777747

10.1016/S0024-3205(97)00008-8

10.1111/j.1600-079X.1999.tb00611.x

10.1016/0024-3205(94)00532-X

10.1111/j.1600-079X.1996.tb00260.x

10.1111/j.1600-079X.1999.tb00597.x

10.1111/j.1600-079X.2005.00261.x

Galano A, 2014, Cyclic 3‐hydroxymelatonin, a key metabolite enhancing the peroxyl radical scavenging activity of melatonin, RSC Adv, 41, 304

10.2174/0929867321666131129113146

10.1016/S0891-5849(00)00435-4

10.2174/1568026023394443

10.1179/135100003225002709

10.1111/j.1600-079X.2006.00379.x

10.1111/j.1600-079X.2009.00701.x

10.1111/j.1600-079X.2008.00614.x

10.1111/jpi.12010

10.3389/fphys.2014.00377

10.1111/j.1753-4887.2001.tb07018.x

10.1093/jxb/err256

10.1111/jpi.12159

10.1111/jpi.12246

10.1111/jpi.12262

10.1111/j.1600-079X.1998.tb00361.x

10.1016/j.mehy.2015.11.018

10.1034/j.1600-079X.2002.01859.x

10.1016/S0304-4165(02)00527-5

10.1034/j.1600-079X.2003.00042.x

10.1034/j.1600-079X.2003.00058.x

10.1111/jpi.12196

10.1196/annals.1306.012

10.1016/j.cbpa.2007.01.678

10.1021/cr040410w

10.1016/j.cbpa.2008.02.019

10.1371/journal.pone.0000334

10.1007/s00795-006-0326-7

10.1080/00365520601075662

10.1111/j.1600-079X.2006.00407.x

10.1111/jpi.12132

10.1385/ENDO:27:2:119

10.1111/j.1600-079X.2012.01014.x

10.1111/j.1742-4658.2006.05322.x

10.1111/j.1469-7793.2003.00335.x

10.1042/BJ20081386

10.1016/0167-4943(91)90022-I

Kalous M, 1996, The role of mitochondria in aging, Physiol Res, 45, 351

10.1159/000046885

10.1097/00003246-199504000-00011

Mistra V, 2007, Oxidative stress and role of antioxidant supplementation in critical illness, Clin Lab, 53, 199

10.1155/2011/194586

10.3109/10715769609149066

10.1016/j.abb.2003.12.025

10.1002/mnfr.201100509

10.1073/pnas.0931245100

10.1016/j.bbadis.2012.07.009

Galley HF, 2012, Bench‐to‐bedside review: targeting antioxidants to mitochondria in sepsis, Crit Care, 14, 230, 10.1186/cc9098

10.1074/jbc.M009093200

10.1074/jbc.M301089200

10.1074/jbc.M404003200

10.1124/jpet.110.176743

10.2174/0929867322666150619104143

10.1093/bja/aes577

10.1111/j.1600-079X.2011.00931.x

10.3390/ijms17060939

10.1111/j.1469-185X.2009.00118.x

10.1097/01.CCM.0000174478.70338.03

10.1002/(SICI)1096-9896(200002)190:3<255::AID-PATH526>3.0.CO;2-6

10.1016/j.redox.2015.08.020

10.1016/j.pharmthera.2014.05.005

10.1155/2016/2592935

10.1111/j.1600-079X.1997.tb00331.x

10.1111/j.1600-079X.2011.00932.x

10.1016/j.neuroscience.2013.01.059

10.1097/00004647-199905000-00005

10.1111/jpi.12156

10.1111/jpi.12148

10.1254/jphs.13220FP

10.1089/biores.2015.0032

10.1034/j.1600-079X.2001.310409.x

10.1038/jp.2014.186

10.1111/j.1600-079X.1998.tb00558.x

10.1152/ajpheart.00163.2009

Liu LF, 2014, Protective effects of melatonin on ischemia‐reperfusion induced myocardial damage and hemodynamic recovery in rats, Eur Rev Med Pharmacol Sci, 18, 3681

10.1111/jpi.12161

10.1111/jpi.12312

10.1111/jpi.12286

10.1111/jpi.12138

10.1016/j.ijcard.2016.07.108

10.1111/jpi.12224

10.1517/14728222.2016.1091882

10.1007/s00018-008-8001-x

10.1016/j.nbd.2015.04.020

10.2174/1568026615666141209160556

10.4330/wjc.v2.i8.233

10.1111/j.1600-079X.2012.01001.x

10.1016/j.ijcard.2012.04.110

10.1016/j.cct.2006.10.007

10.2741/4063

10.4330/wjc.v6.i3.100

10.1016/j.ijcard.2016.07.056

10.2741/E617

10.1111/jpi.12348

10.1016/S1474-4422(16)00114-9

10.1016/j.bcp.2010.07.041

10.3390/ijms14048638

Da A, 2012, Overexpression of melatonin membrane receptors increases calcium‐binding proteins and protects VSC4.1 motoneurons from glutamate toxicity through multiple mechanisms, J Pineal Res, 54, 58

10.1007/BF03033152

10.1111/j.1600-079X.2006.00395.x

10.1016/j.phrs.2013.07.008

10.1111/jpi.12070

10.1016/j.bbrc.2015.08.064

10.1111/jpi.12193

10.3892/br.2016.645

10.1016/S0003-4975(01)03101-0

10.1111/jpi.12020

Sewerynek E, 1996, Oxidative damage in the liver induced by ischemia‐reperfusion: protection by melatonin, Hepatogastroenterology, 43, 898

10.1006/jsre.2001.6185

Okatani Y, 2003, Melatonin and N‐acetylcysteine have beneficial effects during hepatic ischemia and reperfusion, Eur J Pharmacol, 34, 260

10.1111/jpi.12326

10.1111/jpi.12273

10.1034/j.1600-079X.2003.02937.x

10.1016/j.lfs.2004.08.031

10.1034/j.1600-079X.2003.00038.x

10.1111/j.1464-410X.2012.11746.x

10.1016/S0022-5347(05)65048-3

10.1177/147323000403200507

10.1111/j.1600-079X.2005.00215.x

10.1111/j.1600-079X.2009.00703.x

10.1038/sc.2015.204

10.1111/jpi.12251

10.1016/j.trre.2016.02.003

10.1016/j.lfs.2006.06.024

10.1111/j.1600-079X.2004.00193.x

Freitas I, 2006, In situ demonstration of improvement in liver mitochondria function by melatonin after cold ischemia, In Vivo, 20, 229

10.1111/j.1600-079X.2010.00831.x

10.1111/j.1600-079X.2011.00908.x

10.1530/JOE-16-0117

10.1007/s00018-016-2185-2

10.1186/s13048-016-0245-8

10.1211/002235702760345374

10.1023/A:1015376328431

10.1016/j.jacl.2014.03.004

10.5847/wjem.j.issn.1920-8642.2012.01.002

10.1016/j.jacl.2014.02.011

10.1007/s11606-014-3115-3

10.1016/j.amjcard.2012.12.037

Vandolder R, 2000, Rhabdomyolysis, J Am Soc Nephrol, 11, 1553, 10.1681/ASN.V1181553

10.4067/S0716-97602004000400022

10.1016/j.bbadis.2009.06.005

10.1111/jpi.12264

10.1111/jpi.12310

10.1016/j.neuint.2016.05.003

10.1111/j.1600-079X.2011.00892.x

10.1016/j.acthis.2014.09.010

10.1111/iep.12094

Benova T, 2015, Acute anti‐fibrillating and defibrillating potential of atorvastatin melatonin, eicosapentaenoic acid and docosahexaenoic acid demonstrated in isolated heart model, J Physiol Pharmacol, 66, 83

10.1016/j.jfma.2012.01.016

10.5812/ijhrba.25075

10.1002/bdrc.21130

McDonnell‐Dowling K, 2016, The role of oxidative stress in methamphetamine‐induced toxicity and sources of variation in the design of animal studies, Curr Neuropharmacol

10.2174/1381612822666151214125657

10.1111/j.1600-079X.2010.00805.x

10.1007/s12640-012-9350-7

10.1111/jpi.12225

10.1016/j.brainres.2016.05.049

10.1016/j.neulet.2015.09.011

10.1111/jpi.12195

10.1016/j.ejphar.2009.06.044

10.1093/alcalc/agv126

10.1111/jpi.12192

10.1111/jpi.12305

10.1111/jpi.12026

10.1111/jpi.12176

10.1139/cjpp-2015-0277

10.1007/s00232-015-9855-0

10.1111/jpi.12288

10.1111/jpi.12217

10.1016/j.canlet.2009.06.016

10.1517/14728222.2013.834890

10.1111/j.1600-079X.2009.00727.x

10.1111/jpi.12205

10.1016/j.canlet.2015.10.015

10.1111/cpr.12158

10.1111/j.1600-079X.2004.00165.x

10.1371/journal.pone.0102776

10.1111/j.1600-0714.2010.01002.x

10.2174/157015910792246209

10.1111/j.1600-079X.2011.00937.x

10.1111/jpi.12260

10.1385/ENDO:27:2:169

10.2741/4217

10.1111/jpi.12212

10.2174/1568026615666141209160556

10.1111/jpi.12203

10.1111/jpi.12116

10.1089/rej.2013.1542

10.1111/jpi.12252

10.1039/C4FO00317A

10.3109/07853890.2011.586365

10.1111/jpi.12137

10.1111/jpi.12240

10.1203/00006450-200112000-00021

10.1111/jpi.12099

10.1111/jpi.12134

10.1111/jpi.12226

10.1111/j.1600-079X.2011.00888.x

Ma Z, 2016, Melatonin as a potential anticarcinogen for non‐small cell lung cancer, Oncotarget

10.1016/j.cbi.2014.06.016

10.1111/jpi.12279

10.1111/jpi.12207

10.1016/j.actatropica.2013.09.014

10.1111/jpi.12123

10.1111/jpi.12256

10.1016/j.actatropica.2014.12.007

10.1111/j.1600-079X.2009.00688.x

10.1111/jpi.12186

10.1002/jmv.24130

10.1038/nrmicro3524

10.2174/187221412799015317

10.1016/j.phrs.2016.01.018

Carrillo‐Vico A, 2006, The modulatory role of melatonin on immune responsiveness, Curr Opin Investig Drugs, 7, 423

10.1016/j.pneurobio.2015.02.001

10.3390/ijms160816981

10.2174/156720208783565645

Rodella LF, 2013, Vascular endothelial cells and dysfunctions: role of melatonin, Front Biosci, 5, 119

10.1111/j.1600-079X.2007.00499.x

Saiz JC, 2016, Zika virus: the latest newcomer, Front Microbiol, 7, 496

10.1002/rmv.1714

10.1111/j.1600-079X.2012.00995.x

10.1111/jpi.12063

10.1016/j.actatropica.2014.04.021

Reiter RJ, 1980, The pineal gland: a regulator of regulators, Prog Psychobiol Physiol Psychol, 9, 323

Ebadi M, 1993, Pineal gland in synchronizing and refining physiological events, N Physiol Sci, 8, 30

10.1016/0024-3205(78)90191-1

10.1016/S0079-6123(08)81008-4

10.3109/09513590.2014.978851