Enhanced effect of VEGF165 on L-type calcium currents in guinea-pig cardiac ventricular myocytes
Tài liệu tham khảo
Taimeh, 2013, Vascular endothelial growth factor in heart failure, Nat. Rev. Cardiol., 10, 519, 10.1038/nrcardio.2013.94
Yla-Herttuala, 2007, Vascular endothelial growth factors: biology and current status of clinical applications in cardiovascular medicine, J. Am. Coll. Cardiol., 49, 1015, 10.1016/j.jacc.2006.09.053
Zhang, 2015, Cardiac regeneration and stem cells, Physiol. Rev., 95, 1189, 10.1152/physrev.00021.2014
Madonna, 2016, Position Paper of the European Society of Cardiology Working Group Cellular Biology of the Heart: cell-based therapies for myocardial repair and regeneration in ischemic heart disease and heart failure, Eur. Heart J., 37, 1789, 10.1093/eurheartj/ehw113
Ferrara, 1997, The biology of vascular endothelial growth factor, Endocr. Rev., 18, 4, 10.1210/edrv.18.1.0287
Abhinand, 2016, VEGF-A/VEGFR2 signaling network in endothelial cells relevant to angiogenesis, J. Cell Commun. Signal., 10.1007/s12079-016-0352-8
Friehs, 2006, Vascular endothelial growth factor prevents apoptosis and preserves contractile function in hypertrophied infant heart, Circulation, 114, I290, 10.1161/CIRCULATIONAHA.105.001289
Chen, 2010, Overexpression of vascular endothelial growth factor 165 (VEGF165) protects cardiomyocytes against doxorubicin-induced apoptosis, J. Chemother., 22, 402, 10.1179/joc.2010.22.6.402
Bers, 2002, Cardiac excitation-contraction coupling, Nature, 415, 198, 10.1038/415198a
Sims, 2008, Sex, age, and regional differences in L-type calcium current are important determinants of arrhythmia phenotype in rabbit hearts with drug-induced long QT type 2, Circ. Res., 102, e86, 10.1161/CIRCRESAHA.108.173740
Mahajan, 2008, Modifying L-type calcium current kinetics: consequences for cardiac excitation and arrhythmia dynamics, Biophys. J., 94, 411, 10.1529/biophysj.106.98590
Chen, 2013, Effects of wenxin keli on the action potential and L-type calcium current in rats with transverse aortic constriction-induced heart failure, Evid. Based Complement. Alternat. Med., 2013, 572078, 10.1155/2013/572078
Guo, 2007, L-type calcium current reactivation contributes to arrhythmogenesis associated with action potential triangulation, J. Cardiovasc. Electrophysiol., 18, 196, 10.1111/j.1540-8167.2006.00698.x
Rials, 1997, Regression of left ventricular hypertrophy with captopril restores normal ventricular action potential duration, dispersion of refractoriness, and vulnerability to inducible ventricular fibrillation, Circulation, 96, 1330, 10.1161/01.CIR.96.4.1330
Qi, 2015, Heterogeneous distribution of INa-L determines interregional differences in rate adaptation of repolarization, Heart Rhythm, 12, 1295, 10.1016/j.hrthm.2015.02.013
Andrikopoulos, 2011, Angiogenic functions of voltage-gated Na+ Channels in human endothelial cells: modulation of vascular endothelial growth factor (VEGF) signaling, J. Biol. Chem., 286, 16846, 10.1074/jbc.M110.187559
Li, 2015, Orai3 surface accumulation and calcium entry evoked by vascular endothelial growth factor, Arterioscler. Thromb. Vasc. Biol., 35, 1987, 10.1161/ATVBAHA.115.305969
Chen, 2002, L-Type Ca2+ channel density and regulation are altered in failing human ventricular myocytes and recover after support with mechanical assist devices, Circ. Res., 91, 517, 10.1161/01.RES.0000033988.13062.7C
Goonasekera, 2012, Decreased cardiac L-type Ca(2)(+) channel activity induces hypertrophy and heart failure in mice, J. Clin. Invest., 122, 280, 10.1172/JCI58227
Meza, 2013, Potent inhibition of L-type Ca2+ currents by a Rad variant associated with congestive heart failure, Biochem. Biophys. Res. Commun., 439, 270, 10.1016/j.bbrc.2013.08.044
Fu, 2014, Basal and beta-adrenergic regulation of the cardiac calcium channel CaV1.2 requires phosphorylation of serine 1700, Proc. Natl. Acad. Sci. U. S. A., 111, 16598, 10.1073/pnas.1419129111
Schwoerer, 2013, Enhanced Ca(2)+ influx through cardiac L-type Ca(2)+ channels maintains the systolic Ca(2)+ transient in early cardiac atrophy induced by mechanical unloading, Pflugers Arch., 465, 1763, 10.1007/s00424-013-1316-y
Koenig, 2014, Enhanced currents through L-type calcium channels in cardiomyocytes disturb the electrophysiology of the dystrophic heart, Am. J. Physiol. Heart Circ. Physiol., 306, H564, 10.1152/ajpheart.00441.2013
Ruixing, 2007, Intramyocardial injection of vascular endothelial growth factor gene improves cardiac performance and inhibits cardiomyocyte apoptosis, Eur. J. Heart Fail., 9, 343, 10.1016/j.ejheart.2006.10.007
Giordano, 2001, A cardiac myocyte vascular endothelial growth factor paracrine pathway is required to maintain cardiac function, Proc. Natl. Acad. Sci. U. S. A., 98, 5780, 10.1073/pnas.091415198
Han, 2006, Overexpression of HAX-1 protects cardiac myocytes from apoptosis through caspase-9 inhibition, Circ. Res., 99, 415, 10.1161/01.RES.0000237387.05259.a5
Lam, 2013, Novel role of HAX-1 in ischemic injury protection involvement of heat shock protein 90, Circ. Res., 112, 79, 10.1161/CIRCRESAHA.112.279935
Vafiadaki, 2007, Phospholamban interacts with HAX-1, a mitochondrial protein with anti-apoptotic function, J. Mol. Biol., 367, 65, 10.1016/j.jmb.2006.10.057
Zhao, 2009, The anti-apoptotic protein HAX-1 is a regulator of cardiac function, Proc. Natl. Acad. Sci. U. S. A., 106, 20776, 10.1073/pnas.0906998106
Vafiadaki, 2009, The role of SERCA2a/PLN complex, Ca(2+) homeostasis, and anti-apoptotic proteins in determining cell fate, Pflugers Arch., 457, 687, 10.1007/s00424-008-0506-5
Vafiadaki, 2009, The anti-apoptotic protein HAX-1 interacts with SERCA2 and regulates its protein levels to promote cell survival, Mol. Biol. Cell, 20, 306, 10.1091/mbc.e08-06-0587
