G-CSF-producing esophageal cancer with induction of intense bone marrow FDG uptake

Esophagus - Tập 12 - Trang 258-262 - 2014
Taro Oshikiri1, Takashi Yasuda1, Hitoshi Harada1, Masato Ohyama1, Hiroshi Hasegawa1, Tadayuki Ohara1, Hiroyoshi Sendo1, Takemi Sugimoto1, Yasuhiro Fujino1, Masahiro Tominaga1, Yuichi Takahashi2
1Department of Gastroenterological Surgery, Hyogo Cancer Center, Akashi, Japan
2Department of Pathology, Hyogo Cancer Center, Akashi, Japan

Tóm tắt

We report the first case of esophageal squamous cell carcinoma (SCC) producing granulocyte colony-stimulating factor (G-CSF) with induction of high uptake of (18)F-fluorodeoxyglucose (FDG) throughout the bone marrow. A close relationship between tumor status and serum G-CSF concentration was also seen. A 65-year-old man was admitted to our hospital because of esophageal SCC with leukocytosis and pyrexia. His leukocyte count and serum G-CSF concentration were 15900/μl and 140 pg/ml, respectively. Positron emission tomography/computed tomography (PET/CT) showed focal intense bone marrow (18)F-FDG uptake. Hypermetabolic activity in the bone marrow was suspected to be an effect of G-CSF. Under the diagnosis of G-CSF-producing esophageal SCC, the patient underwent minimally invasive esophagectomy in the prone position with 2-field lymph node dissection. After removal of the tumor, the leukocyte count and serum G-CSF concentration rapidly normalized. Immunohistochemically, G-CSF expression was found in the cytoplasm of tumor cells. High uptake of (18)F-FDG throughout the bone marrow decreased. The patient left the hospital on postoperative day 10 without any complications. Changes in bone marrow PET/CT findings and serum G-CSF concentration should be helpful in the definitive diagnosis of G-CSF-producing esophageal SCC and serve as an indicator of therapeutic effect.

Tài liệu tham khảo

Hughes WF, Higley CS. Marked leukocytosis resulting from carcinomatosis. Ann Intern Med. 1952;37:1085–8. Robinson WA. Granulocytosis in neoplasia. Ann N Y Acad Sci. 1974;230:212–8. Asano S, Urabe A, Okabe T, Sato N, Kondo Y, Ueyama Y, et al. Demonstration of granulopoietic factor(s) in the plasma of nude mice transplanted with a human lung cancer and in the tumor tissue. Blood. 1977;49:845–52. Endo K, Kohnoe S, Okamura T, Haraguti M, Adachi E, Toh Y, et al. Gastric adenosquamous carcinoma producing granulocyte-colony stimulating factor. Gastric Cancer. 2005;8:173–7. Aita K, Seki K. Carcinosarcoma of the liver producing granulocyte-colony stimulating factor. Pathol Int. 2006;56:413–9. Furihata M, Shinobe H, Ohtsuki Y, Enzan H, Tokuoka H, Nakanuma Y. An immunohistochemical study on a case of granulocyte-colony stimulating factor-producing gall-bladder carcinoma. Pathol Int. 1999;49:1010–3. Kawakami H, Kuwatani M, Fujita Y, Uebayashi M, Konishi K, Makiyama H, et al. A case of granulocyte-colony stimulating factor producing ductal adenocarcinoma of the pancreas (in Japanese with English abstract). J Jpn Soc Gastroenterol. 2007;104:233–8. Sato Y, Takahashi Y, Nishiie K, Okubo S, Fujikawa K, Shinntani N, et al. A case of granulocyte-colony stimulating factor producing small cell carcinoma of esophagus (in Japanese with English abstract). J Jpn Soc Gastroenterol. 2005;102:888–93. Mimatsu K, Oida T, Kano H, Kawasaki A, Amano S. Aggressive progression of granulocyte colony-stimulating factor producing squamous cell carcinoma of the esophagus: case report and literature review. Esophagus. 2008;5:205–9. Tanabe T, Kanda T, Ishihara N, Kosugi S, Matsuki A, Watanabe G, et al. An esophageal squamous cell carcinoma patient with high serum granulocyte-colony stimulating factor level: report of a case. Esophagus. 2009;6:253–8. Nakamoto Y, Suga T, Hara T, Ishizu K, Togashi K. Inhomogeneous bone marrow uptake caused by G-CSF mimics multiple bone metastases on FDG-PET. Clin Nucl Med. 2010;35:74–6. Mabuchi S, Morimoto A, Fujita M, Isohashi K, Kimura T. G-CSF induces focal intense bone marrow FDG uptake mimicking multiple bone metastases from uterine cervical cancer: a case report and review of the literature. Eur J Gynaecol Oncol. 2012;33:316–7. Morooka M, Kubota K, Murata Y, Shibuya H, Ito K, Mochizuki M, Akashi T, Chiba T, Nomura T, Ito H, Morita T. (18)F-FDG-PET/CT findings of granulocyte colony stimulating factor (G-CSF)-producing lung tumors. Ann Nucl Med. 2008;22:635–9. Hidaka D, Koshizuka H, Hiyama J, Nakatsubo S, Ikeda K, Hayashi A, Fujii A, Sawamoto R, Misumi Y, Miyagawa Y. A case of lung cancer producing granulocyte colony-stimulating factor with a significantly high uptake in the bones observed by a FDG-PET scan. Nihon Kokyuki Gakkai Zasshi. 2009;47:259–63 (in Japanese with English abstract). The Japan Esophageal Society. Guide lines for the clinical and pathologic studies on carcinoma of the esophagus. 10th ed. Tokyo: Kanehara Press; 2007. Kearns CM, Wang WC, Stute N, Ihle JN, Evans WE. Disposition of recombinant human granulocyte colony-stimulating factor in children with severe chronic neutropenia. J Pediatr. 1993;123:471–9. Tsutsumi Y, Shimomura R. In situ hybridization method (in Japanese). Pathol Clin Med. 2005;23:543–51. Sugawara Y, Fisher SJ, Zasodny KR, Kison PV, Baker LH, Wahl RL. Preclinical and clinical studies of bone marrow uptake of fluorine-18-fluorodeoxyglucose with or without granulocyte colony-stimulating factor during chemotherapy. J Clin Oncol. 1998;16:173–80. Murata Y, Kubota K, Yukihiro M, Ito K, Watanabe H, Shibuya H. Correlations between 18F-FDG uptake by bone marrow and hematological parameters: measurements by PET/CT. Nucl Med Biol. 2006;33:999–1004. Tomii K, Iwata T, Oida K, Tanaka F, Kitano M, Kobashi Y. A case of G-CSF producing giant cell type lung cancer with immunohistochemically positive cytoplasmic staining by anti-rhG-CSF polyclonal antibody (in Japanese with English abstract). Jpn J Lung Cancer. 1993;33:563–8. Tachibana M, Miyakawa A, Tazaki H, Nakamura K, Kubo A, Hata J, et al. Autocrine growth of transitional cell carcinoma of the bladder induced by granulocyte-colony stimulating factor. Cancer Res. 1995;55:3438–43. Savarese TM, Mitchell K, McQuain C, Campbell CL, Guardiani R, Wuu J, et al. Coexpression of granulocyte colony stimulating factor and its receptor in primary ovarian carcinomas. Cancer Lett. 2001;162:105–15. Baba M, Hasegawa H, Nakayabu M, Shimizu N, Suzuki S, Kamada N, et al. Establishment and characteristics of a gastriccancer cell line (HuGC-OOHIRA) producing high levels of G-CSF, GM-CSF, and IL-6: the presence of autocrine growth control by G-CSF. Am J Hematol. 1995;49:207–15. Tsuzuki H, Fujieda S, Sunaga H, Noda I, Saito H. Expression of granulocyte colony-stimulating factor receptor correlates with prognosis in oral and mesopharyngeal carcinoma. Cancer Res. 1998;58:794–800. Kyo S, Kanaya T, Takakura M, Inoue M. A case of cervical cancer with aggressive tumor growth: possible autocrine growth stimulation by G-CSF and Il-6. Gynecol Oncol. 2000;78:383–7. Natori T, Sata M, Washida M, Hirata Y, Nagai R, Makuuchi M. G-CSF stimulates angiogenesis and promotes tumor growth: potential contribution of bone marrow-derived endothelial progenitor cells. Biochem Biophys Res Commun. 2002;297:1058–61.