Interaction with surrounding normal epithelial cells influences signalling pathways and behaviour of Src-transformed cells

Journal of Cell Science - Tập 123 Số 2 - Trang 171-180 - 2010
Mihoko Kajita1, Catherine Hogan1, Andrew R. Harris2, Sophie Dupré‐Crochet1, Nobue Itasaki3, Koichi Kawakami4, Guillaume Charras5,2, Masazumi Tada5, Yasuyuki Fujita5,1
1MRC Laboratory for Molecular Cell Biology and Cell Biology Unit, University College London, London WC1E 6BT, UK
2London Centre for Nanotechnology, London, WC1H 0AH, UK
3Division of Developmental Neurobiology, National Institute for Medical Research, London, NW7 1AA, UK
4Division of Molecular and Developmental Biology, National Institute of Genetics and Department of Genetics, The Graduate University for Advanced Studies (SOKENDAI), Mishima, Shizuoka 411-8540, Japan
5Department of Cell and Developmental Biology, University College London, London WC1E 6BT, UK

Tóm tắt

At the initial stage of carcinogenesis, transformation occurs in a single cell within an epithelial sheet. However, it remains unknown what happens at the boundary between normal and transformed cells. Using Madin-Darby canine kidney (MDCK) cells transformed with temperature-sensitive v-Src, we have examined the interface between normal and Src-transformed epithelial cells. We show that Src-transformed cells are apically extruded when surrounded by normal cells, but not when Src cells alone are cultured, suggesting that apical extrusion occurs in a cell-context-dependent manner. We also observe apical extrusion of Src-transformed cells in the enveloping layer of zebrafish gastrula embryos. When Src-transformed MDCK cells are surrounded by normal MDCK cells, myosin-II and focal adhesion kinase (FAK) are activated in Src cells, which further activate downstream mitogen-activated protein kinase (MAPK). Importantly, activation of these signalling pathways depends on the presence of surrounding normal cells and plays a crucial role in apical extrusion of Src cells. Collectively, these results indicate that interaction with surrounding normal epithelial cells influences the signalling pathways and behaviour of Src-transformed cells.

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