Early morning hemodynamic changes and left ventricular hypertrophy and mortality in hemodialysis patients

Springer Science and Business Media LLC - Tập 35 - Trang 1399-1407 - 2022
Francesca Mallamaci1,2, Rocco Tripepi1, Claudia Torino1, Giovanni Tripepi1, Pantelis Sarafidis3, Carmine Zoccali4,5,6
1CNR-IFC, Clinical Epidemiology and Physiopathology of Renal Diseases and Hypertension, Reggio Calabria, Italy
2Divisione di Nefrologia e, Trapianto Renale Grande Ospedale Metropolitano, Reggio Calabria, Italy
3Department of Nephrology, Aristotle University of Thessaloniki, Thessaloniki, Greece
4Renal Research Institute, New York, USA
5Associazione Ipertensione Nefrologia e Trapianto Renale (IPNET), Reggio Calabria, Italy
6IPNET C/o CNR-IFC and Nefrologia Grande Ospedale Metropolitano, Reggio Calabria, Italy

Tóm tắt

An exaggeration of the early morning increase in BP, a phenomenon accompanied by a parallel rise in heart rate (HR), is a marker of high cardiovascular risk. The early morning changes in these parameters have not been investigated in the hemodialysis population. In a pilot, single center study including a series of 58 patients we measured the pre-awakening BP and HR surges and the nocturnal dipping of the same parameters as well as other established indicators of autonomic function (weighted 24 h systolic BP and HR variability) and tested their relationship with the left ventricular mass index (LVMI) and with the risk of death over a median follow up of 40 months. The pre-awakening HR surge (r = − 0.46, P = 0.001) but not the corresponding BP surge (r = − 0.1, P = 0.98) was associated with LVMI and the risk of death [HR (1 unit): 0.89, 95% CI 0.83–0.96, P = 0.001]. The link between the pre-awakening HR surge with these outcome measures was robust and largely independent of established risk factors in the hemodialysis population, including the nocturnal dipping of BP. Weighted 24 h systolic BP and HR variability did not correlate with LVMI (all P > 0.11) nor with the risk of death (P > 0.11) and were also independent of the nocturnal dipping of systolic BP and HR. This pilot study suggests that the low early morning changes in HR, likely reflecting enhanced sympathetic activity, entail a high risk for left ventricular hypertrophy (LVH) and mortality in the hemodialysis population.

Tài liệu tham khảo

Thosar SS, Butler MP, Shea SA (2018) Role of the circadian system in cardiovascular disease. J Clin Invest 128:2157–2167. https://doi.org/10.1172/JCI80590 Kario K (2010) Morning surge in blood pressure and cardiovascular risk. Hypertension 56:765–773. https://doi.org/10.1161/HYPERTENSIONAHA.110.157149 Fang S-C, Wu Y-L, Tsai P-S (2020) Heart rate variability and risk of all-cause death and cardiovascular events in patients with cardiovascular disease: a meta-analysis of cohort studies. Biol Res Nurs 22:45–56. https://doi.org/10.1177/1099800419877442 Tripepi G, Fagugli RM, Dattolo P et al (2005) Prognostic value of 24-hour ambulatory blood pressure monitoring and of night/day ratio in nondiabetic, cardiovascular events-free hemodialysis patients. Kidney Int 68:1294–1302. https://doi.org/10.1111/j.1523-1755.2005.00527.x Parati G, Ochoa JE, Bilo G et al (2016) Hypertension in Chronic Kidney Disease Part 1. Hypertension 67:1093–1101. https://doi.org/10.1161/HYPERTENSIONAHA.115.06895 Agarwal R, Flynn J, Pogue V et al (2014) Assessment and management of hypertension in patients on dialysis. J Am Soc Nephrol 25:1630–1646. https://doi.org/10.1681/ASN.2013060601 Sarafidis PA, Persu A, Agarwal R et al (2017) Hypertension in dialysis patients: a consensus document by the European Renal and Cardiovascular Medicine (EURECA-m) working group of the European Renal Association-European Dialysis and Transplant Association (ERA-EDTA) and the Hypertension and the Kidne. Nephrol Dial Transpl 32:620–640. https://doi.org/10.1093/ndt/gfw433 Parati G, Stergiou G, O’Brien E et al (2014) European society of hypertension practice guidelines for ambulatory blood pressure monitoring. J Hypertens. https://doi.org/10.1097/HJH.0000000000000221 Bilo G, Grillo A, Guida V, Parati G (2018) Morning blood pressure surge: pathophysiology, clinical relevance and therapeutic aspects. Integr Blood Press Control. https://doi.org/10.2147/IBPC.S130277 Parati G, Ochoa JE, Lombardi C, Bilo G (2013) Assessment and management of blood-pressure variability. Nat Rev Cardiol 10:143–155. https://doi.org/10.1038/nrcardio.2013.1 Zoccali C, Benedetto FA, Mallamaci F et al (2001) Prognostic impact of the indexation of left ventricular mass in patients undergoing dialysis. J Am Soc Nephrol 12 Kossaify A, Nasr M (2019) Diastolic dysfunction and the new recommendations for echocardiographic assessment of left ventricular diastolic function: summary of guidelines and novelties in diagnosis and grading. J Diagn Med Sonogr 35:317–325. https://doi.org/10.1177/8756479319836781 Grubbs FE (1950) Sample criteria for testing outlying observations 21:27–58. https://doi.org/10.1214/AOMS/1177729885 Levin NW, Kotanko P, Eckardt K-U et al (2010) Blood pressure in chronic kidney disease stage 5Dreport from a kidney disease: improving global outcomes controversies conference. Kidney Int. https://doi.org/10.1038/ki.2009.469 Ye R, Liu K, Gong S et al (2019) The association between morning blood pressure and subclinical target organ damage in the normotensive population. J Hypertens 37:1427–1436. https://doi.org/10.1097/HJH.0000000000002036 Parati G, Ochoa JE, Bilo G et al (2016) Hypertension in chronic kidney disease part 2: role of ambulatory and home blood pressure monitoring for assessing alterations in blood pressure variability and blood pressure profiles. Hypertension. https://doi.org/10.1161/HYPERTENSIONAHA.115.06896 Bombelli M, Fodri D, Toso E et al (2014) Relationship among morning blood pressure surge, 24-hour blood pressure variability, and cardiovascular outcomes in a white population. Hypertension 64:943–950. https://doi.org/10.1161/HYPERTENSIONAHA.114.03675 Kario K, Pickering TG, Umeda Y et al (2003) Morning surge in blood pressure as a predictor of silent and clinical cerebrovascular disease in elderly hypertensives: a prospective study. Circulation 107:1401–1406. https://doi.org/10.1161/01.CIR.0000056521.67546.AA Carreira MAMQ, Nogueira AB, Pena FM et al (2015) Detection of autonomic dysfunction in hemodialysis patients using the exercise treadmill test: the role of the chronotropic index, heart rate recovery, and R-R variability. PLoS ONE 10:e0128123. https://doi.org/10.1371/journal.pone.0128123 Soomro QH, Charytan DM (2021) Cardiovascular autonomic nervous system dysfunction in chronic kidney disease and end-stage kidney disease: disruption of the complementary forces. Curr Opin Nephrol Hypertens 30:198–207. https://doi.org/10.1097/MNH.0000000000000686 Zoccali C, Ciccarelli M, Maggiore Q (1982) Defective reflex control of heart rate in dialysis patients: evidence for an afferent autonomic lesion. Clin Sci. https://doi.org/10.1042/cs0630285 Drawz PE, Babineau DC, Brecklin C et al (2014) Heart rate variability is a predictor of mortality in chronic kidney disease: a report from the CRIC study: CRIC study investigators. Am J Nephrol 38:517–528. https://doi.org/10.1159/000357200 Doulgerakis D, Moyssakis I, Kapelios CJ et al (2017) Cardiac autonomic neuropathy predicts all-cause and cardiovascular mortality in patients with end-stage renal failure: a 5-year prospective study. J Comput Des Eng 4:686–694. https://doi.org/10.1016/j.ekir.2017.03.002 Salman IM (2015) Cardiovascular autonomic dysfunction in chronic kidney disease: a comprehensive review. Curr Hypertens Rep. https://doi.org/10.1007/S11906-015-0571-Z Eckberg DL (1997) Sympathovagal balance: a critical appraisal. Circulation 96:3224–3232. https://doi.org/10.1161/01.CIR.96.9.3224 Floras JS, Ponikowski P (2015) The sympathetic/parasympathetic imbalance in heart failure with reduced ejection fraction. Eur Heart J 36:1974–1982. https://doi.org/10.1093/eurheartj/ehv087 Converse RLJ, Jacobsen TN, Toto RD et al (2010) Sympathetic overactivity in patients with chronic renal failure. 327:1912–1918. https://doi.org/10.1056/NEJM199212313272704 Gordan R, Gwathmey JK, Xie L-H (2015) Autonomic and endocrine control of cardiovascular function. World J Cardiol 7:204–214. https://doi.org/10.4330/wjc.v7.i4.204 Zoccali C, Mallamaci F, Tripepi G, Benedetto FA (2001) Autonomic neuropathy is linked to nocturnal hypoxaemia and to concentric hypertrophy and remodelling in dialysis patients. Nephrol Dial Transpl 16:70–77. https://doi.org/10.1093/ndt/16.1.70 Siddiqi L, Prakken NH, Velthuis BK et al (2010) Sympathetic activity in chronic kidney disease patients is related to left ventricular mass despite antihypertensive treatment. Nephrol Dial Transpl. https://doi.org/10.1093/ndt/gfq175 Zoccali C, Mallamaci F, Tripepi G et al (2003) Neuropeptide Y, left ventricular mass and function in patients with end stage renal disease. J Hypertens 21:1355–1362. https://doi.org/10.1097/00004872-200307000-00025 Zoccali C, Mallamaci F, Parlongo S et al (2002) Plasma norepinephrine predicts survival and incident cardiovascular events in patients with end-stage renal disease. Circulation. https://doi.org/10.1161/hc1102.105261 Liu M, Takahashi H, Morita Y et al (2003) Non-dipping is a potent predictor of cardiovascular mortality and is associated with autonomic dysfunction in haemodialysis patients. Nephrol Dial Transpl 18:563–569. https://doi.org/10.1093/NDT/18.3.563 Grassi G, Vailati S, Bertinieri G et al (1998) Heart rate as marker of sympathetic activity. J Hypertens 16:1635–1639. https://doi.org/10.1097/00004872-199816110-00010 Muller JE, Tofler GH, Verrier RL (1995) Sympathetic activity as the cause of the morning increase in cardiac events. Circulation 91:2508–2509. https://doi.org/10.1161/01.CIR.91.10.2508 Grassi G, Bombelli M, Seravalle G et al (2010) Diurnal blood pressure variation and sympathetic activity. Hypertens Res 335(33):381–385. https://doi.org/10.1038/hr.2010.26