Activation of Akt protects cancer cells from growth inhibition induced by PKM2 knockdown

Springer Science and Business Media LLC - Tập 4 - Trang 1-5 - 2014
Xiaodong Qin1, Yuping Du1, Xing Chen1, Wuyan Li2, Jinghong Zhang3, Jinbo Yang1,4
1School of Life Science, Lanzhou University, Lanzhou, P. R. China
2Department of Engineering Science, Muskingum University, New Concord, USA
3School of Biomedical Science and Institutes of Molecule Medicine, Huaqiao University, Xiamen, P. R. China
4Department of Cancer Biology, Lerner Research Institute, The Cleveland Clinic Foundation, Cleveland, USA

Tóm tắt

PKM2 is an attractive target for cancer therapy, however, for many cancer cells, PKM2 knockdown only leads to a modest impairment of survival and proliferation. It is not known whether PKM2 knockdown rewires cell signaling pathways in these “PKM2 knockdown resistant” cells, and whether the rewired pathways are needed for their survival. In present study, we investigated the effects of PKM2 knockdown on cellular signaling pathways in “PKM2 knockdown resistant” cancer cells. We found that knockdown of PKM2 leads to activation of Akt. Furthermore, we revealed that activation of Akt in PKM2 knockdown cells is a result of glycolysis disruption. Inhibiton of PI3K-Akt signaling pathway leads to significant growth inhibition and apoptosis in PKM2 knockdown cells. Overall, our results indicate that activation of Akt is necessary for the survival of PKM2 knockdown cells. Combing PKM2 knockdown with PI3K or Akt inhibitors may lead to a better chance to kill tumors. Our research may provide an unexpected opportunity for the development and implementation of drugs targeting cell metabolism and aberrant Akt signaling.

Tài liệu tham khảo

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