Phân Tích Liên Kết Di Truyền của Các Biến Thể ITGB3 với Độ Tuổi Khởi Phát của Bệnh Schizophrenia

Springer Science and Business Media LLC - Tập 51 - Trang 446-453 - 2013
Ke-Sheng Wang1, Xuefeng Liu1, Tania Bedard Arana2, Nicholas Thompson3, Henry Weisman4, Cecilia Devargas4, ChunXiang Mao5, Brenda Bin Su6, Cynthia Camarillo2, Michael A. Escamilla2, Chun Xu2,6
1Department of Biostatistics and Epidemiology, College of Public Health, East Tennessee State University, Johnson City, USA
2Departments of Psychiatry and Neurology and The Center of Excellence in Neuroscience, Texas Tech University Health Sciences Center, El Paso, USA
3Texas Tech University Health Science Center, El Paso, USA
4Departments of Psychiatry and Neurology, Texas Tech University Health Sciences Center, El Paso, USA
5University of Toronto, Toronto, Canada
6College of Bioinformatics Science and Technology, Harbin Medical University, Harbin, People’s Republic of China

Tóm tắt

Bệnh Schizophrenia (SCZ) là một rối loạn nghiêm trọng với tỷ lệ mắc khoảng 1% trên toàn thế giới. SCZ được biết đến với độ đa dạng di truyền và lâm sàng cao, và là một vấn đề sức khỏe lớn trên toàn cầu. Gen tiểu đơn vị integrin-β 3 (ITGB3) tại vị trí 17q21.32 đã được liên kết với các rối loạn tâm thần. Do đó, chúng tôi giả định rằng các biến thể của gen ITGB3 cũng có thể đóng vai trò trong SCZ và độ tuổi khởi phát (AAO) của SCZ. Chúng tôi đã điều tra các mối liên hệ di truyền của 23 biến thể đơn nucleotide (SNPs) của gen ITGB3 với AAO trong SCZ ở hai mẫu người Caucasian (2.166 ca bệnh và 2.525 kiểm soát) bằng cách sử dụng phân tích hồi quy tuyến tính và phân tích tổng hợp. Chúng tôi phát hiện ra bốn SNP-ITGB3 liên quan đến AAO trong một mẫu không phải từ Mạng Lưới Thông Tin Liên Kết Di Truyền (GAIN) (p < 10−3). Ba trong số bốn SNP này đã được xác nhận trong mẫu GAIN. SNP rs16941771 có mối liên quan đáng kể nhất với AAO (p = 7.47 × 10−5). Phân tích tổng hợp cho thấy 6 trong số 23 SNP có liên quan đến AAO. Phân tích haplotype cũng hỗ trợ sự liên kết của ITGB3 với AAO. Ba SNP liên quan đến bệnh nằm ở các vùng bảo tồn giữa các loài, cho thấy tầm quan trọng chức năng. Đây là báo cáo đầu tiên chỉ ra rằng các biến thể ITGB3 có liên quan đến AAO trong SCZ, cung cấp bằng chứng trực tiếp về việc sử dụng AAO làm thuộc tính trung gian để phân tích di truyền phức tạp của SCZ.

Từ khóa

#schizophrenia #ITGB3 #genetic polymorphisms #age at onset #single nucleotide polymorphisms

Tài liệu tham khảo

Addington AM, Rapoport JL (2009) The genetics of childhood-onset schizophrenia: when madness strikes the prepubescent. Curr Psychiatry Rep 11:156–161 Arranz MJ, Rivera M, Munro JC (2011) Pharmacogenetics of response to antipsychotics in patients with schizophrenia. CNS Drugs 25:933–969. doi:10.2165/11595380-000000000-00000 Barrett JC, Fry B, Maller J, Daly MJ (2005) Haploview: analysis and visualization of LD and haplotype maps. Bioinformatics 21:263–265 Cannon TD, Keller MC (2006) Endophenotypes in the genetic analyses of mental disorders. Annu Rev Clin Psychol 2:267–290. doi:10.1146/annurev.clinpsy.2.022305.095232 Di Castelnuovo A, de Gaetano G, Benedetta Donati M, Iacoviello L (2005) Platelet glycoprotein IIb/IIIa polymorphism and coronary artery disease: implications for clinical practice. Am J Pharmacogenomics 5:93–99 Escamilla M, Hare E, Dassori AM, Peralta JM, Ontiveros A, Nicolini H, Raventos H, Medina R, Mendoza R, Jerez A, Munoz R, Almasy L (2009) A schizophrenia gene locus on chromosome 17q21 in a new set of families of Mexican and Central American ancestry: evidence from the NIMH Genetics of schizophrenia in Latino populations study. Am J Psychiatry 166:442–449. doi:10.1176/appi.ajp.2008.08040612 Farago N, Feher LZ, Kitajka K, Das UN, Puskas LG (2011) MicroRNA profile of polyunsaturated fatty acid treated glioma cells reveal apoptosis-specific expression changes. Lipids Health Dis 10:173. doi:10.1186/1476-511X-10-173 Farrer LA, Cupples LA, Haines JL, Hyman B, Kukull WA, Mayeux R, Myers RH, Pericak-Vance MA, Risch N, van Duijn CM (1997) Effects of age, sex, and ethnicity on the association between apolipoprotein E genotype and Alzheimer disease. A meta-analysis. APOE and Alzheimer Disease Meta Analysis Consortium. JAMA 278:1349–1356 Gabriel SB, Schaffner SF, Nguyen H, Moore JM, Roy J, Blumenstiel B, Higgins J, DeFelice M, Lochner A, Faggart M, Liu-Cordero SN, Rotimi C, Adeyemo A, Cooper R, Ward R, Lander ES, Daly MJ, Altshuler D (2002) The structure of haplotype blocks in the human genome. Science 296:2225–2229. doi:10.1126/science.1069424 Gejman PV, Sanders AR, Duan J (2010) The role of genetics in the etiology of schizophrenia. Psychiatr Clin N Am 33:35–66. doi:10.1016/j.psc.2009.12.003 Greenwood TA, Lazzeroni LC, Murray SS, Cadenhead KS, Calkins ME, Dobie DJ, Green MF, Gur RE, Gur RC, Hardiman G, Kelsoe JR, Leonard S, Light GA, Nuechterlein KH, Olincy A, Radant AD, Schork NJ, Seidman LJ, Siever LJ, Silverman JM, Stone WS, Swerdlow NR, Tsuang DW, Tsuang MT, Turetsky BI, Freedman R, Braff DL (2011) Analysis of 94 candidate genes and 12 endophenotypes for schizophrenia from the Consortium on the Genetics of Schizophrenia. Am J Psychiatry 168:930–946. doi:10.1176/appi.ajp.2011.10050723 Guilmatre A, Dubourg C, Mosca AL, Legallic S, Goldenberg A, Drouin-Garraud V, Layet V, Rosier A, Briault S, Bonnet-Brilhault F, Laumonnier F, Odent S, Le Vacon G, Joly-Helas G, David V, Bendavid C, Pinoit JM, Henry C, Impallomeni C, Germano E, Tortorella G, Di Rosa G, Barthelemy C, Andres C, Faivre L, Frebourg T, Saugier Veber P, Campion D (2009) Recurrent rearrangements in synaptic and neurodevelopmental genes and shared biologic pathways in schizophrenia, autism, and mental retardation. Arch Gen Psychiatry 66:947–956. doi:10.1001/archgenpsychiatry.2009.80 Hamshere ML, Holmans PA, McCarthy GM, Jones LA, Murphy KC, Sanders RD, Gray MY, Zammit S, Williams NM, Norton N, Williams HJ, McGuffin P, O'Donovan MC, Craddock N, Owen MJ, Cardno AG (2011) Phenotype evaluation and genomewide linkage study of clinical variables in schizophrenia. Am J Med Genet B Neuropsychiatr Genet 156B:929–940. doi:10.1002/ajmg.b.31240 Hare E, Glahn DC, Dassori A, Raventos H, Nicolini H, Ontiveros A, Medina R, Mendoza R, Jerez A, Munoz R, Almasy L, Escamilla MA (2010) Heritability of age of onset of psychosis in schizophrenia. Am J Med Genet B Neuropsychiatr Genet 153B:298–302. doi:10.1002/ajmg.b.30959 Harrison PJ, Tunbridge EM (2008) Catechol-O-methyltransferase (COMT): a gene contributing to sex differences in brain function, and to sexual dimorphism in the predisposition to psychiatric disorders. Neuropsychopharmacol: Off Publ Am Coll Neuropsychopharmacol 33:3037–3045. doi:10.1038/sj.npp.1301543 Jarskog LF (2006) Apoptosis in schizophrenia: pathophysiologic and therapeutic considerations. Curr Opin Psychiatry 19:307–312. doi:10.1097/01.yco.0000218603.25346.8f Jarskog LF, Selinger ES, Lieberman JA, Gilmore JH (2004) Apoptotic proteins in the temporal cortex in schizophrenia: high Bax/Bcl-2 ratio without caspase-3 activation. Am J Psychiatry 161:109–115 Jonsson AK, Spigset O, Hagg S (2012) Venous thromboembolism in recipients of antipsychotics: incidence, mechanisms and management. CNS Drugs 26:649–662. doi:10.2165/11633920-000000000-00000 Kendler KS, Neale MC (2010) Endophenotype: a conceptual analysis. Mol Psychiatry 15:789–797. doi:10.1038/mp.2010.8 Kucharska-Newton AM, Monda KL, Campbell S, Bradshaw PT, Wagenknecht LE, Boerwinkle E, Wasserman BA, Heiss G (2011) Association of the platelet GPIIb/IIIa polymorphism with atherosclerotic plaque morphology: the Atherosclerosis Risk in Communities (ARIC) Study. Atherosclerosis 216:151–156. doi:10.1016/j.atherosclerosis.2011.01.038 Kumra S, Asarnow R, Grace A, Keshavan M, McClellan J, Sikich L, Wagner A (2009) From bench to bedside: translating new research from genetics and neuroimaging into treatment development for early-onset schizophrenia. Early Interv Psychiatry 3:243–258. doi:10.1111/j.1751-7893.2009.00142.x Momeni P, DeTucci K, Straub RE, Weinberger DR, Davies P, Grafman J, Hardy J, Huey ED (2010) Progranulin (GRN) in two siblings of a Latino family and in other patients with schizophrenia. Neurocase 16:273–279. doi:10.1080/13554790903456209 Nurden AT, Fiore M, Pillois X, Nurden P (2009) Genetic testing in the diagnostic evaluation of inherited platelet disorders. Semin Thromb Hemost 35:204–212. doi:10.1055/s-0029-1220328 O'Donovan MC, Craddock N, Norton N, Williams H, Peirce T, Moskvina V, Nikolov I, Hamshere M, Carroll L, Georgieva L, Dwyer S, Holmans P, Marchini JL, Spencer CC, Howie B, Leung HT, Hartmann AM, Moller HJ, Morris DW, Shi Y, Feng G, Hoffmann P, Propping P, Vasilescu C, Maier W, Rietschel M, Zammit S, Schumacher J, Quinn EM, Schulze TG, Williams NM, Giegling I, Iwata N, Ikeda M, Darvasi A, Shifman S, He L, Duan J, Sanders AR, Levinson DF, Gejman PV, Cichon S, Nothen MM, Gill M, Corvin A, Rujescu D, Kirov G, Owen MJ, Buccola NG, Mowry BJ, Freedman R, Amin F, Black DW, Silverman JM, Byerley WF, Cloninger CR, Molecular Genetics of Schizophrenia C (2008) Identification of loci associated with schizophrenia by genome-wide association and follow-up. Nat Genet 40:1053–1055. doi:10.1038/ng.201 Oud MJ, Meyboom-de Jong B (2009) Somatic diseases in patients with schizophrenia in general practice: their prevalence and health care. BMC Fam Pract 10:32. doi:10.1186/1471-2296-10-32 Price AL, Patterson NJ, Plenge RM, Weinblatt ME, Shadick NA, Reich D (2006) Principal components analysis corrects for stratification in genome-wide association studies. Nat Genet 38:904–909. doi:10.1038/ng1847 Purcell S, Neale B, Todd-Brown K, Thomas L, Ferreira MA, Bender D, Maller J, Sklar P, de Bakker PI, Daly MJ, Sham PC (2007) PLINK: a tool set for whole-genome association and population-based linkage analyses. Am J Hum Genet 81:559–575. doi:10.1086/519795 Sanders AR, Duan J, Levinson DF, Shi J, He D, Hou C, Burrell GJ, Rice JP, Nertney DA, Olincy A, Rozic P, Vinogradov S, Buccola NG, Mowry BJ, Freedman R, Amin F, Black DW, Silverman JM, Byerley WF, Crowe RR, Cloninger CR, Martinez M, Gejman PV (2008) No significant association of 14 candidate genes with schizophrenia in a large European ancestry sample: implications for psychiatric genetics. Am J Psychiatry 165(4):497–506 Sagud M, Muck-Seler D, Mihaljevic-Peles A, Vuksan-Cusa B, Zivkovic M, Jakovljevic M, Pivac N (2010) Catechol-O-methyl transferase and schizophrenia. Psychiatr Danub 22:270–274 Shi J, Levinson DF, Duan J, Sanders AR, Zheng Y, Pe'er I, Dudbridge F, Holmans PA, Whittemore AS, Mowry BJ, Olincy A, Amin F, Cloninger CR, Silverman JM, Buccola NG, Byerley WF, Black DW, Crowe RR, Oksenberg JR, Mirel DB, Kendler KS, Freedman R, Gejman PV (2009) Common variants on chromosome 6p22.1 are associated with schizophrenia. Nature 460:753–757. doi:10.1038/nature08192 Sullivan PF, Kendler KS, Neale MC (2003) Schizophrenia as a complex trait: evidence from a meta-analysis of twin studies. Arch Gen Psychiatry 60:1187–1192. doi:10.1001/archpsyc.60.12.1187 Vincent JB, Petronis A, Strong E, Parikh SV, Meltzer HY, Lieberman J, Kennedy JL (1999) Analysis of genome-wide CAG/CTG repeats, and at SEF2-1B and ERDA1 in schizophrenia and bipolar affective disorder. Mol Psychiatry 4:229–234 Wang KS, Liu X, Zhang Q, Aragam N, Pan Y (2011) Genome-wide association analysis of age at onset in schizophrenia in a European-American sample. Am J Med Genet B Neuropsychiatr Genet 156B:671–680. doi:10.1002/ajmg.b.31209 Weiss LA, Kosova G, Delahanty RJ, Jiang L, Cook EH, Ober C, Sutcliffe JS (2006) Variation in ITGB3 is associated with whole-blood serotonin level and autism susceptibility. Eur J Hum Genet 14:923–931. doi:10.1038/sj.ejhg.5201644 Xu, Blackburn, Gonzalez, Ramirez, Zavala, Armas, Contreras, Contreras-Rojas, Dassori, Flores L, Jerez, Raventós, Lehman, Ontiveros, Nicolini, Escamilla (2012) Rare copy number variants were identified in schizophrenia and bipolar disorder in the Hispanic population. In: American Society of Human Genetics (ASHG) 2012 annual meeting in San Francisco, California, November 6-10, 2012