Effect of calcium channel antagonists on cell membrane potential oscillations and proliferation of cells expressing the ras oncogene

European Journal of Pharmacology - Tập 212 - Trang 105-107 - 1992
E. Wöll1, H. Weiss1, S. Waldegger1, F. Lang1
1Institute for Physiology; University of Innsbruck. Fritz-Pregl-Strasse 3, A-6010 Innsbruck, Austria

Tài liệu tham khảo

Doppler, 1987, Induction of v-mos and activated Ha-ras oncogene expression in quiescent NIH 3T3 cells causes intracellular alkalinisation and cell cycle progression, Gene, 54, 147, 10.1016/0378-1119(87)90357-X Feramisco, 1984, Microinjection of the oncogene form of the human Ha-ras (T24) protein results in rapid proliferation of quiescent cells, Cell, 38, 109, 10.1016/0092-8674(84)90531-2 Glossmann, 1990, Molecular properties of calcium channels, Rev. Physiol. Biochem. Pharmacol., 114, 1, 10.1007/BFb0031018 Jaggi, 1986, The v-mos and H-ras oncogene expression represses glucocorticoid hormone-dependent transcription from the mouse mammary tumor virus LTR, EMBO J., 5, 2609, 10.1002/j.1460-2075.1986.tb04541.x Lang, 1991, Bradykinin-induced oscillations of cell membrane potential in cells expressing the Ha-ras oncogene, J. Biol. Chem., 266, 4938, 10.1016/S0021-9258(19)67739-2 Santos, 1989, Structural and functional properties of ras proteins, FASEB J., 3, 2151, 10.1096/fasebj.3.10.2666231 Stacey, 1984, Transormation of NIH 3T3 cells by microinjection of Ha-ras p21 protein, Nature, 310, 508, 10.1038/310508a0