Characterization of human DNGR-1+ BDCA3+ leukocytes as putative equivalents of mouse CD8α+ dendritic cells

Journal of Experimental Medicine - Tập 207 Số 6 - Trang 1261-1271 - 2010
Lionel Franz Poulin1,2,3, Mariolina Salio4, Emmanuel Griessinger1,2,5, Fernando Anjos‐Afonso1,2,5, Ligia Craciun6, Ji‐Li Chen4, Anna M. Keller1,2,5, Olivier Joffre1,2,5, Santiago Zelenay1,2,5, Emma Nye1,2,5, Alaín Le Moine6, Florence Faure7, Vincent Donckier6, David Sancho8, Vincenzo Cerundolo4, Dominique Bonnet1,2,5, Caetano Reis e Sousa1,2,5
1Experimental Pathology Laboratories, Cancer Research UK, London Research Institute, London WC2A 3PX, UK 1 , 2 , 3
2Haematopoietic Stem Cell Laboratory 1 , 2 , 3
3Immunobiology Laboratory, Cancer Research UK, London Research Institute, London WC2A 3PX, UK
4Nuffield Department of Clinical Medicine, Weatherall Institute of Molecular Medicine, Oxford OX3 9DS, UK 4
5Immunobiology Laboratory, Haematopoietic Stem Cell Laboratory, Experimental Pathology Laboratories, Cancer Research UK, London Research Institute, London WC2A 3PX, UK
6Institute for Medical Immunology, Université Libre de Bruxelles, 6041 Gosselies, Belgium 5
7INSERM U932, Paris, France, and Institut Curie, Centre de Recherche, 75248 Paris, France 6
8Department of Vascular Biology and Inflammation, CNIC- Spanish National Centre for Cardiovascular Research “Carlos III”, 28029 Madrid, Spain 7

Tóm tắt

In mouse, a subset of dendritic cells (DCs) known as CD8α+ DCs has emerged as an important player in the regulation of T cell responses and a promising target in vaccination strategies. However, translation into clinical protocols has been hampered by the failure to identify CD8α+ DCs in humans. Here, we characterize a population of human DCs that expresses DNGR-1 (CLEC9A) and high levels of BDCA3 and resembles mouse CD8α+ DCs in phenotype and function. We describe the presence of such cells in the spleens of humans and humanized mice and report on a protocol to generate them in vitro. Like mouse CD8α+ DCs, human DNGR-1+ BDCA3hi DCs express Necl2, CD207, BATF3, IRF8, and TLR3, but not CD11b, IRF4, TLR7, or (unlike CD8α+ DCs) TLR9. DNGR-1+ BDCA3hi DCs respond to poly I:C and agonists of TLR8, but not of TLR7, and produce interleukin (IL)-12 when given innate and T cell–derived signals. Notably, DNGR-1+ BDCA3+ DCs from in vitro cultures efficiently internalize material from dead cells and can cross-present exogenous antigens to CD8+ T cells upon treatment with poly I:C. The characterization of human DNGR-1+ BDCA3hi DCs and the ability to grow them in vitro opens the door for exploiting this subset in immunotherapy.

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