Electron microscopical study of experimentally induced poisoning due to organic mercury compound

Springer Science and Business Media LLC - Tập 14 Số 2 - Trang 126-136 - 1969
Taihei Miyakawa1, Motonori Deshimaru1
1Department of Neuropsychiatry, Kumamoto University, School of Medicine, Kumamoto, Japan

Tóm tắt

Từ khóa


Tài liệu tham khảo

Brown, W. J., andN. Yoshida: Organic mercurial encephalopathy. Advanc. Neurol. Sci.9, 34?42 (1965).

Das, H. K., S. K. Chatterjee, andS. C. Roy: Protein synthesis in plant mitochondria. I. Incorporation of amino acid in peptide linkage. J. biol. Chem.236, 1126?1133 (1964).

Hirakawa, K.: Isolation and identification of organic mercury compounds contained in Hibarigaimodoki obtained from Minamata bay. J. Kumamoto med. Soc.36, 746?763 (1962).

Hunter, D., R. R. Bombord, andD. Russell: Poisoning by methyl mercury compounds. Quart. J. Med.33, 193?219 (1940).

?, andD. S. Russell: Focal cerebral and cerebellar atrophy in human subject due to organic mercurial compounds. Neurol. Neurosurg. Psychiat.17, 235?241 (1954).

Inoue, T.: Identification of an organic mercury compound, methyl methylmercuric sulfide. Its synthesis analytical chemical study and the role in the etiology of Minamata's disease. J. Kumamoto med. Soc.36, 877?889 (1962).

Irukayama, K.: Studies on the mercury compounds in the fish and shellfish from Minamata bay as the causative agent of Minamata disease and the formation of the poison (I?VI). Nippon Eiseigaku Z.16, 385 (1961);16, 467 (1962);19, 238, 246 (1964).

Littlefield, J. W., E. B. Keller, J. Gross, andP. C. Zamecnik: Studies on cytoplasmic ribonucleoprotein particles from the liver of the rat. J. biol. Chem.217, 111?121 (1955).

McLean, J. R., C. L. Cohn, I. R. Brandt, andM. V. Simpsom: Incorporation of labeled amino acids into the protein of muscle and liver mitochondria. J. biol. Chem.233, 657?663 (1958).

Palay, S., S. Mcgee-Russell, G. Spencer, Jr., andM. A. Grills: Fixation of neural tissues for electron microscopy by perfusion with solution of osmium tetroxide. J. Cell Biol.12, 385?410 (1962).

Takeuchi, T., andN. Morikawa: Pathological studies on Minamata diease, especially on the cause of this disease. Psychiat. Neurol. Jap.62, 1850 (1960) (in Japanese, with English abstract).

??,H. Matsumoto, andY. Shiraishi: A pathological study of Minamata disease in Japan. Acta neuropath. (Berl.)2, 40?57 (1962).

?,H. Matsumoto, Y. Shiraishi, G. Koya, M. Sasaki, Y. Hirata, K. Fukimoto, T. Miyazaki, andJ. Ogi: An experimental pathological study on the etiology of Minamata's disease, especially the role of methylmercuric sulfide. J. Kumamoto med. Soc.36, 713?735 (1962).

??,H. Deguchi, M. Sonoda, andF. Kai: Experimental formation of cerebellar atrophy. Cerebellar disturbance by alkylmercury poisoning. Kumamoto Igk. Z.40, 1003 (1966).

??,G. Koya, H. Deguchi, M. Sonoda, andF. Kai: A histopathological study of the brain of cats poisoned with methylmercuric compounds. Kumamoto Igk. Z.40, 1016 (1966).

Tokuomi, H., andT. Okajima: Minamata disease. Wld Neurol.2, 536?545 (1961).

Uchida, M., K. Hirakawa, andT. Inoue: Biochemical studies on Minamata disease (III?IV). Kumamoto med. J.14, 171, 181 (1961);15, 149 (1962).

Yoshino, Y., T. Mozai, andK. Nakao: Distribution of mercury in the brain and its subcellular units in experimental organic mercury poisoning. J. Neurochem.13, 397?406 (1966).

???: Biochemical changes in the brain in rats poisoned with an alkyl mercury compound, with special reference to the inhibition of the protein synthesis in brain cortex slices. J. Neurochem.13, 1223?1230 (1966).