ERK1/2 signaling is required for the initiation but not progression of TGFβ-induced lens epithelial to mesenchymal transition (EMT)
Tài liệu tham khảo
Bakin, 2000, Phosphatidylinositol 3-kinase function is required for Transforming Growth Factor β-mediated epithelial to mesenchymal transition and cell migration, J. Biol. Chem., 275, 36803, 10.1074/jbc.M005912200
Boros, 2006, Sef and Sprouty expression in the developing ocular lens: implications for regulating lens cell proliferation and differentiation, Seminars Cell. Dev. Biol., 17, 741, 10.1016/j.semcdb.2006.10.007
Cano, 2000, The transcription factor Snail controls epithelial-mesenchymal transitions by repressing E-cadherin expression, Nat. Cell Biol., 2, 76, 10.1038/35000025
Chambers, 2000, Expression of sprouty2 during early development of the chick embryo is coincident with known sites of FGF signaling, Mech. Dev., 91, 361, 10.1016/S0925-4773(99)00288-9
Chatani, 1995, Cell type-specific modulation of cell growth by Transforming Growth Factor β1 does not correlate with mitogen-activated protein kinase activation, J. Biol. Chem., 270, 30686, 10.1074/jbc.270.51.30686
Chen, 2014, ERK1/2 pathway mediates epithelial-mesenchymal transition by cross-interacting with TGFβ/Smad and jagged/noth signaling pathways in lens epithelial cells, Int. J. Mol. Med., 33, 1664, 10.3892/ijmm.2014.1723
Cho, 2007, Snail is required for Transforming Growth Factor-β-induced epithelial-mesenchymal transition by activating PI3 kinase/Akt signal pathway, Biochem. Biophysical Res. Commun., 353, 337, 10.1016/j.bbrc.2006.12.035
Dawes, 2008, TGFβ-Induced contraction is not promoted by fibronectin-fibronectin receptor interaction, of αSMA expression, Investigative Ophthalmol. Vis. Sci., 49, 650, 10.1167/iovs.07-0586
Dawes, 2009, TGFβ/Smad4-dependent and -independent regulation of human lens epithelial cells, Investigative Ophthalmol. Vis. Sci., 50, 5318, 10.1167/iovs.08-3223
de Iongh, 2005, Transforming Growth Factor-ß induced epithelial-mesenchymal transition in the lens: a model for cataract formation, Cells Tissues Organs, 179, 43, 10.1159/000084508
Hales, 1995, Cataract induction in lenses cultured with Transforming Growth Factor-ß, Investigative Ophthalmol. Vis. Sci., 36, 1709
Hales, 1994, TGF-beta 1 induces lens cells to accumulate alpha-smooth muscle actin, a marker for subcapsular cataracts, Curr. Eye Res., 13, 885, 10.3109/02713689409015091
Iyengar, 2009, Growth factors involved in aqueous-induced lens cell proliferation, Growth Factors., 27, 50, 10.1080/08977190802610916
Iyengar, 2007, Duration of ERK1/2 phosphorylation induced by FGF or ocular media determines lens cell fate, Differentiation, 75, 662, 10.1111/j.1432-0436.2007.00167.x
Jiang, 2010, Role of cross-talk between the Smad2 and MAPK pathways in TGF-β1-induced collagen IV expression in mesangial cells, Int. J. Mol. Med., 26, 571
Khairallah, 2015, Number of people blind or visually impaired by cataract worldwide and in word regions, 1990 to 2010, Investigative Ophthalmol. Vis. Sci., 56, 6762, 10.1167/iovs.15-17201
Kovalenko, 2003, Sef inhibits fibroblast growth factor signaling by inhibiting FGFR1 tryosine phosphorylation and subsequent ERK activation, J. Biol. Chem., 278, 14087, 10.1074/jbc.C200606200
Kramer, 1999, Sprouty: a common antagonist of FGF and EGF signaling pathways in drosophila, Development, 126, 2515, 10.1242/dev.126.11.2515
Le, 2001, FGF signaling in chick lens development, Dev. Biol., 233, 394, 10.1006/dbio.2001.0194
Li, 2009, MAP kinase phosphotase-1, a critical negative regulator of the innate immune response, Int. J. Clin. Exp. Med., 2, 48
Liu, 1994, Induction of cataract-like changes in rat lens epithelial explants by Transforming Growth Factor- β, Investigative Ophthalmol. Vis. Sci., 35, 388
Lovicu, 2001, FGF-induced lens cell proliferation and differentiation is dependent on MAPK (ERK1/2) signalling, Development, 128, 5075, 10.1242/dev.128.24.5075
Lovicu, 2002, TGFβ induces morphological and molecular changes similar to human anterior subcapsular cataract, Br. J. Ophthalmol., 86, 220, 10.1136/bjo.86.2.220
Maruno, 2002, Apoptosis is a feature of TGF beta-induced cataract, Clin. Exp. Optometry, 85, 76, 10.1111/j.1444-0938.2002.tb03012.x
Mason, 2006, Sprouty proteins: multifaceted negative-feedback regulators of receptor tyrosine kinase signaling, Trends Cell Biol., 16, 45, 10.1016/j.tcb.2005.11.004
Massague, 1998, TGF-β signal transduction, Annu. Rev. Biochem., 67, 753, 10.1146/annurev.biochem.67.1.753
Meng, 2012, Tyrosines 303/343/353 within the Sprouty-related domain of Spred2 are essential for its interaction with p85 and inhibitory effect on Ras/ERK activation, Int. J. Biochem. Cell Biol., 44, 748, 10.1016/j.biocel.2012.01.014
Mukesh, 2006, Development of cataract and associated risk factors: the Visual Impairment Project, Arch Ophthalmol, 124, 79, 10.1001/archopht.124.1.79
Nathu, 2009, Temporal changes in MMP mRNa expression in the lens epithelium during anterior subcapsular cataract formation, Exp. Eye Res., 88, 323, 10.1016/j.exer.2008.08.014
Okado, 2002, Smad7 mediates Transforming Growth Factor-β-induced apoptosis in mesangial cells, Kidney Int., 62, 1178, 10.1111/j.1523-1755.2002.kid583.x
Prunotto, 2015, Stable incorporation of α-smooth muscle actin into stress fibers is dependent on specific tropomyosin isoforms, Cytoskeleton (Hoboken), 72, 257, 10.1002/cm.21230
Schlessinger, 2000, Cell signaling by receptor tyrosine kinases, Cell, 103, 211, 10.1016/S0092-8674(00)00114-8
Sebe, 2008, Nephrol. Dial. Transplant., 23, 1537, 10.1093/ndt/gfm789
Shi, 2003, Mechanisms of TGF-β signaling from cell membrane to the nucleus, Cell, 113, 685, 10.1016/S0092-8674(03)00432-X
Shin, 2012, Sprouty is a negative regulator of Transforming Growth Factor β-induced epithelial-to-mesenchymal transition and cataract, Mol. Med., 18, 861, 10.2119/molmed.2012.00111
Shu, 2017, Bone morphogenetic protein-7 suppresses TGFβ2-induced epithelial-mesenchymal transition in the lens: implications for cataract prevention, Investigative Ophthalmol. Vis. Sci., 58, 781, 10.1167/iovs.16-20611
Tefft, 1999, Conserved function of mSpry-2, a murine homolog of Drosophila sprouty, which negatively modulates respiratory organogenesis, Cuurent Biol., 9, 219, 10.1016/S0960-9822(99)80094-3
Tiwari, 2016, Control of fibrotic changes through the synergistic effects of anti-fibronectin antibody and an RGDS-tagged form of the same antibody, Sci. Rep., 6, 30872, 10.1038/srep30872
Upadhya, 2013, MAPK1 is required for establishing the pattern of cell proliferation and for cell survival during lens development, Development, 140, 1573, 10.1242/dev.081042
Valcz, 2012, Increase of α-SMA+ and CK+ cells as an early sign of epithelial-mesenchymal transition during colorectal carcinogenesis, Pathology Oncol. Res., 18, 371, 10.1007/s12253-011-9454-z
Wormstone, 2009, Posterior capsule opacification, Exp. Eye Res., 88, 257, 10.1016/j.exer.2008.10.016
Wrana, 2000, The Smad pathway, Cytokine & Growth Factor Rev., 11, 5, 10.1016/S1359-6101(99)00024-6
Xie, 2004, Activation of the Erk pathway is required for TGF-β1-induced EMT in vitro, Neoplasia, 6, 603, 10.1593/neo.04241
Yan, 2009, Regulation of TGF-β signaling by Smad7, Acta Biochimica Biophysica Sinica, 41, 263, 10.1093/abbs/gmp018
Yu, 2002, TGF-β receptor-activated p38 MAP kinase mediates Smad-independent TGF-β responses, EMBO J., 21, 3749, 10.1093/emboj/cdf366
Zavadil, 2001, Genetic programs of epithelial cell plasticity directed by Transforming Growth Factor-β, Proc. Natl. Acad. Sci., 98, 6686, 10.1073/pnas.111614398
Zhao, 2015, Negative regulation of TGFβ-induced lens epithelial to mesenchymal transition (EMT) by RTK antagonists, Exp. Eye Res., 132, 9, 10.1016/j.exer.2015.01.001
Zhu, 2001, Regulation of Transforming Growth Factor-β signaling, Mol. Cell Biol. Res. Commun., 4, 321, 10.1006/mcbr.2001.0301