Enhancement of Outward Potassium Current May Participate in β-Amyloid Peptide-Induced Cortical Neuronal Death
Tài liệu tham khảo
Alnemri, 1996, Human ICE/CED-3 protease nomenclature, Cell, 87, 171, 10.1016/S0092-8674(00)81334-3
Arispe, 1993, Alzheimer disease amyloid β protein forms calcium channels in bilayer membranes: Blockade by tromethamine and aluminum, Proc. Natl. Acad. Sci. USA, 90, 567, 10.1073/pnas.90.2.567
Behrens, 1995, Tyrosine kinase inhibitors induce neuronal apoptosis, but attenuate NMDA-mediated neurotoxicity in cortical cell cultures, Soc. Neurosci. Abs., 21, 1585
Bortner, 1997, A primary role for K++, J. Biol. Chem., 272, 32436, 10.1074/jbc.272.51.32436
Burdick, 1992, Assembly and aggregation properties of synthetic Alzheimer's A4/β amyloid peptide analogs, J. Biol. Chem., 267, 546, 10.1016/S0021-9258(18)48529-8
Colom, 1997, Role of potassium channels in amyloid-induced cholinergic cell death, Soc. Neurosci. Abstr., 23, 1629
Forloni, 1993, Apoptosis mediated neurotoxicity induced by chronic application of β amyloid fragment 25-35, Neuroreport, 4, 523, 10.1097/00001756-199305000-00015
Franklin, 1992, Suppression of programmed neuronal death by sustained elevation of cytoplasmic calcium, TINS, 15, 501
Fraser, 1996, Membrane currents induced inXenopus, J. Neurochem., 66, 2034, 10.1046/j.1471-4159.1996.66052034.x
Fraser, 1997, Ionic effects of the Alzheimer's disease β-amyloid precursor protein and its metabolic fragments, TINS, 20, 67
Furukawa, 1994, Amyloid beta protein-induced irreversible current in rat cortical neurones, Neuroreport, 5, 2016, 10.1097/00001756-199410270-00006
Furukawa, 1996, Activation of K+, Nature, 379, 74, 10.1038/379074a0
Gallo, 1987, The role of depolarization in the survival and differentiation of cerebellar granule cells in culture, J. Neurosci., 7, 2203, 10.1523/JNEUROSCI.07-07-02203.1987
Hardy, 1992, Alzheimer's disease: The amyloid cascade hypothesis, Science, 256, 184, 10.1126/science.1566067
Heldin, 1995, Dimerization of cell surface receptors in signal transduction, Cell, 80, 213, 10.1016/0092-8674(95)90404-2
Hille, 1967, The selective inhibition of delayed potassium currents in nerve by tetraethylammonium ion, J. Gen. Physiol., 50, 1287, 10.1085/jgp.50.5.1287
Holmes, 1997, Expression of voltage-gated potassium channels decreases cellular protein tyrosine phosphorylation, J. Neurosci., 17, 8964, 10.1523/JNEUROSCI.17-23-08964.1997
Hughes, 1997, Intracellular K+, J. Biol. Chem., 272, 30567, 10.1074/jbc.272.48.30567
Jalonen, 1997, β-Amyloid peptide-induced morphological changes coincide with increased K+−, Brain Res., 746, 85, 10.1016/S0006-8993(96)01189-4
Johnson, 1994, Possible role of neuronal apoptosis in Alzheimer's disease, Neurobiol. Aging, 15, S187, 10.1016/0197-4580(94)90202-X
Johnson, 1992, A “calcium set-point hypothesis” of neuronal dependence on neurotrophic factor, Exp. Neurol., 115, 163, 10.1016/0014-4886(92)90242-I
Keller, 1997, 17β-Estradiol attenuates oxidative impairment of synaptic Na++, J. Neurosci. Res., 50, 522, 10.1002/(SICI)1097-4547(19971115)50:4<522::AID-JNR3>3.0.CO;2-G
Knox, 1993, Protein tyrosine phosphorylation is mandatory for CD40-mediated rescue of germinal center B cells from apoptosis, Eur. J. Immunol., 23, 2578, 10.1002/eji.1830231030
Koh, 1987, Quantitative determination of glutamate mediated cortical neuronal injury in cell culture by lactate dehydrogenase efflux assay, J. Neurosci. Methods, 20, 83, 10.1016/0165-0270(87)90041-0
Koike, 1989, Role of Ca2+2+, Proc. Natl. Acad. Sci. USA, 86, 6421, 10.1073/pnas.86.16.6421
Kowall, 1992, In vivo, Neurobiol. Aging, 13, 537, 10.1016/0197-4580(92)90053-Z
LaFerla, 1995, The Alzheimer's Aβ peptide induces neurodegeneration and apoptotic cell death in transgenic mice, Nature Genet., 9, 21, 10.1038/ng0195-21
Lavretsky, 1992, A group of potassium-channel blockers–acetylcholine releasers: New potentials for Alzheimer's disease? A review, J. Clin. Psychopharmacol., 12, 110, 10.1097/00004714-199204000-00006
Li, 1995, Neuronal membrane conductance activated by amyloid β peptide: Importance of peptide conformation, Brain Res., 682, 207, 10.1016/0006-8993(95)00264-Q
Loo, 1993, Apoptosis is induced by β-amyloid in cultured central nervous system neurons, Proc. Natl. Acad. Sci. USA, 90, 7951, 10.1073/pnas.90.17.7951
Mattson, 1992, β-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity, J. Neurosci., 12, 376, 10.1523/JNEUROSCI.12-02-00376.1992
Mirzabekov, 1994, Channel formation in planar lipid bilayers by a neurotoxic fragment of the beta-amyloid peptide, Biochem. Biophys. Res. Commun., 202, 1142, 10.1006/bbrc.1994.2047
Neher, 1972, Differential action of TEA++, Pflüger's Arch., 336, 87, 10.1007/BF00592924
Otani, 1993, Tyrosine kinase(s) regulate apoptosis and bcl-2 expression in a growth factor-dependent cell line, J. Biol. Chem., 268, 22733, 10.1016/S0021-9258(18)41588-8
Perregaux, 1994, Interleukin-1β maturation and release in response to ATP and nigericin. Evidence that potassium depletion mediated by these agents is a necessary and common feature of their activity, J. Biol. Chem., 269, 15195, 10.1016/S0021-9258(17)36591-2
Pike, 1996, Attenuation of β-amyloid neurotoxicityin vitro, J. Neurochem., 67, 1774, 10.1046/j.1471-4159.1996.67041774.x
Pike, 1993, Neurodegeneration induced by β-amyloid peptidesin vitro, J. Neurosci., 13, 1676, 10.1523/JNEUROSCI.13-04-01676.1993
Rose, 1993, Cytotoxicity in murine neocortical cell culture
Sanchez, 1992, Voltage-dependent block of fast chloride channels from rat cortical neurons by external tetraethylammonium ion, J. Gen. Physiol., 100, 217, 10.1085/jgp.100.2.217
Simmons, 1993, Amyloid β peptides act directly on single neurons, Neurosci. Lett., 150, 133, 10.1016/0304-3940(93)90519-Q
Snyder, 1994, Amyloid-β aggregation: Selective inhibition of aggregation in mixtures of amyloid with different chain lengths, Biophys. J., 67, 1216, 10.1016/S0006-3495(94)80591-0
Su, 1994, Immunohistochemical evidence for apoptosis in Alzheimer's disease, Neuroreport, 5, 2529, 10.1097/00001756-199412000-00031
Tilly, 1992, Epidermal growth factor and basic fibroblast growth factor suppress the spontaneous onset of apoptosis in cultured rat ovarian granulosa cells and follicles by a tyrosine kinase-dependent mechanism, Mol. Endocrinol., 6, 1942
Walev, 1995, Potassium-inhibited processing of IL-1β in human monocytes, EMBO J., 14, 1607, 10.1002/j.1460-2075.1995.tb07149.x
Watt, 1994, Ultrastructural analysis of β-amyloid-induced apoptosis in cultured hippocampal neurons, Brain Res., 661, 147, 10.1016/0006-8993(94)91191-6
Weiss, 1994, Ca2+, J. Neurochem., 62, 372, 10.1046/j.1471-4159.1994.62010372.x
Wesseling, 1984, Effects of 4-aminopyridine in elderly patients with Alzheimer's disease, New Engl. J. Med., 310, 988, 10.1056/NEJM198404123101514
Yankner, 1990, Neurotrophic and neurotoxic effects of amyloid β protein: Reversal by tachykinin neuropeptides, Science, 250, 279, 10.1126/science.2218531
Yu, 1997, β-Amyloid-induced enhancement of delayed rectifier K+, Soc. Neurosci. Abstr., 23, 1629
Yu, 1997, Mediation of neuronal apoptosis by enhancement of outward potassium current, Science, 278, 114, 10.1126/science.278.5335.114
Yuan, 1993, TheC.elegans, Cell, 75, 641, 10.1016/0092-8674(93)90485-9