Pellino1-mediated TGF-β1 synthesis contributes to mechanical stress induced cardiac fibroblast activation
Tóm tắt
Từ khóa
Tài liệu tham khảo
Morisco, 2003, Is treating cardiac hypertrophy salutary or detrimental: the two faces of Janus, Am J Physiol Heart Circ Physiol, 284, H1043, 10.1152/ajpheart.00990.2002
Burchfield, 2013, Pathological ventricular remodeling: mechanisms: part 1 of 2, Circulation, 128, 388, 10.1161/CIRCULATIONAHA.113.001878
Baudino, 2006, Cardiac fibroblasts: friend or foe?, Am J Physiol Heart Circ Physiol, 291, H1015, 10.1152/ajpheart.00023.2006
Porter, 2009, Cardiac fibroblasts: at the heart of myocardial remodeling, Pharmacol Ther, 123, 255, 10.1016/j.pharmthera.2009.05.002
Bujak, 2007, The role of TGF-beta signaling in myocardial infarction and cardiac remodeling, Cardiovasc Res, 74, 184, 10.1016/j.cardiores.2006.10.002
Huang, 2010, Smad3 mediates cardiac inflammation and fibrosis in angiotensin II-induced hypertensive cardiac remodeling, Hypertension, 55, 1165, 10.1161/HYPERTENSIONAHA.109.147611
Ruwhof, 2000, Cyclic stretch induces the release of growth promoting factors from cultured neonatal cardiomyocytes and cardiac fibroblasts, Mol Cell Biochem, 208, 89, 10.1023/A:1007046105745
Lee, 2006, NF-kappaB and activator protein 1 response elements and the role of histone modifications in IL-1beta-induced TGF-beta1 gene transcription, J Immunol, 176, 603, 10.4049/jimmunol.176.1.603
Schauvliege, 2006, Pellino proteins are more than scaffold proteins in TLR/IL-1R signalling: a role as novel RING E3-ubiquitin-ligases, FEBS Lett, 580, 4697, 10.1016/j.febslet.2006.07.046
Jiang, 2003, Pellino 1 is required for interleukin-1 (IL-1)-mediated signaling through its interaction with the IL-1 receptor-associated kinase 4 (IRAK4)–IRAK–tumor necrosis factor receptor-associated factor 6 (TRAF6) complex, J Biol Chem, 278, 10952, 10.1074/jbc.M212112200
Choi, 2006, Smad6 negatively regulates interleukin 1-receptor–Toll-like receptor signaling through direct interaction with the adaptor Pellino-1, Nat Immunol, 7, 1057, 10.1038/ni1383
Xiao, 2013, Peli1 promotes microglia-mediated CNS inflammation by regulating Traf3 degradation, Nat Med, 19, 595, 10.1038/nm.3111
Li, 2004, NF-kappaB activation is required for the development of cardiac hypertrophy in vivo, Am J Physiol Heart Circ Physiol, 287, H1712, 10.1152/ajpheart.00124.2004
Lebeche, 2004, In vivo cardiac gene transfer of Kv4.3 abrogates the hypertrophic response in rats after aortic stenosis, Circulation, 110, 3435, 10.1161/01.CIR.0000148176.33730.3F
Weber, 1992, Pathologic hypertrophy with fibrosis: the structural basis for myocardial failure, Blood Press, 1, 75, 10.3109/08037059209077497
Lopez, 2010, Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects, Am J Physiol Heart Circ Physiol, 299, H1, 10.1152/ajpheart.00335.2010
Creemers, 2011, Molecular mechanisms that control interstitial fibrosis in the pressure-overloaded heart, Cardiovasc Res, 89, 265, 10.1093/cvr/cvq308
Chang, 2009, Peli1 facilitates TRIF-dependent Toll-like receptor signaling and proinflammatory cytokine production, Nat Immunol, 10, 1089, 10.1038/ni.1777
Chen, 2012, TWEAK/Fn14 promotes the proliferation and collagen synthesis of rat cardiac fibroblasts via the NF-small ka, CyrillicB pathway, Mol Biol Rep, 39, 8231, 10.1007/s11033-012-1671-3
Deb, 2014, Cardiac fibroblast in development and wound healing, J Mol Cell Cardiol, 70, 47, 10.1016/j.yjmcc.2014.02.017
Moynagh, 2009, The Pellino family: IRAK E3 ligases with emerging roles in innate immune signalling, Trends Immunol, 30, 33, 10.1016/j.it.2008.10.001
Schauvliege, 2007, Pellino proteins: novel players in TLR and IL-1R signalling, J Cell Mol Med, 11, 453, 10.1111/j.1582-4934.2007.00040.x
Zhu, 2011, The TIR/BB-loop mimetic AS-1 prevents cardiac hypertrophy by inhibiting IL-1R-mediated MyD88-dependent signaling, Basic Res Cardiol, 106, 787, 10.1007/s00395-011-0182-z
Zelarayan, 2009, NF-kappaB activation is required for adaptive cardiac hypertrophy, Cardiovasc Res, 84, 416, 10.1093/cvr/cvp237
Heineke, 2006, Regulation of cardiac hypertrophy by intracellular signalling pathways, Nat Rev Mol Cell Biol, 7, 589, 10.1038/nrm1983
Esposito, 2010, Induction of mitogen-activated protein kinases is proportional to the amount of pressure overload, Hypertension, 55, 137, 10.1161/HYPERTENSIONAHA.109.135467
Wu, 2014, Silencing of Pellino1 improves post-infarct cardiac dysfunction and attenuates left ventricular remodelling in mice, Cardiovasc Res, 102, 46, 10.1093/cvr/cvu007
Herum, 2013, Syndecan-4 signaling via NFAT regulates extracellular matrix production and cardiac myofibroblast differentiation in response to mechanical stress, J Mol Cell Cardiol, 54, 73, 10.1016/j.yjmcc.2012.11.006
Weber, 2013, Myofibroblast-mediated mechanisms of pathological remodelling of the heart, Nat Rev Cardiol, 10, 15, 10.1038/nrcardio.2012.158
Leemans, 2009, The role of Toll-like receptor 2 in inflammation and fibrosis during progressive renal injury, PLoS ONE, 4, e5704, 10.1371/journal.pone.0005704
Sugiura, 2009, Activation of Toll-like receptor 3 augments myofibroblast differentiation, Am J Respir Cell Mol Biol, 40, 654, 10.1165/rcmb.2008-0371OC
Pulskens, 2010, TLR4 promotes fibrosis but attenuates tubular damage in progressive renal injury, J Am Soc Nephrol, 21, 1299, 10.1681/ASN.2009070722
Jin, 2012, Peli: a family of signal-responsive E3 ubiquitin ligases mediating TLR signaling and T-cell tolerance, Cell Mol Immunol, 9, 113, 10.1038/cmi.2011.60
Adhikari, 2007, Ubiquitin-mediated activation of TAK1 and IKK, Oncogene, 26, 3214, 10.1038/sj.onc.1210413
Vesely, 2009, Translational regulation mechanisms of AP-1 proteins, Mutat Res, 682, 7, 10.1016/j.mrrev.2009.01.001
Huang, 2009, Angiotensin II promotes poly(ADP-ribosyl)ation of c-Jun/c-Fos in cardiac fibroblasts, J Mol Cell Cardiol, 46, 25, 10.1016/j.yjmcc.2008.10.019
Karin, 1995, The regulation of AP-1 activity by mitogen-activated protein kinases, J Biol Chem, 270, 16483, 10.1074/jbc.270.28.16483
McDermott, 2002, Ras participates in the activation of p38 MAPK by interleukin-1 by associating with IRAK, IRAK2, TRAF6, and TAK-1, J Biol Chem, 277, 7808, 10.1074/jbc.M108133200
Tanos, 2005, Phosphorylation of c-Fos by members of the p38 MAPK family. Role in the AP-1 response to UV light, J Biol Chem, 280, 18842, 10.1074/jbc.M500620200
Calleros, 2006, Low cell cholesterol levels increase NFkappaB activity through a p38 MAPK-dependent mechanism, Cell Signal, 18, 2292, 10.1016/j.cellsig.2006.05.012
Li, 1997, Differential protein expression and subcellular distribution of TGFbeta1, beta2 and beta3 in cardiomyocytes during pressure overload-induced hypertrophy, J Mol Cell Cardiol, 29, 2213, 10.1006/jmcc.1997.0457
Pauschinger, 1999, Dilated cardiomyopathy is associated with significant changes in collagen type I/III ratio, Circulation, 99, 2750, 10.1161/01.CIR.99.21.2750