IgA nephropathy in a laboratory worker that progressed to end-stage renal disease: a case report

Berger J, Hinglais N. Intercapillary deposits of IgA-IgG. J Urol Nephrol (Paris). 1968;74(9):694–5.

Yu HH, Chiang BL. Diagnosis and classification of IgA nephropathy. Autoimmun Rev. 2014;13(4–5):556–9.

Galla JH. IgA nephropathy. Kidney Int. 1995;47(2):377–87.

Barratt J, Feehally J. IgA nephropathy. J Am Soc Nephrol. 2005;16(7):2088–97.

Floege J, Feehally J. IgA nephropathy: recent developments. J Am Soc Nephrol. 2000;11(12):2395–403.

Suzuki H, et al. The pathophysiology of IgA nephropathy. J Am Soc Nephrol. 2011;22(10):1795–803.

Lauwerys R, et al. Kidney disorders and hematotoxicity from organic solvent exposure. Scand J Work Environ Health. 1985;11 Suppl 1:83–90.

Brautbar N. Industrial solvents and kidney disease. Int J Occup Environ Health. 2004;10(1):79–83.

Emancipator S. Immunoregulatory factors in the pathogenesis of IgA nephropathy. Kidney Int. 1990;38(6):1216–29.

Porro A, et al. Chronic glomerulonephritis and exposure to solvents: a case-referent study. Br J Ind Med. 1992;49(10):738–42.

Stengel B, et al. Glomerular nephropathies and exposure to organic solvents, a case–control study. Bull Acad Natl Med. 1996;180(4):871–9. discussion 879–83.

Jacob S, et al. Effect of organic solvent exposure on chronic kidney disease progression: the GN-PROGRESS cohort study. J Am Soc Nephrol. 2007;18(1):274–81.

Jacob S, et al. New insight into solvent-related end-stage renal disease: occupations, products and types of solvents at risk. Occup Environ Med. 2007;64(12):843–8.

Haas M. Histologic subclassification of IgA nephropathy: a clinicopathologic study of 244 cases. Am J Kidney Dis. 1997;29(6):829–42.

Miller L, et al. Acute tubular necrosis after inhalation exposure to methylene chloride: Report of a case. Arch Intern Med. 1985;145(1):145–6.

Ana-Lilia G-Y, et al. Occupational exposure to toluene and its possible causative role in renal damage development in shoe workers. Int Arch Occup Environ Health. 2006;79(3):259–64.

Ravnskov U. Hydrocarbons may worsen renal function in glomerulonephritis: a meta-analysis of the case–control studies. Am J Ind Med. 2000;37(6):599–606.

Churchill D, Fine A, Gault M. Association between hydrocarbon exposure and glomerulonephritis. An appraisal of the evidence. Nephron. 1983;33(3):169–72.

Ravnskov U. Hydrocarbon exposure may cause glomerulonephritis and worsen renal function: evidence based on Hill’s criteria for causality. QJM. 2000;93(8):551–6.

Yaqoob M, et al. Primary glomerulonephritis and hydrocarbon exposure: a case–control study and literature review. QJM. 1992;83(2):409–18.

Ravnskov U. Experimental glomerulonephritis induced by hydrocarbon exposure: a systematic review. BMC Nephrol. 2005;6(1):15.

Fored CM, et al. Absence of association between organic solvent exposure and risk of chronic renal failure: a nationwide population-based case–control study. J Am Soc Nephrol. 2004;15(1):180–6.

Asal NR, et al. Hydrocarbon exposure and chronic renal disease. Int Arch Occup Environ Health. 1996;68(4):229–35.

Fernández J, et al. Follow-up of two patients with mesangial IgA glomerulonephritis exposed to cadmium and organic solvents. In: Anales del sistema sanitario de Navarra. 2009.

Albrecht WN, Boiano JM, Smith RD. IgA glomerulonephritis in a plumber working with solvent-based pipe cement. Ind Health. 1987;25(3):157–8.

Nogué S, et al. Chronic overexposure to cadmium fumes associated with IgA mesangial glomerulonephritis. Occup Med. 2004;54(4):265–7.

Qin H, Zhang J, Xu Q. A case of IgA nephropathy union protein in workers after getting rid of exposure to cadmium. Chinese journal of industrial hygiene and occupational diseases. 2010;28(12):947.

Fujii Y, et al. A case of IgA nephropathy associated with silicosis. Nihon Jinzo Gakkai shi. 2001;43(7):613–8.

Wakai K, et al. Risk factors for IgA nephropathy: a case–control study in Japan. Am J Kidney Dis. 1999;33(4):738–45.

Radican L, et al. A retrospective occupational cohort study of end-stage renal disease in aircraft workers exposed to trichloroethylene and other hydrocarbons. Journal of occupational and environmental medicine/American College of Occupational and Environmental Medicine. 2006;48(1):1.

D’Amico G. Natural history of idiopathic IgA nephropathy and factors predictive of disease outcome. IgA Nephropathy. 2004;24(3):179-196.

Geddes CC, et al. A tricontinental view of IgA nephropathy. Nephrol Dial Transplant. 2003;18(8):1541–8.

Schena FP. A retrospective analysis of the natural history of primary IgA nephropathy worldwide. Am J Med. 1990;89(2):209–15.

Le W, et al. Long-term renal survival and related risk factors in patients with IgA nephropathy: results from a cohort of 1155 cases in a Chinese adult population. Nephrol Dial Transplant. 2012;27(4):1479–85.

Coppo R, D’Amico G. Factors predicting progression of IgA nephropathies. J Nephrol. 2005;18(5):503–12.

Johnston PA, et al. Clinico-pathological correlations and long-term follow-up of 253 United Kingdom patients with IgA nephropathy. A report from the MRC Glomerulonephritis Registry. Q J Med. 1992;84(304):619–27.

Xie J, et al. Predicting progression of IgA nephropathy: new clinical progression risk score. PLoS One. 2012;7(6):e38904.

Berthoux F, et al. Predicting the risk for dialysis or death in IgA nephropathy. J Am Soc Nephrol. 2011;22(4):752–61.

Radford M, et al. Predicting renal outcome in IgA nephropathy. J Am Soc Nephrol. 1997;8(2):199–207.

Bonfiglioli R, et al. Bilateral hearing loss after dichloromethane poisoning: a case report. Am J Ind Med. 2014;57(2):254–7.

Sakai T, Morita Y, Wakui C. Biological monitoring of workers exposed to dichloromethane, using head-space gas chromatography. J Chromatogr B. 2002;778(1):245–50.