A Chromatin-Mediated Reversible Drug-Tolerant State in Cancer Cell Subpopulations
Tóm tắt
Từ khóa
Tài liệu tham khảo
Ahmed, 2004, A novel protein with similarities to Rb binding protein 2 compensates for loss of Chk1 function and affects histone modification in fission yeast, Mol. Cell. Biol., 24, 3660, 10.1128/MCB.24.9.3660-3669.2004
Baege, 2004, IGFBP-3, a marker of cellular senescence, is overexpressed in human papillomavirus-immortalized cervical cells and enhances IGF-1-induced mitogenesis, J. Virol., 78, 5720, 10.1128/JVI.78.11.5720-5727.2004
Balaban, 2004, Bacterial persistence as a phenotypic switch, Science, 305, 1622, 10.1126/science.1099390
Bolden, 2006, Anticancer activities of histone deacetylase inhibitors, Nat. Rev. Drug Discov., 5, 769, 10.1038/nrd2133
Buck, 2008, Feedback mechanisms promote cooperativity for small molecule inhibitors of epidermal and insulin-like growth factor receptors, Cancer Res., 68, 8322, 10.1158/0008-5472.CAN-07-6720
Cara, 2001, Retreatment of patients with the same chemotherapy: implications for clinical mechanisms of drug resistance, Ann. Oncol., 12, 23, 10.1023/A:1008389706725
Casa, 2008, The type I insulin-like growth factor receptor pathway: a key player in cancer therapeutic resistance, Front. Biosci., 13, 3273, 10.2741/2925
Chakravarti, 2002, Insulin-like growth factor receptor I mediates resistance to anti-epidermal growth factor receptor therapy in primary human glioblastoma cells through continued activation of phosphoinositide 3-kinase signaling, Cancer Res., 62, 200
Chang, 2008, Transcriptome-wide noise controls lineage choice in mammalian progenitor cells, Nature, 453, 544, 10.1038/nature06965
Christensen, 2007, RBP2 belongs to a family of demethylases, specific for tri-and dimethylated lysine 4 on histone 3, Cell, 128, 1063, 10.1016/j.cell.2007.02.003
Cloos, 2008, Erasing the methyl mark: histone demethylases at the center of cellular differentiation and disease, Genes Dev., 22, 1115, 10.1101/gad.1652908
Cohen, 2008, Dynamic proteomics of individual cancer cells in response to a drug, Science, 322, 1511, 10.1126/science.1160165
Dallas, 2009, Chemoresistant colorectal cancer cells, the cancer stem cell phenotype, and increased sensitivity to insulin-like growth factor-I receptor inhibition, Cancer Res., 69, 1951, 10.1158/0008-5472.CAN-08-2023
Defeo-Jones, 1991, Cloning of cDNAs for cellular proteins that bind to the retinoblastoma gene product, Nature, 352, 251, 10.1038/352251a0
Dhar, 2007, Microbial phenotypic heterogeneity and antibiotic tolerance, Curr. Opin. Microbiol., 10, 30, 10.1016/j.mib.2006.12.007
Eckstein, 2009, Hyperactivation of the insulin-like growth factor receptor I signaling pathway is an essential event for cisplatin resistance of ovarian cancer cells, Cancer Res., 69, 2996, 10.1158/0008-5472.CAN-08-3153
Eot-Houllier, 2009, Histone deacetylase inhibitors and genomic instability, Cancer Lett., 274, 169, 10.1016/j.canlet.2008.06.005
Ernst, 1990, Increased activity of insulin-like growth factor (IGF) in osteoblastic cells in the presence of growth hormone (GH): positive correlation with the presence of the GH-induced IGF-binding protein BP-3, Endocrinology, 127, 807, 10.1210/endo-127-2-807
Eyler, 2008, Survival of the fittest: cancer stem cells in therapeutic resistance and angiogenesis, J. Clin. Oncol., 26, 2839, 10.1200/JCO.2007.15.1829
Fattaey, 1993, Characterization of the retinoblastoma binding proteins RBP1 and RBP2, Oncogene, 8, 3149
Galhardo, 2007, Mutation as a stress response and the regulation of evolvability, Crit. Rev. Biochem. Mol. Biol., 42, 399, 10.1080/10409230701648502
Garcia-Echeverria, 2004, In vivo antitumor activity of NVP-AEW541-A novel, potent, and selective inhibitor of the IGF-IR kinase, Cancer Cell, 5, 231, 10.1016/S1535-6108(04)00051-0
Gascoigne, 2008, Cancer cells display profound intra- and interline variation following prolonged exposure to antimitotic drugs, Cancer Cell, 14, 111, 10.1016/j.ccr.2008.07.002
Glasspool, 2006, Epigenetics as a mechanism driving polygenic clinical drug resistance, Br. J. Cancer, 94, 1087, 10.1038/sj.bjc.6603024
Hemmati, 2003, Cancerous stem cells can arise from pediatric brain tumors, Proc. Natl. Acad. Sci. USA, 100, 15178, 10.1073/pnas.2036535100
Iwase, 2007, The X-linked mental retardation gene SMCX/JARID1C defines a family of histone H3 lysine 4 demethylases, Cell, 128, 1077, 10.1016/j.cell.2007.02.017
Ji, 2006, The impact of human EGFR kinase domain mutations on lung tumorigenesis and in vivo sensitivity to EGFR-targeted therapies, Cancer Cell, 9, 485, 10.1016/j.ccr.2006.04.022
Kang, 2007, Tumorigenesis of chemotherapeutic drug-resistant cancer stem-like cells in brain glioma, Stem Cells Dev., 16, 837, 10.1089/scd.2007.0006
Klose, 2007, The retinoblastoma binding protein RBP2 is an H3K4 demethylase, Cell, 128, 889, 10.1016/j.cell.2007.02.013
Kurata, 2004, Effect of re-treatment with gefitinib (‘Iressa’, ZD1839) after acquisition of resistance, Ann. Oncol., 15, 173, 10.1093/annonc/mdh006
Lan, 2008, Mechanisms involved in the regulation of histone lysine demethylases, Curr. Opin. Cell Biol., 20, 316, 10.1016/j.ceb.2008.03.004
Lavin, 2008, Ataxia-telangiectasia: from a rare disorder to a paradigm for cell signalling and cancer, Nat. Rev. Mol. Cell Biol., 9, 759, 10.1038/nrm2514
Lewis, 2007, Persister cells, dormancy and infectious disease, Nat. Rev. Microbiol., 5, 48, 10.1038/nrmicro1557
McDermott, 2007, Identification of genotype-correlated sensitivity to selective kinase inhibitors by using high-throughput tumor cell line profiling, Proc. Natl. Acad. Sci. USA, 104, 19936, 10.1073/pnas.0707498104
Pollak, 2008, Insulin and insulin-like growth factor signalling in neoplasia, Nat. Rev. Cancer, 8, 915, 10.1038/nrc2536
Quinlan, 2008, Activated Kras, but not Hras or Nras, may initiate tumors of endodermal origin via stem cell expansion, Mol. Cell. Biol., 28, 2659, 10.1128/MCB.01661-07
Rothenberg, 2008, Modeling oncogene addiction using RNA interference, Proc. Natl. Acad. Sci. USA, 105, 10.1073/pnas.0803217105
Secombe, 2007, The Trithorax group protein Lid is a trimethyl histone H3K4 demethylase required for dMyc-induced cell growth, Genes Dev., 21, 537, 10.1101/gad.1523007
Seligson, 2005, Global histone modification patterns predict risk of prostate cancer recurrence, Nature, 435, 1262, 10.1038/nature03672
Stewart, 2006, Clonal isolation of hESCs reveals heterogeneity within the pluripotent stem cell compartment, Nat. Methods, 3, 807, 10.1038/nmeth939
Trumpp, 2008, Mechanisms of disease: cancer stem cells–targeting the evil twin, Nat. Clin. Pract. Oncol., 5, 337, 10.1038/ncponc1110
Vermeulen, 2008, Single-cell cloning of colon cancer stem cells reveals a multi-lineage differentiation capacity, Proc. Natl. Acad. Sci. USA, 105, 13427, 10.1073/pnas.0805706105
