Complementation analysis of Chediak-Higashi Syndrome: The same gene may be responsible for the defect in all patients and species

Springer Science and Business Media LLC - Tập 19 - Trang 459-468 - 1993
Charles M. Perou1, Jerry Kaplan1
1Division of Cell Biology and Immunology, Department of Pathology, University of Utah College of Medicine, Salt Lake City

Tóm tắt

Chediak-Higashi Syndrome is an autosomal recessive disorder, characterized by the presence of large intracellular granules, particularly lysosomes and melanosomes. While the Chediak-Higashi Syndrome is a rare disorder in humans, phenotypically similar syndromes are found in other species. Fusion of normal fibroblasts to Chediak fibroblasts complements the Chediak disorder, restoring normal lysosome size and distribution. Fusion of wild-type with Chediak fibroblasts from human, mouse, or mink demonstrates that wild-type fibroblasts can complement any of the Chediak fibroblasts. Complementation was not observed in interspecific hybrids between Chediak fibroblasts from these species, suggesting that the same gene product is defective in humans, mice, and mink.

Tài liệu tham khảo

Oliver, C., and Essner, E. (1975).Lab. Invest. 3217–27. White, J.G., and Clawson, C.C. (1980).Am. J. Pathol. 98151–194. Barak, Y., and Nir, E. (1987).Am. J. Pediatr. Hematol./Oncol. 942–55. Gallin, J.I., Bujak, J.S., Patten, E., and Wolff, S.M. (1974).Blood 43201–206. Root, R.K., Rosenthal, A.S., and Balestra, D.J. (1972).J. Clin. Invest. 51649–665. Bennett, J.M., Blume, R.S., and Wolff, S.M. (1969).J. Lab. Clin. Med. 73235–240. Leader, R.W., Padgett, G.A., and Gorham, J.R. (1963).Blood 22477–484. Nishimura, M., Inoue, M., Nakano, T., Nishikawa, T., Miyamoto, M., Kobayashi, T., and Kitamura, Y. (1989).Blood 74270–273. Prieur, D.J. (1982).An Updated Bibliography of the Chediak-Higashi Syndrome of Man and Animals (Washington State University, Pullman, Washington). Haak, R.A., Ingraham, L.A., Baehner, R.L., and Boxer, L.A. (1979).J. Clin. Invest. 64138–143. Oliver, J.M., Zurier, R.B., and Berlin, R.D. (1975).Nature 253471–473. Oliver, J.M., Krawiec, J.A., and Berlin, R.D. (1976).J. Cell Biol. 69205–210. Boxer, L.A., Albertini, D.E., Baehner, R.L., and Oliver, J.M. (1979).Br. J. Haematol 43207–213. Jenkins, N.A., Justice, M.J., Gilbert, D.J., Chu, M., and Copeland, N.G. (1991).Genomics 9401–403. Prenner, J.D., and Prieur, D.J. (1987).Am. J. Med. Genet. 28455–470. Frankel, F.R., Tucker, R.W., Bruce, J., and Stenberg, R. (1978).J. Cell Biol. 79401–408. Pryzwansky, K.B., Schliwa, M., and Boxer, L.A. (1985).Blood 661398–1403. White, J.G., and Clawson, C.C. (1979).Am. J. Hematol. 7349–356. Schlegel, R.S., and Rechsteiner, M.C. (1975).Cell 5371–379. Deng, Y., and Storrie, B. (1988).Proc. Natl. Acad. Sci. U.S.A. 853860–3864. Botstein, D., Waddel, C.H., and King, J. (1973).J. Mol. Biol. 80669–695. Poteete, A.R., Jarvik, V., and Botstein, D. (1979).Virology 95550–564.